| Name | fibronectin |
|---|---|
| Synonyms | CIG; MSF; Cold insoluble globulin; FINC; FN; FN1; Fibronectin; Fibronectin 1… |
| Name | TCA |
|---|---|
| CAS | 2,2,2-trichloroacetic acid |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 18347039 | Shanks RM, Meehl MA, Brothers KM, Martinez RM, Donegan NP, Graber ML, Cheung AL, O'Toole GA: Genetic evidence for an alternative -dependent biofilm formation pathway in Staphylococcus aureus that is dependent on fibronectin binding proteins and the GraRS two-component regulatory system. Infect Immun. 2008 Jun;76(6):2469-77. Epub 2008 Mar 17. |
2(0,0,0,2) | Details |
| 14611684 | Tseng L, Tang M, Wang Z, Mazella J: Progesterone receptor (hPR) upregulates the fibronectin promoter activity in human decidual fibroblasts. DNA Cell Biol. 2003 Oct;22(10):633-40. |
2(0,0,0,2) | Details |
| 18519664 | Cervera AM, Apostolova N, Crespo FL, Mata M, McCreath KJ: Cells silenced for SDHB expression display characteristic features of the tumor phenotype. Cancer Res. 2008 Jun 1;68(11):4058-67. Of interest, SDHB-silenced cells had a greater capacity to adhere to extracellular matrix components, including fibronectin and laminin, than control cells, thus suggesting a possible mechanism of tumor initiation. |
2(0,0,0,2) | Details |
| 18061331 | Soto A, DelRaso NJ, Schlager JJ, Chan VT: exposure resulted in gene expression changes indicative of activation of fibrogenic pathways and down-regulation of energy metabolism and oxidative stress response. Toxicology. 2008 Jan 14;243(1-2):177-92. Epub 2007 Oct 23. Consistent with the fibrogenic function of the TGF-beta signaling pathway in various experimental renal diseases, genes encoding major extracellular matrix components such as collagens, laminins, fibronectin 1 and tenascins are also strongly up-regulated. |
1(0,0,0,1) | Details |
| 15756348 | Al-Aqeel AI: Al-Aqeel Sewairi syndrome, a new autosomal recessive disorder with multicentric osteolysis, nodulosis and arthropathy. Saudi Med J. 2005 Jan;26(1):24-30. RESULTS: Some affected individuals were homoallelic for a nonsense mutation (TCA> TAA) in codon 244 of exon 5, predicting the replacement of a residue by a stop codon in the first fibronectin type II domain (Y244X). |
1(0,0,0,1) | Details |