| Name | PARP |
|---|---|
| Synonyms | COL11A2; PARP; Collagen alpha 2(XI) chain; Collagen alpha 2(XI) chain precursor; Collagen type XI alpha 2; Collagen type XI alpha 2 isoform 1; DFNA13; DFNB53… |
| Name | sodium azide |
|---|---|
| CAS | sodium azide |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 7786036 | Mizumoto K, Farber JL: Growth inhibition and cell killing by N-methyl-N-nitrosourea: metabolic alterations that accompany poly (ADP-ribosyl) ation. Arch Biochem Biophys. 1995 Jun 1;319(2):512-8. The effect of inhibition of poly (ADP- polymerase (PARP) on the growth arrest and cell killing induced by N-methyl-N-nitrosourea (MNU) was studied in L929 fibroblasts. |
3(0,0,0,3) | Details |
| 14687485 | Zhao J, Schmid-Kotsas A, Gross HJ, Gruenert A, Bachem MG: Sensitivity and specificity of different staining methods to monitor apoptosis induced by oxidative stress in adherent cells. Chin Med J. 2003 Dec;116(12):1923-9. METHODS: Sensitivity and specificity of several common methods for apoptosis determination were evaluated (Apo2.7-expression, Annexin V-binding, TUNEL-reaction, poly-(ADP--polymerase-(PARP) cleavage and single-stranded-DNA (ssDNA) staining). Apoptosis was induced by oxidative stress generated by peroxide in 3 cultured cells types growing as adherent monolayer (MiaPaCa-2, Hep-G2 and human skin fibroblasts), necrosis was induced by depletion of cellular ATP using sodium azide. |
3(0,0,0,3) | Details |
| 9987014 | Joashi UC, Greenwood K, Taylor DL, Kozma M, Mazarakis ND, Edwards AD, Mehmet H: Poly (ADP polymerase cleavage precedes neuronal death in the hippocampus and cerebellum following injury to the developing rat forebrain. Eur J Neurosci. 1999 Jan;11(1):91-100. Here, we have investigated the proteolytic cleavage of poly (ADP polymerase (PARP) and the occurrence of apoptosis in the hippocampus and the cerebellum following either HI or traumatic brain injury. |
2(0,0,0,2) | Details |
| 18948185 | Woo HD, Kim BM, Kim YJ, Lee YJ, Kang SJ, Cho YH, Choi JY, Chung HW: prevents necrotic cell death induced by co-exposure to benzo (a) pyrene and UVA radiation. Toxicol In Vitro. 2008 Dec;22(8):1840-5. Epub 2008 Oct 7. Intracellular reactive species (ROS) levels were increased significantly in the co-exposed cells, and treatment with the polyphenol but not with sodium azide or decreased ROS levels and increased cell viability in BaP/UVA-treated cells. |
0(0,0,0,0) | Details |