Name | NMDA receptors (protein family or complex) |
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Synonyms | Glutamate [NMDA] receptor; Glutamate [NMDA] receptors; N methyl D aspartate receptor; N methyl D aspartate receptors; NMDA receptor; NMDA receptors |
Name | cycloheximide |
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CAS |
PubMed | Abstract | RScore(About this table) | |
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19140302 | Solntseva SV, Nikitin P: [Neurochemical mechanisms of food aversion conditioning consolidation in snail Helix lucorum]. Ross Fiziol Zh Im I M Sechenova. 2008 Nov;94(11):1259-69. Effects of cycloheximide, protein synthesis inhibitors, as well as serotonin receptor antagonist and NMDA receptor antagonist on food aversion conditioning consolidation were studied in snail Helix lucorum. |
31(0,1,1,1) | Details |
18825927 | Solntseva SV, Nikitin VP: [Neurochemical mechanisms of food aversion conditioning consolidation in snail helix lucorum]. Ross Fiziol Zh Im I M Sechenova. 2008 Aug;94(8):860-70. Effects of cycloheximide, protein synthesis inhibitor as well as serotonin receptor antagonist and NMDA receptor antagonist, on food aversion conditioning consolidation were studied in snail Helix lucorum. |
31(0,1,1,1) | Details |
19923273 | Baumbauer KM, Huie JR, Hughes AJ, Grau JW: Timing in the absence of supraspinal input II: regularly spaced stimulation induces a lasting alteration in spinal function that depends on the NMDA receptor, BDNF release, and protein synthesis. J Neurosci. 2009 Nov 18;29(46):14383-93. Administration of the protein synthesis inhibitor cycloheximide after training blocked the long-term effect. |
3(0,0,0,3) | Details |
16353388 | Wang Z, Ruan Q, Wang D: Different effects of intracochlear sensory and neuronal injury stimulation on expression of synaptic N-methyl-D-aspartate receptors in the auditory cortex of rats in vivo. Acta Otolaryngol. 2005 Nov;125(11):1145-51. |
3(0,0,0,3) | Details |
16606372 | Cossenza M, Cadilhe DV, Coutinho RN, Paes-de-Carvalho R: Inhibition of protein synthesis by activation of NMDA receptors in cultured retinal cells: a new mechanism for the regulation of production. J Neurochem. 2006 Jun;97(5):1481-93. Epub 2006 Apr 10. Although L-[3H] uptake measured in cultured chick retina cells incubated in the presence of cycloheximide (CHX, a protein synthesis inhibitor) was inhibited approximately 75% at equilibrium, quantitative thin-layer chromatography analysis showed that free intracellular L-[3H] was six times higher in CHX-treated than in control cultures. |
3(0,0,0,3) | Details |
16109729 | Vickers CA, Dickson KS, Wyllie DJ: Induction and maintenance of late-phase long-term potentiation in isolated dendrites of rat hippocampal CA1 pyramidal neurones. J Physiol. 2005 Nov 1;568(Pt 3):803-13. Epub 2005 Aug 18. Expression of (NMDA) receptor-dependent long-term potentiation (LTP) in the CA1 region of the hippocampus can be divided into an early (1-2 h), protein synthesis-independent phase and a late (> 4 h), protein synthesis-dependent phase. Incubation of the slices with the mRNA translation inhibitor cycloheximide or the mammalian target of rapamycin (mTOR) inhibitor rapamycin blocked late-LTP in both 'intact' and 'isolated' slice preparations. |
1(0,0,0,1) | Details |
19490087 | Zhang L, Meng K, Li YH, Han TZ: NR2A-containing NMDA receptors are required for L-LTP induction and depotentiation in CA1 region of hippocampal slices. Eur J Neurosci. 2009 Jun;29(11):2137-44. Epub 2009 May 21. L-LTP partial reversal was also induced by HI-PP-LFS, when the protein synthesis inhibitors anisomycin (25 microm) and cycloheximide (60 microm) were applied following LTP induction. |
2(0,0,0,2) | Details |
18784289 | Argilli E, Sibley DR, Malenka RC, England PM, Bonci A: Mechanism and time course of cocaine-induced long-term potentiation in the ventral tegmental area. J Neurosci. 2008 Sep 10;28(37):9092-100. This cocaine-induced LTP appears to be mediated via D (5) receptor activation of NMDA receptors and to require protein synthesis. |
1(0,0,0,1) | Details |
15772359 | Nosyreva ED, Huber KM: Developmental switch in synaptic mechanisms of hippocampal metabotropic glutamate receptor-dependent long-term depression. J Neurosci. 2005 Mar 16;25(11):2992-3001. Instead, mGluR-LTD at neonatal synapses results in large decreases in presynaptic function, measured by changes in paired-pulse facilitation and the rate of blockade by the use-dependent NMDA receptor blocker (+)-5-methyl-10,11-dihydro-5H-dibenzo [a,d] cyclohepten-5,10-imine |
1(0,0,0,1) | Details |
19247210 | Kim J, Park S, Lee S, Choi S: Amygdala depotentiation ex vivo requires mitogen-activated protein kinases and protein synthesis. Neuroreport. 2009 Mar 25;20(5):517-20. U0126, a mitogen-activated protein kinase inhibitor, and cycloheximide, a protein synthesis inhibitor, blocked depotentiationex vivo. |
0(0,0,0,0) | Details |