| Name | corticotropin releasing factor |
|---|---|
| Synonyms | CRF; CRH; Corticoliberin; Corticoliberin precursor; Corticotropin releasing factor; Corticotropin releasing hormone; Corticoliberins; Corticoliberin precursors… |
| Name | nicotine |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 19635511 | Kamdi SP, Nakhate KT, Dandekar MP, Kokare DM, Subhedar NK: Participation of corticotropin-releasing factor type 2 receptors in the acute, chronic and withdrawal actions of nicotine associated with feeding behavior in rats. Appetite. 2009 Dec;53(3):354-62. Epub 2009 Jul 25. |
162(2,2,2,2) | Details |
| 19429100 | Mano-Otagiri A, Iwasaki-Sekino A, Ohata H, Arai K, Shibasaki T: Nicotine suppresses energy storage through activation of sympathetic outflow to brown adipose tissue via corticotropin-releasing factor type 1 receptor. Neurosci Lett. 2009 May 8;455(1):26-9. Epub 2009 Mar 20. |
112(1,2,2,2) | Details |
| 20117222 | McKlveen JM, Wilson JM, Rubin RT, Rhodes ME: Sexually diergic, dose-dependent hypothalamic-pituitary- axis responses to nicotine in a dynamic in vitro perfusion system. J Pharmacol Toxicol Methods. 2010 Feb 1. Sampling ports between tissue baths were used to collect buffer before and after addition of various doses of nicotine, for measurement of AVP and corticotropin-releasing hormone (CRH) from the hypothalamus bath, ACTH from the pituitary bath, and CORT from the bath. |
6(0,0,1,1) | Details |
| 20202080 | Yu G, Chen H, Wu X, Matta SG, Sharp BM: Nicotine self-administration differentially modulates and transmission in hypothalamic paraventricular nucleus to enhance the hypothalamic-pituitary- response to stress. J Neurochem. 2010 Feb 25. Nicotine self-administration alters neuropeptide expression in corticotropin-releasing factor (CRF) neurons within paraventricular nucleus (PVN) and increases PVN responsiveness to during mild footshock stress. |
6(0,0,1,1) | Details |
| 19349312 | Brady KT, McRae AL, Moran-Santa Maria MM, DeSantis SM, Simpson AN, Waldrop AE, Back SE, Kreek MJ: Response to corticotropin-releasing hormone infusion in cocaine-dependent individuals. Arch Gen Psychiatry. 2009 Apr;66(4):422-30. Individuals with dependence on other substances (except or nicotine) or with major depression, posttraumatic stress disorder, bipolar disorder, or psychotic or eating disorders were excluded. |
2(0,0,0,2) | Details |
| 19878140 | Nelson EC, Agrawal A, Pergadia ML, Wang JC, Whitfield JB, Saccone FS, Kern J, Grant JD, Schrage AJ, Rice JP, Montgomery GW, Heath AC, Goate AM, Martin NG, Madden PA: H2 haplotype at chromosome 17q21.31 protects against childhood sexual abuse-associated risk for consumption and dependence. Addict Biol. 2010 Jan;15(1):1-11. Epub . Animal research supports a central role for corticotropin-releasing factor (CRF) in actions of on brain function. Here, we examine whether G x E interactions involving this haplotype and childhood sexual abuse (CSA) are associated with risk for consumption and dependence in Australian participants (n = 1128 respondents from 476 families) of the Nicotine Addiction Genetics project. |
1(0,0,0,1) | Details |
| 20028457 | Yu G, Sharp BM: Nicotine self-administration diminishes stress-induced secretion but augments adrenergic-responsiveness in the hypothalamic paraventricular nucleus and enhances adrenocorticotropic hormone and release. J Neurochem. 2010 Mar;112(5):1327-37. Epub 2009 Dec 17. Altered neuropeptide expression within corticotropin-releasing factor (CRF) neurons in the hypothalamic paraventricular nucleus (PVN) contributes to this enhanced HPA response to stress. |
1(0,0,0,1) | Details |
| 19145226 | Marcinkiewcz CA, Prado MM, Isaac SK, Marshall A, Rylkova D, Bruijnzeel AW: Corticotropin-releasing factor within the central nucleus of the amygdala and the nucleus accumbens shell mediates the negative affective state of nicotine withdrawal in rats. Neuropsychopharmacology. 2009 Jun;34(7):1743-52. Epub 2009 Jan 14. Previous research has shown that an increased central release of corticotropin-releasing factor (CRF) at least partly mediates the deficit in brain reward function associated with nicotine withdrawal in rats. In all experiments, the nicotinic receptor antagonist mecamylamine (3 mg/kg) elevated the brain reward thresholds of the nicotine-dependent rats (9 mg/kg per day of nicotine salt) and did not affect the brain reward thresholds of the saline-treated control rats. |
1(0,0,0,1) | Details |
| 19217073 | Bruijnzeel AW, Prado M, Isaac S: Corticotropin-releasing factor-1 receptor activation mediates nicotine withdrawal-induced deficit in brain reward function and stress-induced relapse. Biol Psychiatry. 2009 Jul 15;66(2):110-7. Epub 2009 Feb 12. Previous research has shown that blockade of corticotropin-releasing factor (CRF) receptors with a nonspecific CRF1/CRF2 receptor antagonist prevents the deficit in brain reward function associated with nicotine withdrawal and stress-induced reinstatement of extinguished nicotine-seeking in rats. |
1(0,0,0,1) | Details |
| 19220484 | Gangitano D, Salas R, Teng Y, Perez E, De Biasi M: modulation of alpha5 nAChR subunits influences anxiety-related behavior during estrus cycle. Genes Brain Behav. 2009 Jun;8(4):398-406. Epub 2009 Feb 11. In addition, stress-related disorders such as anxiety, post-traumatic stress syndrome and depression are often associated with chronic nicotine use. We studied the hypothalamus-pituitary- axis by measuring plasma and hypothalamic corticotropin-releasing factor. |
1(0,0,0,1) | Details |
| 19359422 | Choudhury BK, Shi XZ, Sarna SK: mediates the transcriptional effects of heterotypic chronic stress on colonic motor function. Am J Physiol Gastrointest Liver Physiol. 2009 Jun;296(6):G1238-47. Epub 2009 Apr 9. The inhibition of receptors for glucocortocoids, corticotropin-releasing hormone or nicotine also does not block the effects of heterotypic chronic stress. |
0(0,0,0,0) | Details |