| Name | tumor necrosis factor alpha |
|---|---|
| Synonyms | Cachectin; DIF; TNF; TNF alpha; TNF a; TNFA; TNFSF 2; TNFSF2… |
| Name | MAA |
|---|---|
| CAS | methylarsonic acid |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 14649623 | McComsey G, Maa JF: Host factors may be more important than choice of antiretrovirals in the development of lipoatrophy. AIDS Read. 2003 Nov;13(11):539-42 CD4 cell count and plasma tumor necrosis factor (TNF), soluble TNF receptors, and leptin levels were assessed and correlated with the development of lipoatrophy. |
1(0,0,0,1) | Details |
| 15869794 | Leu TH, Charoenfuprasert S, Yen CK, Fan CW, Maa MC: Lipopolysaccharide-induced c-Src expression plays a role in and TNFalpha secretion in macrophages. 559. |
2(0,0,0,2) | Details |
| 11558258 | Petrova EE, Valiakina TI, Khaidukov SV, Nesmeianov VA: [Endogenous Tumor necrosis factor alpha unmasks the binding sites for N-acetylglucosaminyl-(beta1-4)-N-acetylmuramyl-alanyl-D-isoglutamine on the surface of melanoma cells]. Bioorg Khim. 2001 Jul-Aug;27(4):249-56. |
2(0,0,0,2) | Details |
| 9548797 | Sakurai T, Kaise T, Matsubara C: Inorganic and methylated arsenic compounds induce cell death in murine macrophages via different mechanisms. BMC Anesthesiol. 2005 Apr 12;5(1):3. The inorganic arsenicals also induced marked release of an inflammatory cytokine, tumor necrosis factor alpha (TNF alpha), at cytotoxic doses. |
2(0,0,0,2) | Details |
| 2506138 | Maio M, Gulwani B, Morgano A, Ferrone S: Differential modulation by tumor necrosis factor and immune interferon of HLA class-II antigens expressed by melanoma cells. Int J Cancer. 1989 Sep 15;44(3):554-9. |
2(0,0,0,2) | Details |
| 18832572 | Matta B, Jha P, Bora PS, Bora NS: Tolerance to -associated antigen in autoimmune uveitis is mediated by CD4+CD25+ T-regulatory cells. Am J Pathol. 2008 Nov;173(5):1440-54. Epub 2008 Oct 2. Expression levels of both interleukin-10 and transforming growth factor-beta2 were elevated whereas reduced levels of tumor necrosis factor-alpha, interferon-gamma, and interleukin-2 were detected in tolerance-induced animals. |
1(0,0,0,1) | Details |
| 17659698 | Zhang HY, Han DW, Zhao ZF, Liu MS, Wu YJ, Chen XM, Ji C: Multiple pathogenic factor-induced complications of cirrhosis in rats: a new model of hepatopulmonary syndrome with intestinal endotoxemia. World J Gastroenterol. 2007 Jul 7;13(25):3500-7. In rats with cirrhosis, there were a decreased PaO (2) and PaCO (2) in arterial blood, markedly decreased arterial O (2) content, a significantly increased alveolar to arterial gradient, an increased number of bacterial translocated within mesenteric lymph node, a significant higher level of LPS and tumor necrosis factor-alpha (TNF-alpha) in plasma, and a significant greater ratio of Tc99m-MAA brain-over-lung radioactivity. |
1(0,0,0,1) | Details |
| 15641147 | Zhang HY, Han DW, Wang XG, Zhao YC, Zhou X, Zhao HZ: Experimental study on the role of endotoxin in the development of hepatopulmonary syndrome. World J Gastroenterol. 2005 Jan 28;11(4):567-72. Supernatant was harvested at 3 h after treatment with LPS for measurement of tumor necrosis factor-alpha (TNF-alpha). |
1(0,0,0,1) | Details |
| 9863543 | Hill GE, Miller JA, Baxter BT, Klassen LW, Duryee MJ, Tuma DJ, Thiele GM: Association of malondialdehyde- (MAA) adducted proteins with atherosclerotic-induced vascular inflammatory injury. Atherosclerosis. 1998 Nov;141(1):107-16. Atherosclerosis is a vascular injury characterized by elevated tissue levels of tumor necrosis factor-alpha (TNF-alpha), increased expression of endothelial cell adhesion molecules, and vascular wall inflammatory cell infiltration. |
1(0,0,0,1) | Details |
| 15826301 | Thiele GM, Hill GE, Pavlik JA, Freeman TL, Tuma DJ, Duryee MJ, Klassen LW: Halothane potentiates the -adduct induced TNF-alpha release in heart endothelial cells. Mol Immunol. 2006 Feb;43(4):308-16. METHODS: We have recently demonstrated that the MAA-adduct induces tumor necrosis factor-alpha (TNF-alpha) release by heart endothelial cells (HECs). |
1(0,0,0,1) | Details |
| 15608611 | Duryee MJ, Klassen LW, Freeman TL, Willis MS, Tuma DJ, Thiele GM: Lipopolysaccharide is a cofactor for malondialdehyde- adduct-mediated cytokine/chemokine release by rat sinusoidal liver endothelial and Kupffer cells. Chem Res Toxicol. 1998 Apr;11(4):273-83. Cells were stimulated with media alone, bovine serum albumin (Alb), or MAA-modified Alb (MAA-Alb) in the presence or absence of LPS 1 ng/ml, and the supernatants were assayed by enzyme-linked immunosorbent assay for tumor necrosis factor alpha, macrophage chemotactic protein 1, and macrophage inhibitory protein. |
1(0,0,0,1) | Details |
| 10561459 | Rapoport E, Pendu JL: Glycosylation alterations of cells in late phase apoptosis from colon carcinomas. Glycobiology. 1999 Dec;9(12):1337-45. These changes occurred irrespective of the mode of apoptosis induction since similar results were obtained after UV, TNFalpha, or anti-Fas treatment. |
1(0,0,0,1) | Details |
| 11008078 | Aiba M, Takeyoshi I, Sunose Y, Iwazaki S, Tsutsumi H, Ohwada S, Tomizawa N, Oriuchi N, Matsumoto K, Morishita Y: FR167653 ameliorates pulmonary damage in ischemia-reperfusion injury in a canine lung transplantation model. J Heart Lung Transplant. 2000 Sep;19(9):879-86. BACKGROUND: Interleukin (IL)-1 and tumor necrosis factor-alpha (TNF-alpha) are recognized as important factors in ischemia-reperfusion (I/R) injury. |
1(0,0,0,1) | Details |