| Name | myeloperoxidase |
|---|---|
| Synonyms | 38 kDa MYELOPEROXIDASE; MPO; Myeloperoxidase; Myeloperoxidase precursor; Peroxidase (Myeloperoxidase); Myeloperoxidases; Myeloperoxidase precursors; Peroxidase (Myeloperoxidase)s |
| Name | eugenol |
|---|---|
| CAS | 2-methoxy-4-(2-propen-1-yl)phenol |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 9525278 | Bodell WJ, Ye Q, Pathak DN, Pongracz K: Oxidation of eugenol to form DNA adducts and role of methide derivative in DNA adduct formation. Carcinogenesis. 1998 Mar;19(3):437-43. In vitro activation of eugenol with either horseradish peroxidase or myeloperoxidase and H2O2 produced three DNA adducts that were inhibited by the addition of either or by 66 and 90%, respectively. |
87(1,1,2,2) | Details |
| 18215676 | Chen DC, Lee YY, Yeh PY, Lin JC, Chen YL, Hung SL: Eugenol inhibited the antimicrobial functions of neutrophils. . J Endod. 2008 Feb;34(2):176-80. In addition, through the suppression of the extracellular release of myeloperoxidase and the intracellular production of reactive species, eugenol at sufficient concentrations impaired the activation of neutrophils by cytochalasin B and -- (CB/fMLP). |
81(1,1,1,1) | Details |
| 20036707 | Yogalakshmi B, Viswanathan P, Anuradha CV: Investigation of antioxidant, anti-inflammatory and DNA-protective properties of eugenol in thioacetamide-induced liver injury in rats. Toxicology. 2010 Feb 9;268(3):204-12. Epub 2009 Dec 29. Markers of liver injury transaminase, transaminase, alkaline phosphatase, gamma-glutamyl transferase and inflammation (myeloperoxidase, tumor necrosis factor-alpha and interleukin-6), oxidative stress (lipid peroxidation indices, protein carbonyl and antioxidant status) and cytochrome P4502E1 activity were assessed. |
1(0,0,0,1) | Details |
| 19769960 | Kar Mahapatra S, Chakraborty SP, Majumdar S, Bag BG, Roy S: Eugenol protects -induced mediated oxidative damage in murine peritoneal macrophages in vitro. Eur J Pharmacol. 2009 Nov 25;623(1-3):132-40. Epub 2009 Sep 19. There was a significant increase in the level of radical generation, oxidase and myeloperoxidase activity, lipid, protein, DNA damage and level in -treated group, which were significantly reduced by eugenol and supplementation. |
31(0,1,1,1) | Details |