| PubMed |
Abstract |
RScore(About this table) |
| 10685507 |
Attia AM: Possible involvement of beta-adrenergic receptors in the enhancement of nocturnal pineal N-acetyltransferase activity due to parathion administration. Toxicology. 2000 Jan 3;142(2):79-86.
|
7(0,0,1,2) |
Details |
| 1714339 |
Attia AM, Reiter RJ, Stokkan KA, Mostafa MH, Soliman SA, el-Sebae AK: Parathion (O,O-dimethyl-O-p-nitrophenyl phosphorothioate) induces pineal melatonin synthesis at night. Brain Res Bull. 1991 Apr;26(4):553-7.
The effects of parathion on male rat pineal N-acetyltransferase (NAT) activity, hydroxyindole-O-methyltransferase (HIOMT) activity and pineal and serum melatonin levels at the end of light period (2000 h) and at night (2300 h and 0100 h) were studied. |
6(0,0,1,1) |
Details |
| 7716784 |
Attia AM, Mostafa MH, Richardson BA, Reiter RJ: Changes in nocturnal pineal indoleamine metabolism in rats treated with parathion are prevented by beta-adrenergic antagonist administration. Toxicology. 1995 Mar 31;97(1-3):183-9.
Parathion, an organophosphorous insecticide, was previously shown to enhance the nighttime rise in pineal N-acetyltransferase (NAT) activity and serum melatonin levels. |
6(0,0,1,1) |
Details |