| Name | catalase |
|---|---|
| Synonyms | CAT; Catalase; Erythrocyte derived growth promoting factor; Carnitine O acetyltransferase; Carnitine acetylase; Carnitine acetyltransferase; CAT; Catalases… |
| Name | acrylonitrile |
|---|---|
| CAS | 2-propenenitrile |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 12011484 | Zhang H, Kamendulis LM, Klaunig JE: Mechanisms for the induction of oxidative stress in Syrian hamster embryo cells by acrylonitrile. Toxicol Sci. 2002 Jun;67(2):247-55. Xanthine oxidase activity was increased following 24 and 48 h treatment with acrylonitrile. 1-aminobenzotriazole, a suicidal P450 enzyme inhibitor, attenuated the effects of acrylonitrile on catalase and xanthine oxidase in SHE cells, suggesting that P450 metabolism is required for acrylonitrile to produce its effects on these enzymes. |
32(0,1,1,2) | Details |
| 17171660 | Wang ZG, Ke BB, Xu ZK: Covalent immobilization of redox enzyme on electrospun nonwoven poly (acrylonitrile-co-acrylic acid) nanofiber mesh filled with carbon nanotubes: a comprehensive study. Biotechnol Bioeng. 2007 Jul 1;97(4):708-20. The covalent immobilization of catalase from bovine liver on the neat PANCAA nanofiber mesh or the composite one was processed in the presence of EDC/NHS. |
4(0,0,0,4) | Details |
| 20021157 | Jacob S, Ahmed AE: Acrylonitrile-induced neurotoxicity in normal human astrocytes: oxidative stress and formation. Toxicol Mech Methods. 2003;13(3):169-79. We assessed cell viability; levels of endogenous antioxidants, GSH, and catalase; levels of reactive species; and secretion of TNF- alpha, a cellular marker for oxidative stress and oxidative damage to nuclear DNA, after treatment with VCN. |
3(0,0,0,3) | Details |
| 10048137 | Jiang J, Xu Y, Klaunig JE: Induction of oxidative stress in rat brain by acrylonitrile (ACN). Toxicol Sci. 1998 Dec;46(2):333-41. Both the levels of nonenzymatic antioxidants (GSH, and the activities of enzymatic antioxidants (catalase, superoxide dismutase, peroxidase) in rat brains and livers were measured. |
2(0,0,0,2) | Details |
| 19913070 | Guangwei X, Rongzhu L, Wenrong X, Suhua W, Xiaowu Z, Shizhong W, Ye Z, Aschner M, Kulkarni SK, Bishnoi M: pretreatment protects against acute acrylonitrile-induced oxidative damage in rats. Toxicology. 2010 Jan 12;267(1-3):140-6. Epub 2009 Nov 11. These increases were accompanied by a significant decrease in (GSH) content and a significant reduction in catalase (CAT) activity in the same tissues. |
1(0,0,0,1) | Details |
| 15497133 | Godjevargova T, Dayal R, Turmanova S: production in bioreactor with immobilized oxidase plus catalase on polymer membrane adjacent to anion-exchange membrane. Macromol Biosci. 2004 Oct 20;4(10):950-6. |
2(0,0,0,2) | Details |
| 8035742 | Soleo L: [The detoxification pathways of electrophilic intermediate compounds] . Med Lav. 1994 Jan-Feb;85(1):22-36. Detoxification of the radical species occurs via cellular constituents, such as and via enzymes, such as superoxide dismutase, GSH peroxidase, catalase. For acrylonitrile the route of conjugation with is the main detoxification route. |
1(0,0,0,1) | Details |
| 17337135 | Carrera MP, Antolin I, Martin V, Sainz RM, Mayo JC, Herrera F, Garcia-Santos G, Rodriguez C: Antioxidants do not prevent acrylonitrile-induced toxicity. Toxicol Lett. 2007 Mar 30;169(3):236-44. Epub 2007 Feb 6. Additionally, we measured different parameters of oxidative stress such as catalase activity, lipid peroxidation and GSH concentration, as indicators of the potential oxidative stress mediated by the toxicity of ACN, after exposure of Wistar rats to a concentration of 200 ppm ACN for 14 days. |
1(0,0,0,1) | Details |
| 9804465 | Biasioli S, Schiavon R, Petrosino L, Cavallini L, Cavalcanti G, De Fanti E: Dialysis kinetics of and reactive species. ASAIO J. 1998 Sep-Oct;44(5):M423-32. Plasma (P) (HCY), (CYS), malondialdehyde (MDA), E- (GSH), -6-phosphodehydrogenase, glutathione reductase (GR), peroxidase (GP), catalase (CAT), and superoxide dismutase (SOD) were determined. All six membranes (Hemophan [HEMO]; cellulose [DIAC]; acrylonitrile-69 [AN69]; polymethylmethacrylate [PMMA]; cuprammonium rayon [CURAY]; polysulfone plus hemophan [PS + HEMO]) induced a significant decrease in plasma lipid peroxidation (p < 0.001) and an increase in E-GSH, GR, GR + flavinadenine dinucleotide, GP, and SOD (p < 0.001). |
1(0,0,0,1) | Details |
| 9708882 | Whysner J, Steward RE 3rd, Chen D, Conaway CC, Verna LK, Richie JP Jr, Ali N, Williams GM: Formation of 8-oxodeoxyguanosine in brain DNA of rats exposed to acrylonitrile. Arch Toxicol. 1998 Jun;72(7):429-38. |
0(0,0,0,0) | Details |
| 10426806 | Kamendulis LM, Jiang J, Xu Y, Klaunig JE: Induction of oxidative stress and oxidative damage in rat glial cells by acrylonitrile. Carcinogenesis. 1999 Aug;20(8):1555-60. Following 1 and 4 h of ACN exposure, the levels of the non-enzymatic antioxidant as well as the activities of the enzymatic antioxidants catalase and superoxide dismutase were significantly decreased in the rat glial cells. |
1(0,0,0,1) | Details |
| 18752313 | El-Sayed el-SM, Abo-Salem OM, Abd-Ellah MF, Abd-Alla GM: an antioxidant prevents acrylonitrile-induced oxidative stress in rat brain. J Biochem Mol Toxicol. 2008 Jul;22(4):268-73. In addition, ACN administration resulted in significant reductions in the enzymatic antioxidant parameters of brain; superoxide dismutase (SOD), catalase (CAT), peroxidase (GSH-Px), and glutathione-S-transferase (GST) recording 43%, 64%, 52%, and 43%, respectively. |
1(0,0,0,1) | Details |
| 4770372 | Abe K, Hiraga M, Anan FK: The chemical modification of beef liver catalase. J Biochem. 1973 Nov;74(5):889-904. The properties of acrylonitrile- or glyoxal-treated products of catalase. |
82(1,1,1,2) | Details |