| Name | kallikrein |
|---|---|
| Synonyms | KLK 1; HK1; KLK1; KLKR; Kallikrein; Kallikrein 1; Kallikrein 1 isoform 3 preproprotein; Kallikrein serine protease 1… |
| Name | diatomaceous earth |
|---|---|
| CAS | diatomite |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 1280470 | Mackie IJ, Gallimore M, Machin SJ: Contact factor proteases and the complexes formed with alpha 2-macroglobulin can interfere in protein C assays by cleaving amidolytic substrates. Blood Coagul Fibrinolysis. 1992 Oct;3(5):589-95. The protein C substrates were cleaved by purified kallikrein and alpha- and beta-FXIIa. |
2(0,0,0,2) | Details |
| 17045938 | Nielsen VG, Steenwyk BL, Gurley WQ: Contact activation prolongs clot lysis time in human plasma: role of thrombin-activatable fibrinolysis inhibitor and Factor XIII. J Heart Lung Transplant. 2006 Oct;25(10):1247-52. BACKGROUND: Contact activation system proteins (e.g., Factor XII, kallikrein) have been implicated as direct or indirect activators of plasminogen. |
1(0,0,0,1) | Details |
| 17967670 | Dong Y, Nuttall GA, Oliver WC Jr, Agarwal S, Ereth MH: Aprotinin does not prolong the Sonoclot aprotinin-insensitive activated clotting time. J Clin Anesth. 2007 Sep;19(6):424-8. (2 U/mL) and escalating concentrations of aprotinin of 160 to 500 kallikrein inhibitory units (KIU)/mL were added in vitro. |
1(0,0,0,1) | Details |
| 8638779 | Despotis GJ, Filos KS, Levine V, Alsoufiev A, Spitznagel E: Aprotinin prolongs activated and nonactivated whole blood clotting time and potentiates the effect of in vitro. Anesth Analg. 1996 Jun;82(6):1126-31. Specimens obtained from the first consecutive 20 patients were mixed with either normal saline (NS) or aprotinin (400 kallikrein inhibiting units (KIU)/mL), inserted into Hemochron tubes containing either NS or (0.3 or 0.6 U/mL) and then used to measure celite-activated (celite ACT) and nonactivated whole blood clotting time (WBCT1) using four Hemochron instruments. |
1(0,0,0,1) | Details |
| 11684986 | Avidan MS, Da Fonseca J, Parmar K, Alcock E, Ponte J, Hunt BJ: The effects of aprotinin on thromboelastography with three different activators. Anesthesiology. 2001 Nov;95(5):1169-74. Aprotinin, through inhibition of kallikrein, causes prolongation of the celite-activated clotting time and the activated partial thromboplastin ratio. |
1(0,0,0,1) | Details |