| Name | ha 1 |
|---|---|
| Synonyms | D1013901; Ha 1; HLA HA1; HMHA 1; HMHA1; HMHA1 protein; Minor histocompatibility antigen HA 1; Minor histocompatibility antigen HA1… |
| Name | sodium arsenite |
|---|---|
| CAS | sodium arsenenite |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 8425921 | Laszlo A, Li GC: Effect of amino acid analogs on the development of thermotolerance and on thermotolerant cells. J Cell Physiol. 1993 Feb;154(2):419-32. Exposure of HA-1 cells to amino acid analogs inhibited the development of thermotolerance following a mild heat shock or treatment with sodium arsenite. |
32(0,1,1,2) | Details |
| 1397103 | Ohtsuka K, Laszlo A: The relationship between hsp 70 localization and heat resistance. Exp Cell Res. 1992 Oct;202(2):507-18. The kinetics of the nuclear accumulation of hsp 70 were similar for normal HA-1 cells, their heat-resistant variants, and transiently thermotolerant cells (triggered by prior exposure to a brief heat shock or to sodium arsenite). |
32(0,1,1,2) | Details |
| 6841458 | Li GC: Induction of thermotolerance and enhanced heat shock protein synthesis in Chinese hamster fibroblasts by sodium arsenite and by J Cell Physiol. 1983 May;115(2):116-22. To test this hypothesis, I have examined the effect of sodium arsenite or treatment on protein synthesis and cell survival in Chinese hamster ovary HA-1 cells. |
6(0,0,1,1) | Details |
| 3169135 | Laszlo A: The relationship of heat-shock proteins, thermotolerance, and protein synthesis. Exp Cell Res. 1988 Oct;178(2):401-14. The relationship of heat-induced inhibition of protein synthesis (HIIPS) and thermotolerance, the transient ability to survive otherwise lethal heat treatments, was studied in HA-1 Chinese hamster fibroblasts exposed to various treatments. A mild heatshock or exposure to sodium arsenite induced a refractoriness to HIIPS, while exposure to the amino acid analog of azetidine, did not. |
2(0,0,0,2) | Details |
| 11162468 | Suzuki T, Blank V, Sesay JS, Crawford DR: Maf genes are involved in multiple stress response in human. Biochem Biophys Res Commun. 2001 Jan 12;280(1):4-8. We have previously reported that the mRNA to one of these members, mafG/adapt66, is induced by oxidative stress in hamster HA-1 cells. |
1(0,0,0,1) | Details |
| 11892989 | Chen MS, Goswami PC, Laszlo A: Differential accumulation of U14 snoRNA and hsc70 mRNA in Chinese hamster cells after exposure to various stress conditions. Cell Stress Chaperones. 2002 Jan;7(1):65-72. We have previously characterized the unique organization of the U14 small nucleolar ribonucleic acid (snoRNA) gene in Chinese hamster HA-1 cells. On the other hand, exposure to sodium arsenite or azetidine induced an increased accumulation of hsc70 mRNA, but did not lead to a concomitant increase in the level of U14 snoRNA. |
1(0,0,0,1) | Details |
| 3170639 | Lee KJ, Hahn GM: Abnormal proteins as the trigger for the induction of stress responses: heat, diamide, and sodium arsenite. J Cell Physiol. 1988 Sep;136(3):411-20. Both of these agents induced heat shock proteins and thermotolerance in CHO (HA-1) cells. |
1(0,0,0,1) | Details |
| 1383016 | Laszlo A: The thermoresistant state: protection from initial damage or better repair?. Exp Cell Res. 1992 Oct;202(2):519-31. The induction of and recovery from heat-induced perturbations in several cellular parameters were examined in normal, transiently thermotolerant, and permanently heat-resistant HA-1 Chinese hamster fibroblasts. This response was observed in cells in which transient thermotolerance was induced by either a mild heat shock or exposure to sodium arsenite. |
1(0,0,0,1) | Details |
| 6954473 | Li GC, Werb Z: Correlation between synthesis of heat shock proteins and development of thermotolerance in Chinese hamster fibroblasts. Proc Natl Acad Sci U S A. 1982 May;79(10):3218-22. Because a brief, nonlethal heat treatment can dramatically induce a transient resistance to a subsequent lethal heat treatment (thermotolerance), we examined the effect of heat treatment (41-46 degrees C) on protein synthesis and cell survival in plateau-phase Chinese hamster fibroblast (HA-1) cells. Sodium arsenite, hypoxia, and also induced both the synthesis of heat shock proteins and transient thermotolerance. |
1(0,0,0,1) | Details |