| Name | beta adrenoceptor (protein family or complex) |
|---|---|
| Synonyms | Beta adrenoceptor; Beta adrenoceptor; Beta adrenergic receptor; Beta adrenergic receptors; Beta adrenoceptor; Beta adrenoceptors; Beta adrenoceptors |
| Name | methyl bromide |
|---|---|
| CAS | bromomethane |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 3025415 | Woodman OL, Constantine JW, Vatner SF: Nifedipine attenuates both alpha-1 and alpha-2 adrenoceptor-mediated pressor and vasoconstrictor responses in conscious dogs and primates. J Pharmacol Exp Ther. 1986 Dec;239(3):648-53. (NE), a mixed alpha-1 and alpha-2 adrenoceptor agonist, (PE) and methoxamine (M), selective alpha-1 adrenoceptor agonists, and B-HT 920, a selective alpha-2 adrenoceptor agonist, were injected i.v. after ganglionic (hexamethonium), beta adrenoceptor and muscarinic receptor (atropine methyl bromide) blockade. |
81(1,1,1,1) | Details |
| 6675648 | Ludbrook J, Graham WF: Mechanical effects of right atrial pressure on heart rate in the conscious rabbit. Aust J Exp Biol Med Sci. 1983 Dec;61 ( Pt 6):675-85. Then, in addition, the main neurohumoral effects on the cardiac pacemaker were eliminated by three different pharmacologic treatments: (1) the cardiac nerves were blocked by instilling 2% procaine into the pericardial sac, (2) cardiac beta-adrenoceptors and cholinergic receptors were blocked with intravenous and hyoscine methyl bromide, (3) the above treatments were combined with autonomic ganglion-blockade by intravenous pentolinium. |
31(0,1,1,1) | Details |
| 2884351 | Lew MJ, Ludbrook J, Pavia JM, Quail AW, Rutter PC: Selective manipulation of neurohumoral control of the cardiac pacemaker by drugs given intrapericardially. J Pharmacol Methods. 1987 Apr;17(2):137-48. A technique of intrapericardial administration of beta-adrenoceptor and muscarinic cholinergic receptor antagonist drugs has been tested in conscious rabbits. Intrapericardial hyoscine methyl bromide (10 micrograms/kg) abolished baroreflex vagal effects on heart rate as effectively as did the conventional, 5-fold greater, intravenous dose. |
2(0,0,0,2) | Details |
| 649464 | Maher JT, Deniiston JC, Wolfe DL, Cymerman A: Mechanism of the attenuated cardiac response to beta-adrenergic stimulation in chronic hypoxia. J Appl Physiol. 1978 May;44(5):647-51. No significant group differences in cardiac frequency and various indices of myocardial performance (peak dP/dt, time-to-peak dP/dt, Vmax) were demonstrable either before or after cholinergic blockade with intravenous atropine methyl bromide, 1 mg/kg. Thus, the attenuation in cardiac responsiveness to beta-adrenoceptor stimulation in chronic hypoxia appears unrelated to the level of vagal activity, but may be attributable to enhanced enzymatic inactivation of catecholamines. |
1(0,0,0,1) | Details |
| 10535685 | Abdel-Rahman AA: Gender difference in baroreflex-mediated bradycardia in young rats: role of cardiac sympathetic and parasympathetic components. Can J Physiol Pharmacol. 1999 May;77(5):358-66. Blockade of cardiac muscarinic receptors with atropine methyl bromide elicited greater attenuation of BRS (PE) in male than in female rats (72+/-4.6 vs. 53+/-6.7% inhibition; p < 0.01) and abolished the gender difference. In male rats cardiac muscarinic blockade attenuated BRS (PE) significantly more than did cardiac beta-adrenergic receptor blockade with (72+/-4.6 vs. 43+/-2.7; p < 0.01), which suggests greater dependence of BRS (PE) on the parasympathetic component. |
1(0,0,0,1) | Details |
| 6167811 | McRitchie RJ, Chalmers JP: Paradoxical inotropic effects of clonidine and labetalol in the conscious rabbit. J Cardiovasc Pharmacol. 1981 Jul-Aug;3(4):818-27. Following cardiac autonomic blockade with and methyl-bromide, both clonidine and labetalol produce a positive inotropic response. |
0(0,0,0,0) | Details |