PubMed |
Abstract |
RScore(About this table) |
17576240 |
Fumagalli M, Musso T, Vermi W, Scutera S, Daniele R, Alotto D, Cambieri I, Ostorero A, Gentili F, Caposio P, Zucca M, Sozzani S, Stella M, Castagnoli C: Imbalance between activin A and follistatin drives postburn hypertrophic scar formation in human skin. Exp Dermatol. 2007 Jul;16(7):600-10.
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6(0,0,0,6) |
Details |
19074460 |
Rose FF Jr, Mattis VB, Rindt H, Lorson CL: Delivery of recombinant follistatin lessens disease severity in a mouse model of spinal muscular atrophy. Hum Mol Genet. 2009 Mar 15;18(6):997-1005. Epub 2008 Dec 12.
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4(0,0,0,4) |
Details |
19477958 |
Sumner CJ, Wee CD, Warsing LC, Choe DW, Ng AS, Lutz C, Wagner KR: Inhibition of myostatin does not ameliorate disease features of severe spinal muscular atrophy mice. Hum Mol Genet. 2009 Sep 1;18(17):3145-52. Epub 2009 May 28.
In this study, we evaluated the effects of blocking myostatin signaling in severe SMA mice (hSMN2/delta7SMN/mSmn (-/-)) by two independent strategies: (i) transgenic overexpression of the myostatin inhibitor follistatin and (ii) post-natal administration of a soluble activin receptor IIB (ActRIIB-Fc). |
1(0,0,0,1) |
Details |
20233786 |
Corpuz PS, Lindaman LL, Mellon PL, Coss D: FoxL2 Is Required for Activin Induction of the Mouse and Human Follicle-Stimulating Hormone {beta}-Subunit Genes. Mol Endocrinol. 2010 Mar 16.
We identify FoxL2 as a critical component in activin induction of FSHbeta, both for the mouse gene, induction of which is Sma- and Mad-related protein (Smad) dependent, and for the human gene that is Smad independent. |
0(0,0,0,0) |
Details |