| Name | serum cholinesterase |
|---|---|
| Synonyms | Acylcholine acylhydrolase; BCHE; BCHE protein; Butyrylcholine esterase; Butyrylcholinesterase; CHE1; Choline esterase II; Cholinesterase… |
| Name | fenthion |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 3388751 | Dellinger J, Mostrom M: Effects of topical fenthion on blood cholinesterase and vagal tone in dogs. Vet Hum Toxicol. 1988 Jun;30(3):229-34. |
6(0,0,1,1) | Details |
| 879915 | Gokhale VS, Bapat VM, Kanitkar SV, Kulkarni SD: Altered sensitivity to during chronic administration of organophosphorus anticholinesterase (fenthion) in albino mice. Arch Int Pharmacodyn Ther. 1977 Apr;226(2):331-8. |
6(0,0,1,1) | Details |
| 17698941 | Singh S, Bhardwaj U, Verma SK, Bhalla A, Gill K: Hyperamylasemia and acute pancreatitis following anticholinesterase poisoning. Hum Exp Toxicol. 2007 Jun;26(6):467-71. Except for fenthion, significant persistent increase in serum amylase was not observed with individual compounds. |
5(0,0,0,5) | Details |
| 16119488 | Roberts DM, Fraser JF, Buckley NA, Venkatesh B: Experiences of anticholinesterase pesticide poisonings in an Australian tertiary hospital. Anaesth Intensive Care. 2005 Aug;33(4):469-76. Two cases of intermediate syndrome were attributed to fenthion and diazinon, and one case of delayed polyneuropathy to trichlorfon. |
5(0,0,0,5) | Details |
| 8747743 | Moser VC: Comparisons of the acute effects of cholinesterase inhibitors using a neurobehavioral screening battery in rats. Neurotoxicol Teratol. 1995 Nov-Dec;17(6):617-25. The acute effects of two carbamates (carbaryl, aldicarb) and five organophosphates (OP) (chlorpyrifos, diazinon, parathion, fenthion, and diisopropyl fluorophosphate, or DFP) were evaluated on the day of dosing at the time of peak effect, at 1 and 3 days, and 1 week after dosing (oral gavage, in corn oil). |
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| 11743808 | Pardio VT, Ibarra N, Rodriguez MA, Waliszewski KN: Use of cholinesterase activity in monitoring organophosphate pesticide exposure of cattle produced in tropical areas. J Agric Food Chem. 2001 Dec;49(12):6057-62. Results of multivariate analysis showed that the general responses between the control and experimental groups (in vivo, monitoring and case studies) treated with Coumaphos and Fenthion were statistically different, and the general responses of these experimental groups were statistically different over time as well. |
3(0,0,0,3) | Details |
| 8222687 | De Bleecker J, Van den Neucker K, Colardyn F: Intermediate syndrome in organophosphorus poisoning: a prospective study. Crit Care Med. 1993 Nov;21(11):1706-11. Agents such as methylparathion, fenthion, and dimethoate carry a high risk, but we also noted a prolonged intermediate syndrome in an ethyl-parathion-poisoned patient. OBJECTIVES: To estimate the frequency of the intermediate syndrome in organophosphorus-poisoned patients, and examine its relationship to cholinesterase inhibition and electromyographic findings. |
3(0,0,0,3) | Details |
| 15253053 | Fleischli MA, Franson JC, Thomas NJ, Finley DL, Riley W Jr: Avian mortality events in the United States caused by anticholinesterase pesticides: a retrospective summary of National Wildlife Health Center records from 1980 to 2000. Arch Environ Contam Toxicol. 2004 May;46(4):542-50. Of 24 different pesticides identified, the most frequent were famphur (n = 59: 18%), carbofuran (n = 52; 15%), diazinon (n = 40; 12%), and fenthion (n = 17; 5.1%). |
3(0,0,0,3) | Details |
| 17423767 | Frank R, Braun HE, Wilkie I, Ewing R: A review of insecticide poisonings among domestic livestock in southern Ontario, Canada, 1982-1989. Can Vet J. 1991 Apr;32(4):219-226. Signs typical of cholinesterase inhibition caused by organophosphorus poisoning were observed in most cases. There were six poisoning cases as a result of excessive topical applications of the three insecticides coumpahos, fenthion, and lindane as dusts or sprays to control external parasites. |
2(0,0,0,2) | Details |
| 6968357 | Hall RJ, Kolbe E: Bioconcentration of organophosphorus pesticides to hazardous levels by amphibians. J Toxicol Environ Health. 1980 Jul;6(4):853-60. Frogs are resistant to cholinesterase inhibitors; thus it was suspected that they might accumulate the pesticides. Tadpoles concentrated pesticides from water up to 60 times; those exposed to 1 ppm parathion and 5 ppm fenthion were lethal when they were fed to mallard ducks. |
2(0,0,0,2) | Details |
| 8027507 | Dementi B: Ocular effects of organophosphates: a historical perspective of Saku disease. J Appl Toxicol. 1994 Mar-Apr;14(2):119-29. Follow-up studies in animals performed in Japan using such agents as ethylthiometon, fenthion and fenitrothion demonstrate adverse effects of organophosphates on the visual system. An important question arising from this work is that of the role of cholinesterase inhibition in the etiology of the effects. |
2(0,0,0,2) | Details |
| 8727219 | Nagayama M, Akahori F, Chiwata H, Shirai M, Motoya M, Masaoka T, Sakaguchi K: Effects of selected organophosphate insecticides on serum cholinesterase isoenzyme patterns in the rat. Vet Hum Toxicol. 1996 Jun;38(3):196-9. The effects of organophosphates (fenthion, chlorpyrifos, diazinon, bromophos, propaphos, haloxon, and DFP) on serum ChE isoenzyme bands were studied in 32 male and 32 female 6-w-old Sprague-Dawley rats. |
2(0,0,0,2) | Details |
| 1855619 | Takahashi H, Kojima T, Ikeda T, Tsuda S, Shirasu Y: Differences in the mode of lethality produced through intravenous and oral administration of organophosphorus insecticides in rats. Fundam Appl Toxicol. 1991 Apr;16(3):459-68. This study was undertaken to investigate the possibility that mechanisms other than cholinesterase (ChE) inhibition account for the acute toxicity of organophosphorus insecticide. Both the P = O type insecticide (direct ChE inhibitors: chlorfenvinphos and dichlorvos) and the P = S type insecticide (indirect ChE inhibitors: diazinon and fenthion) were employed. |
1(0,0,0,1) | Details |
| 2376956 | De Wilde V, Vogelaers D, Colardyn F: recovery from severe cholinesterase-inhibitor poisoning--remarks on classification and therapy of organophosphate poisoning. Klin Wochenschr. 1990 Jun 19;68(12):615-8. |
1(0,0,0,1) | Details |
| 8027509 | Boyes WK, Tandon P, Barone S Jr, Padilla S: Effects of organophosphates on the visual system of rats. J Appl Toxicol. 1994 Mar-Apr;14(2):135-43. The possibility that exposure to organophosphate insecticides can lead to ocular damage is suggested by Japanese studies from the 1960s and 1970s indicating that exposed humans developed chronic ocular degeneration, in addition to showing more commonly accepted effects of cholinesterase-inhibiting compounds. Notably, retinas from rats treated with a single subcutaneous injection of 100 mg kg-1 fenthion showed decreases in carbachol-stimulated release of an indicator of cholinergically-mediated intracellular second messenger systems. |
1(0,0,0,1) | Details |
| 8693683 | Tsatsakis AM, Aguridakis P, Michalodimitrakis MN, Tsakalov AK, Alegakis AK, Koumantakis E, Troulakis G: Experiences with acute organophosphate poisonings in Crete. Vet Hum Toxicol. 1996 Apr;38(2):101-7. Significant fluctuations of the plasma cholinesterase activity were observed during therapy. Postmortem analysis revealed higher levels of pesticides in organs (eg 23.1 micrograms fenthion/g kidney) and in fat (135.2 micrograms fenthion/g) than in blood (eg 4.8 micrograms fenthion/ml) and vitreous humor. |
1(0,0,0,1) | Details |
| 2385836 | Veronesi B, Jones K, Pope C: The neurotoxicity of subchronic acetylcholinesterase (AChE) inhibition in rat hippocampus. Toxicol Appl Pharmacol. 1990 Jul;104(3):440-56. These observations indicate that in both young adult and aged animals, subchronic, low-level exposure to anticholinesterase compounds can result in serious consequences to the mammalian hippocampus. The effects of long-term, low-level exposure to the commercially available insecticide, Fenthion, were examined in the present study. |
1(0,0,0,1) | Details |
| 2733059 | Tuler SM, Bowen JM: Toxic effects of organophosphates on nerve cell growth and ultrastructure in culture. J Toxicol Environ Health. 1989;27(2):209-23. In this study, cultured chick dorsal root ganglia (DRG) were used to study the effect of fenthion (FEN), an OP pesticide, on isolated nerve cell growth and ultrastructure. Cultured explants were treated with a lower concentration of FEN (10 (-6) M) and morphologic alterations were compared to those observed in explants treated with 10 (-6) M paraoxon, a more acutely toxic OP, or 10 (-6) M neostigmine, a non-OP inhibitor of acetyl-cholinesterase. |
1(0,0,0,1) | Details |
| 3586207 | Henny CJ, Kolbe EJ, Hill EF, Blus LJ: Case histories of bald eagles and other raptors killed by organophosphorus insecticides topically applied to livestock. J Wildl Dis. 1987 Apr;23(2):292-5. Brain cholinesterase (ChE) activity was first determined, then if ChE was depressed greater than or equal to 50%, stomach and/or crop contents were evaluated for anti-ChE compounds. |
1(0,0,0,1) | Details |
| 17448116 | Fidan H, Sahin O, Ela Y, Kilbas A, Bas O, Yavuz Y, Sahin DA, Altuntas I: Influence of different atropine therapy strategies on fenthion-induced organ dysfunction in rats. Basic Clin Pharmacol Toxicol. 2007 May;100(5):308-15. Pseudocholinesterase and aspartate aminotransferase and alanine aminotransferase levels and histopathological markers of lung, brain and liver were studied. |
1(0,0,0,1) | Details |
| 8308950 | Buckley NA, Dawson AH, Whyte IM: Organophosphate poisoning: peripheral vascular resistance--a measure of adequate atropinization. J Toxicol Clin Toxicol. 1994;32(1):61-8. One patient, who had taken fenthion, also had a significantly delayed peak and prolonged, 2-3 week, systemic toxicity. We propose that stability of the plasma cholinesterase at 6 to 8 h after temporarily suspending oxime provides a rapid guide to the duration of therapy, especially in patients whose complications make clinical assessment difficult. |
1(0,0,0,1) | Details |
| 8171435 | Tandon P, Padilla S, Barone S Jr, Pope CN, Tilson HA: Fenthion produces a persistent decrease in muscarinic receptor function in the adult rat retina. Toxicol Appl Pharmacol. 1994 Apr;125(2):271-80. The animals were killed 4, 14, or 56 days after treatment and cholinesterase (ChE) activity as well as muscarinic receptor (mChR) function measured in the retina and frontal cortex. |
1(0,0,0,1) | Details |
| 941389 | Ignatov M: [Changes in some blood biochemical indices in chickens poisoned with Thimet and Letaycide]. Vet Med Nauki. 1976;13(3):14-20. The treated birds were kept under observation in the course of 72 days recording their cholinesterase activity (ChEA), serumglutamatoxal- transaminase (SGOT), serumglutamatpyruvate transaminase (SGPT), and alkaline phosphatase (APh), and blood sugar level. |
1(0,0,0,1) | Details |
| 4007878 | Misra UK, Nag D, Misra NK, Mehra MK, Ray PK: Some observations on the macula of pesticide workers. Hum Toxicol. 1985 Mar;4(2):135-45. To study the retinal changes in occupationally exposed pesticide workers, 79 subjects exposed to an organophosphate, fenthion, and 18 exposed to an organochlorine pesticide DDT [1,1,1-trichloro-2,2-bis (p-chlorophenyl) ethane], were subjected to a detailed study, including history taking, physical examination and ophthalmic evaluation. Serum cholinesterase level in 22 workers and serum DDT residue in 17 workers of the respective groups were also estimated. |
1(0,0,0,1) | Details |
| 19479785 | Uner N, Sevgiler Y, Piner P: Tissue-specific in vivo inhibition of cholinesterases by the organophosphate fenthion in Oreochromis niloticus. Environ Toxicol. 2009 May 28. |
87(1,1,2,2) | Details |
| 8171444 | Christenson WR, Van Goethem DL, Schroeder RS, Wahle BS, Dass PD, Sangha GK, Thyssen JH: Interlaboratory cholinesterase determinations and the effect on the results of statistical evaluation of cholinesterase inhibition. Toxicol Lett. 1994 Apr;71(2):139-50. RChe, PChe, and BChe from animals exposed to fenthion, a known cholinesterase inhibitor, were determined at 8 different laboratories with experience in cholinesterase determination. |
86(1,1,1,6) | Details |
| 6128165 | Eastin WC Jr, Fleming WJ, Murray HC: Organophosphate inhibition of avian salt gland Na, K-ATPase activity. Comp Biochem Physiol C. 1982;73(1):101-7. Fenthion generally reduced plasma and brain cholinesterase activity and depressed cholinesterase and Na, K-ATPase activities in salt glands of birds drinking saltwater. |
81(1,1,1,1) | Details |
| 1241238 | Imai H: [Toxicity of organophorous pesticide (fenthion) on the retina. Nippon Ganka Gakkai Zasshi. 1975 Aug;79(8):1067-76. Correlative study especially on its residue actions on the retina, liver and blood cholinesterase activities and on electroretinogram (author's transl)] |
1(0,0,0,1) | Details |
| 11097804 | Kim YA, Lee HS, Park YC, Lee YT: A convenient method for oxidation of organophosphorus pesticides in organic solvents. Environ Res. 2000 Nov;84(3):303-9. Since organophosphorus pesticides can be oxidized to oxons in vivo and in the environment and their determination based on inhibition of cholinesterases can be more sensitive after their oxidation to oxons, development of an efficient method for their in vitro oxidation is important for their toxicological and analytical studies. All reactions gave the respective oxons as single major product, except that of fenthion, which gave two major products, the respective oxon and another product from further oxidation of the oxon. |
1(0,0,0,1) | Details |
| 9891917 | Astroff AB, Young AD: The relationship between maternal and fetal effects following maternal organophosphate exposure during gestation in the rat. Toxicol Ind Health. 1998 Nov-Dec;14(6):869-89. This study compared the maternal and fetal effects, including cholinesterase inhibition, following gestational exposure to six organophosphates: tribufos, oxydemeton-methyl, azinphos-methyl, fenamiphos, isofenphos, and fenthion in the Sprague-Dawley rat. |
36(0,1,1,6) | Details |
| 1886875 | Lavandero S, Neira M, Lopez C, Gallardo R, Guerrero E, Rutman M: Research note: ability of fenthion to increase gizzard erosion in broiler chicks. Poult Sci. 1991 Jul;70(7):1633-6. Fenthion, an irreversible cholinesterase inhibitor, was used to study the role of the cholinergic system on the development of gizzard erosion. |
32(0,1,1,2) | Details |
| 10509433 | Tuler SM, Bowen JM: Chronic fenthion toxicity in laying hens. Vet Hum Toxicol. 1999 Oct;41(5):302-7. Inhibition of serum cholinesterase and brain acetylcholinesterase was greater in the high-dose hens. |
1(0,0,0,1) | Details |
| 1697627 | Yoshikawa H, Yoshida M, Hara I: Electroretinographic changes induced by organophosphorus pesticides in rats. J Toxicol Sci. 1990 May;15(2):87-95. Male Wistar rats were intraperitoneally injected with fenthion, chlorpyrifos, fenitrothion, dichlorvos or chlorfenvinphos at doses of 0.01 mmol/kg and/or 0.05 mmol/kg. The electroretinogram (ERG) was recorded at 5 hours and 2 days after the administration, and brain and retinochoroid cholinesterase (ChE) activities was assayed at 3 days after the injections. |
1(0,0,0,1) | Details |
| 985225 | Stone BF, Wilson JT, Youlton NJ: Linkage and dominance characteristics of genes for resistance to organophosphorus acaricides and allelic inheritance of decreased brain cholinesterase activity in three strains of the cattle tick, Boophilus microplus. Aust J Biol Sci. 1976 Jul;29(3):251-63. A very similar mode of inheritance of fenthion resistance in strain B has now been demonstrated with no departure in degree of dominance of resistance from the mean value of +0-57 common to these strains exposed to these chemicals. |
1(0,0,0,1) | Details |
| 8357910 | Kojima T, Tsuda S, Shirasu Y: Inhibitory effect of fenthion and diazinon on the contraction of rat aorta, and its contribution to lethality. J Vet Med Sci. 1993 Jun;55(3):383-5. Fenthion and diazinon, P = S type organothiophosphates which are precursors of cholinesterase inhibitors, cause remarkable atropine-insensitive hypotension in rats when administered intravenously in lethal doses. |
31(0,1,1,1) | Details |
| 6497117 | Clemmons RM, Meyer DJ, Sundlof SF, Rappaport JJ, Fossler ME, Hubbell J, Dorsey-Lee MR: Correction of organophosphate-induced neuromuscular blockade by Am J Vet Res. 1984 Oct;45(10):2167-9. Dogs exposed to topical organophosphate (fenthion) developed decreased plasma and muscle cholinesterase activities. |
8(0,0,1,3) | Details |
| 7175987 | Mahieu P, Hassoun A, Van Binst R, Lauwerys R, Deheneffe Y: Severe and prolonged poisoning by fenthion. J Toxicol Clin Toxicol. 1982 Jul;19(5):425-32. Correlation studies between clinical signs and plasma and erythrocyte cholinesterase activities are presented together with plasma fenthion levels and the anticholinesterase capacity of the patient's plasmas. |
8(0,0,1,3) | Details |
| 16243090 | Eddleston M, Eyer P, Worek F, Mohamed F, Senarathna L, von Meyer L, Juszczak E, Hittarage A, Azhar S, Dissanayake W, Sheriff MH, Szinicz L, Dawson AH, Buckley NA: Differences between organophosphorus insecticides in human self-poisoning: a prospective cohort study. Lancet. 2005 Oct 22-28;366(9495):1452-9. METHODS: We prospectively studied 802 patients with chlorpyrifos, dimethoate, or fenthion self-poisoning admitted to three hospitals. Blood cholinesterase activity and insecticide concentration were measured to determine the compound and the patients' response to insecticide and therapy. |
1(0,0,0,1) | Details |
| 8511793 | Veronesi B, Ehrich M: Differential cytotoxic sensitivity in mouse and human cell lines exposed to organophosphate insecticides. Toxicol Appl Pharmacol. 1993 Jun;120(2):240-6. Baseline activities of the major target esterases, i.e., cholinesterase, carboxylesterase, and neurotoxic esterase, were assayed in mouse and several human neural candidate cell lines. IC50 data indicated that the tested mouse cell line was consistently more sensitive than the human cell line to equimolar doses of various OP compounds (e.g., mipafox, parathion, paraoxon, DFP, leptophos oxon, fenthion, and fenitrothion). |
1(0,0,0,1) | Details |
| 12046724 | Tsatsakis AM, Bertsias GK, Liakou V, Mammas IN, Stiakakis I, Tzanakakis GN: Severe fenthion intoxications due to ingestion and inhalation with survival outcome. Hum Exp Toxicol. 2002 Jan;21(1):49-54. Different concentrations of pralidoxime were added to the first patient's poisoned serum in order to assess in vitro the effect of pralidoxime on cholinesterase reactivation. |
1(0,0,0,1) | Details |
| 1645823 | De Wilde V, Vogelaers D, Colardyn F, Vanderstraeten G, Van den Neucker K, De Bleecker J, De Reuck J, Van den Heede M: Postsynaptic neuromuscular dysfunction in organophosphate induced intermediate syndrome. Klin Wochenschr. 1991 Feb 26;69(4):177-83. Cholinesterase activity in the blood, plasma and red cells was monitored daily by the method according to Nenner and serial serum fenthion levels were measured by capillary gas chromatography. |
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| 9004458 | Sakaguchi K, Nagayama M, Masaoka T, Nishimura A, Kageyama K, Shirai M, Akahori F: Effects of fenthion, isoxathion, dichlorvos and propaphos on the serum cholinesterase isoenzyme patterns of dogs. Vet Hum Toxicol. 1997 Feb;39(1):1-5. |
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| 7991222 | De Bleecker J, Van den Abeele K, De Reuck J: Electromyography in relation to end-plate acetylcholinesterase in rats poisoned by different organophosphates. Neurotoxicology. 1994 Summer;15(2):331-40. Various types of impairment of neuromuscular transmission coexist, probably to a different extent at distinct stages of anticholinesterase poisoning. We investigated in vivo the relationship between clinical, histochemical and electromyographic (EMG) parameters in rats at various stages of poisoning by paraoxon or fenthion. |
1(0,0,0,1) | Details |
| 18259962 | Ela Y, Fidan H, Sahin O, Kilbas A, Bas O, Yavuz Y, Kucuker H, Altuntas I: Influence of atropine therapy on fenthion-induced pancreatitis. . Clin Toxicol. 2008 Feb;46(2):141-5. Pseudo-cholinesterase and lipase concentrations and histopathological markers of pancreatitis were studied. |
1(0,0,0,1) | Details |
| 1898122 | Hunt KA, Bird DM, Mineau P, Shutt L: Secondary poisoning hazard of fenthion to American kestrels. Arch Environ Contam Toxicol. 1991 Jul;21(1):84-90. Brain cholinesterase (ChE) activity in kestrels was depressed to levels diagnostic of poisoning by a ChE-inhibiting compound. |
1(0,0,0,1) | Details |
| 16914134 | Edwards JW, Lee SG, Heath LM, Pisaniello DL: Worker exposure and a risk assessment of malathion and fenthion used in the control of Mediterranean fruit fly in South Australia. Environ Res. 2007 Jan;103(1):38-45. Epub 2006 Aug 17. |
0(0,0,0,0) | Details |
| 7224673 | Fleming WJ: Recovery of brain and plasma cholinesterase activities in ducklings exposed to organophosphorus pesticides. Arch Environ Contam Toxicol. 1981;10(2):215-29. Brain and plasma cholinesterase (ChE) activities were determined for mallard ducklings (Anas platyrhynchos) exposed to dicrotophos and fenthion. |
7(0,0,1,2) | Details |
| 9554058 | Sakaguchi K, Akahori F, Shirai M, Masaoka T, Arishima K, Kounenis G: Effect of combined fenthion and cimetidine use in rats on lethality, blood cholinesterase activities, and serum cholinesterase isoenzymes. Vet Hum Toxicol. 1998 Apr;40(2):77-82. |
7(0,0,1,2) | Details |
| 3983971 | Misra UK, Nag D, Bhushan V, Ray PK: Clinical and biochemical changes in chronically exposed organophosphate workers. Toxicol Lett. 1985 Feb-Mar;24(2-3):187-93. Health effects of occupational organophosphate exposure were investigated by subjecting 22 workers chronically exposed to an organophosphate pesticide, fenthion (O,O-dimethyl-O-(4-methylmercapto-3-methylphenyl)-phosphorothioate) to clinical evaluation, estimation of serum cholinesterase, serum alkaline phosphatase (SAP), serum glutamic oxaloacetic transaminase (SGOT) and serum glutamic pyruvic transaminase (SGPT). |
7(0,0,1,2) | Details |
| 1295583 | Brunetto MR, Burguera JL, Burguera M, Villegas F, Bastidas C: Observation on a human intentional poisoning case by the organophosphorus insecticide fenthion. Invest Clin. 1992;33(3):89-94. |
0(0,0,0,0) | Details |
| 10220078 | Lifshitz M, Shahak E, Sofer S: and organophosphate poisoning in young children. Pediatr Emerg Care. 1999 Apr;15(2):102-3. The agents were identified as methomyl or aldicarb, and the organophosphate as parathion, fenthion, malathion, and diazinon. |
0(0,0,0,0) | Details |
| 3190448 | Misra UK, Nag D, Khan WA, Ray PK: A study of nerve conduction velocity, late responses and neuromuscular synapse functions in organophosphate workers in India. Arch Toxicol. 1988;61(6):496-500. Fenthion exposure was monitored by serum acetyl cholinesterase (AchE) levels. |
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| 6730296 | Sundlof SF, Clemmons RM, Meyer DJ, Rappaport JJ, Fossler ME, Hubbell J: Cholinesterase activity in plasma, red blood cells, muscle, and brain of dogs following repeated exposure to Spotton (fenthion). Vet Hum Toxicol. 1984 Apr;26(2):112-7. |
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| 94259 | Hart RJ, Cavey WA, Moore B, Strong MB: Efficiency and safety of methidathion applied as a pour-on systemic insecticide for control of cattle lice. Aust Vet J. 1979 Dec;55(12):575-9. Treatment with fenthion at 50 mg/kg, 7.4 times the average recommended rate, famphur at 75 mg/kg, 3 times the average rate and chlorpyrifos at 85 mg/kg, 5 times average rate, caused reductions in whole blood cholinesterase activity of 52%, 27% and 47% respectively which were similar to the reductions in cholinesterase activity found in calves treated with methidathion at similar levels above the recommended commercial dose rates. |
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