| Name | guanylate cyclase |
|---|---|
| Synonyms | AMDM; Natriuretic peptide receptor; ANP B; ANPRB; Atrial natriuretic peptide B type receptor; Atrial natriuretic peptide receptor B; Atrial natriuretic peptide receptor B precursor (ANP B) (ANPRB) (GC B) (Guanylate cyclase B); Atrionatriuretic peptide receptor B… |
| Name | sodium cyanide |
|---|---|
| CAS | sodium cyanide (Na(CN)) |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 8823992 | Nakanishi H, Ariga H, Yamada Y, Ono T, Matsuoka I, Nakahata N: Antagonism of relaxing responses to nitrovasodilators by in rabbit isolated aortic preparation. Fukushima J Med Sci. 1995 Dec;41(2):125-34. These findings may indicate that inhibits soluble guanylate cyclase activated by nitroprusside, or through interaction with and heme binding site of the enzyme. Effect of sodium cyanide on the relaxing response (decrease of intraluminal pressure) to sodium nitroprusside, and human atrial natriuretic peptide (HANP) was investigated in isolated rabbit aortic preparations in vitro. |
1(0,0,0,1) | Details |
| 6149944 | Rapoport RM, Murad F: Effect of on nitrovasodilator-induced relaxation, cyclic GMP accumulation and guanylate cyclase activation in rat aorta. Eur J Pharmacol. 1984 Sep 3;104(1-2):61-70. The effects of sodium cyanide on relaxation, increases in cyclic GMP accumulation and guanylate cyclase activation induced by sodium nitroprusside and other nitrovasodilators were examined in rat thoracic aorta. |
34(0,1,1,4) | Details |
| 6127356 | Lewicki JA, Brandwein HJ, Mittal CK, Arnold WP, Murad F: Properties of purified soluble guanylate cyclase activated by and sodium nitroprusside. J Cyclic Nucleotide Res. 1982;8(1):17-25. The activation with hemin and sodium nitroprusside was prevented in a dose-dependent manner by sodium cyanide. |
4(0,0,0,4) | Details |
| 8100588 | Lonart G, Cassels KL, Johnson KM: induces -dependent [3H] release from striatal slices. J Neurosci Res. 1993 Jun 1;35(2):192-8. Sodium cyanide was used as a model compound to test the possibility that NO acted through blockade of mitochondrial electron transport. |
0(0,0,0,0) | Details |