| Name | cholinesterase |
|---|---|
| Synonyms | Acylcholine acylhydrolase; BCHE; BCHE protein; Butyrylcholine esterase; Butyrylcholinesterase; CHE1; Choline esterase II; Cholinesterase… |
| Name | pirimiphos-methyl |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 9528704 | Ibrahim H, Kheir R, Helmi S, Lewis J, Crane M: Effects of organophosphorus, pyrethroid and organochlorine pesticides, and a heavy metal on survival and cholinesterase activity of Chironomus riparius Meigen. Bull Environ Contam Toxicol. 1998 Mar;60(3):448-55. |
1(0,0,0,1) | Details |
| 7154146 | Nasir SM, Ahmad N, Shah MA, Azam CM: A large-scale evaluation of pirimiphos-methyl 25% WP during 1980-1981 for malaria control in Pakistan. J Trop Med Hyg. 1982 Dec;85(6):239-44. |
0(0,0,0,0) | Details |
| 3385134 | Rajini PS, Krishnakumari MK: Toxicity of pirimiphos-methyl: II. J Environ Sci Health B. 1988 Apr;23(2):145-58. |
0(0,0,0,0) | Details |
| 11758396 | Yin D, Jin H, Yu H, Chen L: [A comparative study on the sensitivity and specificity of cholinesterase and glutathione s-transferase in Gammarus pulex L.]. Ying Yong Sheng Tai Xue Bao. 2001 Aug;12(4):615-8. Studies on the influences of lindane, pirimiphos methyl, permethrin, zinc and dodecyl linear alkybenzene sulfonate (LAS) on the activity and toxicity of cholinesterase (ChE) and glutathione s-transferase (GST) in Gammarus pulex L. showed that only pirimiphos methyl caused a change in ChE activity in Gammarus, with a significant reduction in enzyme activity after 24 h and 48 h exposure. |
32(0,1,1,2) | Details |
| 2600363 | Rajini PS, Muralidhara, Krishnakumari MK: Inhibitory pattern of tissue esterases in rats fed dietary pirimiphos-methyl. J Environ Sci Health B. 1989 Oct;24(5):509-24. Activities of Acetylcholinesterase (AChE) in brain and erythrocytes, pseudocholinesterase (PChE) in plasma and non-specific carboxylesterase (NSE) in brain, liver, plasma and kidney were assayed at weekly intervals in growing male rats fed dietary pirimiphos-methyl at dosages of 1000, and 1500 ppm for a period of 28 days. |
6(0,0,1,1) | Details |
| 1527361 | Yeboah PO, Semanhyia CB, Melfah PT: Bioavailability and biological activity of bound residues of radiolabelled pirimiphos-methyl in maize grains. J Environ Sci Health B. 1992 Aug;27(4):377-86. Also a significant reduction in blood cholinesterase activity was observed in both male and female rats fed on grain bound pirimiphos-methyl residues at two dose levels. |
81(1,1,1,1) | Details |
| 16289700 | Abdel-Halim KY, Salama AK, El-Khateeb EN, Bakry NM: Organophosphorus pollutants (OPP) in aquatic environment at Damietta Governorate, Egypt: implications for monitoring and biomarker responses. Chemosphere. 2006 Jun;63(9):1491-8. Epub 2005 Nov 14. The obtained results are in parallel to that found in case of cholinesterase activity where the activity of both acetylcholinesterase (AChE) and butyrylcholinesterase (BuChE) was declined at these seasonal period. Chlorpyrifos, chlorpyrifos-methyl, malathion, diazinon, pirimiphos-methyl and profenofos were detected in most samples. |
3(0,0,0,3) | Details |
| 4086212 | Rajini PS, Krishnakumari MK: Effect of supplementation on the toxicity of pirimiphos-methyl to albino rats. Int J Vitam Nutr Res. 1985;55(4):421-4. The biochemical assessments included estimations of brain and plasma cholinesterases, levels of in liver, kidney and adrenals, urinary levels of and |
2(0,0,0,2) | Details |
| 1496821 | Akay MT, Yilmazoglu G, Yasacan S, Turk H, Kolanyaka D: Bioavailability and toxicological potential of wheat-bound pirimiphos-methyl residues in rats. Xenobiotica. 1992 Mar;22(3):293-302. A dietary level of 7.5 ppm showed a significant decrease in serum cholinesterase activity and in lymphocytes and monocytes, and an increase in alkaline phosphatase activity and of urea 3. |
1(0,0,0,1) | Details |
| 2588705 | Goedicke HJ, Hermes H, Wagner R: [Exposure to residues on plant surfaces following the use of plant pesticides in the greenhouse]. Z Gesamte Hyg. 1989 Sep;35(9):531-3. Initial residues on the surface of cucumber, tomato and ornamental plants, and half-life periods for residue degradation are outlined for carbendazim, dimethoate, fenazox, malathion, methamidophos and pirimiphos-methyl. Residues on plants, concentration in the air, dermal exposition, and inhibition of serum choline esterase activity are shown for methamidophos and aldicarb, respective reentry times being discussed. |
1(0,0,0,1) | Details |
| 2590497 | Neskovic NK, Karan VZ, Sabovljevic V, Vitorovic SL: Toxic effects of pirimiphos-methyl residues on rats. Biomed Environ Sci. 1989 Jun;2(2):115-30. During and after the animal feeding study, changes in body weight gain, organ weight, cholinesterase activity, serum enzyme activity, and hematology were investigated. |
1(0,0,0,1) | Details |
| 1527360 | Qureshi MJ, Jamil FF, Haq A, Naqvi SH: Bioavailability and toxicity to rats of bound residues of 14C-pirimiphos-methyl in stored wheat. J Environ Sci Health B. 1992 Aug;27(4):369-75. In a 90-day feeding study, inhibition of plasma cholinesterase and brain acetylcholinesterase strongly suggest that the bound residues possess a toxicological potential. |
1(0,0,0,1) | Details |
| 3385133 | Rajini PS, Krishnakumari MK: Toxicity of pirimiphos-methyl: I. J Environ Sci Health B. 1988 Apr;23(2):127-44. Time-course inhibition pattern of both cholinesterase (ChE) and non-specific carboxylesterase (NSE) activities in brain and plasma revealed maximum inhibition at 24 h post-treatment which correlated well with the intensity of symptoms. |
1(0,0,0,1) | Details |