| Name | glucose 6 phosphatase |
|---|---|
| Synonyms | G 6 Pase; G6PC; G6PT; G6Pase; GSD1a; Glucose 6 phosphatase; Glucose 6 phosphatase hydrolytic subunit; G6Pases… |
| Name | sodium arsenite |
|---|---|
| CAS | sodium arsenenite |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 18505831 | Chanda D, Kim SJ, Lee IK, Shong M, Choi HS: Sodium arsenite induces orphan nuclear receptor SHP gene expression via AMP-activated protein kinase to inhibit gluconeogenic enzyme gene expression. Am J Physiol Endocrinol Metab. 2008 Aug;295(2):E368-79. Epub 2008 May 27. Sodium arsenite inhibited the promoter activity of PEPCK and G6Pase, and this repression was abolished by small interfering (si) RNA SHP treatments. |
169(2,2,3,4) | Details |
| 15198077 | Pal S, Chatterjee AK: Protective effect of against arsenic-induced depletion in vivo of carbohydrate. Drug Chem Toxicol. 2004 May;27(2):179-89. Arsenic (as sodium arsenite) treatment (i.p.) of male Wistar rats (120-140 g b.w.) at a dose of 5.55 mg/kg body weight (35% of LD50) per day for a period of 30 days produced a significant decrease in blood level (hypoglycemia) and a fall in liver and contents. Arsenic also enhanced the liver lactate dehydrogenase activity whereas glucose 6-phosphatase activity in both liver and kidney decreased significantly following arsenic treatment. |
2(0,0,0,2) | Details |
| 15664430 | Pal S, Chatterjee AK: Prospective protective role of against arsenic-induced metabolic toxicity in Wistar rats. Toxicology. 2005 Mar 1;208(1):25-33. Arsenic treatment (as sodium arsenite) of male Wistar rats (weighing 130-150 g) at a dose of 5.55 mg kg (-1) body weight (equivalent to 35% of LD (50)) (i.p.) per day for a period of 30 days produced hypoglycemia, with associated increased urinary excretion of and depletion of liver and contents. Glucose 6-phosphatase activity in liver tissue was also significantly decreased after arsenic treatment. |
1(0,0,0,1) | Details |