| Name | hemoglobin (protein family or complex) |
|---|---|
| Synonyms | Hemoglobin; Hemoglobins |
| Name | sodium cyanide |
|---|---|
| CAS | sodium cyanide (Na(CN)) |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 8825074 | Gilad E, Zisapel N: High-affinity binding of to hemoglobin. . Biochem Mol Med. 1995 Dec;56(2):115-20. Sodium cyanide had no effect on 125I- binding, indicating that 125I- does not bind to the heme group. |
7(0,0,0,7) | Details |
| 9372690 | Noriyuki T, Ohdan H, Yoshioka S, Miyata Y, Asahara T, Dohi K: Near-infrared spectroscopic method for assessing the tissue oxygenation state of living lung. Am J Respir Crit Care Med. 1997 Nov;156(5):1656-61. In in vitro experiments, we assayed the effect of photon scattering on the absorption spectra of an in vitro system simulating structures of lung, which consists of test tube containing air in hematocrit tubes and red blood cell suspension with various predetermined hemoglobin concentrations. |
4(0,0,0,4) | Details |
| 1308986 | Balla J, Jacob HS, Balla G, Nath K, Vercellotti GM: Endothelial cell heme oxygenase and ferritin induction by heme proteins: a possible mechanism limiting shock damage. Trans Assoc Am Physicians. 1992;105:1-6. needs to be released from methemoglobin, since sodium cyanide, haptoglobin, and hemopexin inhibit the induction of these proteins. Neutrophils can oxidize hemoglobin to methemoglobin, which can subsequently induce both heme oxygenase and ferritin. |
2(0,0,0,2) | Details |
| 2985190 | Acker H, Eyzaguirre C, Goldman WF: Redox changes in the mouse carotid body during hypoxia. . Brain Res. 1985 Mar 18;330(1):158-63. Wavelengths of 550 nm (maximal absorbance, MA) and 540 nm (isosbestic point, IP) were employed to study cytochrome c, 600 nm (MA) and 620 nm (IP) for a pigment presumed to be cytochrome aa3, 580 nm (MA) and 570 or 542 nm (IP) to control for possible effects of hemoglobin (Hb). |
1(0,0,0,1) | Details |
| 3991596 | Kruszyna H, Kruszyna R, Smith RP: and sulfide interact with nitrogenous compounds to influence the relaxation of various smooth muscles. Proc Soc Exp Biol Med. 1985 May;179(1):44-9. In each of these cases the relaxation produced by nitroprusside was at least partially reversed by the subsequent addition of excess sodium cyanide. Unlike however, sulfide augmented the relaxation induced by nitroprusside, and it reversed the effects of hemoglobin, nitroglycerin, and |
1(0,0,0,1) | Details |
| 1592224 | Scharf BA, Fricke RF, Baskin SI: Comparison of methemoglobin formers in protection against the toxic effects of Gen Pharmacol. 1992 Jan;23(1):19-25. Certain compounds that oxidize hemoglobin to methemoglobin (MHb) also protect against 2. The compounds were administered 15 or 60 min before an intramuscular (i.m.) challenge with a 2 x LD50 dose (5.0-5.6 mg/kg) of sodium cyanide (NaCN). 5. |
1(0,0,0,1) | Details |
| 6289701 | Kruszyna H, Kruszyna R, Smith RP: Nitroprusside increases cyclic monophosphate concentrations during relaxation of rabbit aortic strips and both effects are antagonized by Anesthesiology. 1982 Oct;57(4):303-8. The authors have confirmed previous observations that sodium cyanide (CN-) partially reverses the vasodilator effects of sodium nitroprusside (SNP) on vascular smooth muscle. The ED50 values for relaxation of aortic strips for a series of directly acting agonists vasodilators") were: azide (N-3) 2.1 X 10 (-7) M; SNP 2.7 X 10 (-7) M; (H2NOH) hydrochloride 2.5 X 10 (-6) M; human hemoglobin (HbNO) 3.5 X 10 (-6) M; and (NO-2) 1.2 X 10 (-4) M. |
1(0,0,0,1) | Details |
| 9414026 | Asano S, Matsuda T, Nakasu Y, Maeda S, Nogi H, Baba A: Inhibition by of the uptake of [3H] into rat brain synaptosomes. Jpn J Pharmacol. 1997 Oct;75(2):123-8. The inhibitory effect was blocked by reduced hemoglobin. NO-CYS and SNP, like iodoacetic acid and sodium cyanide, decreased the ATP content in cortical synaptosomes, but the effect on ATP content was not related to that on [3H] uptake. |
1(0,0,0,1) | Details |
| 6676476 | Ten Eyck RP, Schaerdel AD, Lynett JE, Marks DH, Patrissi GA, Ottinger WE, Stansell MJ: Stroma-free methemoglobin solution as an antidote for poisoning: a preliminary study. J Toxicol Clin Toxicol. 1983-1984;21(3):343-58. Rats injected with an LD100 intravenous dose of were treated with SFMS equal to 1.5% of their total body hemoglobin. |
1(0,0,0,1) | Details |
| 8100588 | Lonart G, Cassels KL, Johnson KM: induces -dependent [3H] release from striatal slices. J Neurosci Res. 1993 Jun 1;35(2):192-8. Sodium cyanide was used as a model compound to test the possibility that NO acted through blockade of mitochondrial electron transport. |
0(0,0,0,0) | Details |
| 6665758 | Schwerin FT, Rosenstein R, Smith RP: prevents the inhibition of platelet aggregation by nitroprusside, and azide. Thromb Haemost. 1983 Dec 30;50(4):780-3. Sodium cyanide (CN-) in concentrations of 10 uM or more prevented the inhibition of (2.5 uM) and of ADP (4.0 uM) induced primary and secondary aggregation brought about by 10 uM sodium nitroprusside (SNP). |
0(0,0,0,0) | Details |
| 14985415 | Reinert KC, Dunbar RL, Gao W, Chen G, Ebner TJ: Flavoprotein autofluorescence imaging of neuronal activation in the cerebellar cortex in vivo. J Neurophysiol. 2004 Jul;92(1):199-211. Epub 2004 Feb 25. Hypothesized to be the result of oxidation and subsequent reduction of flavoproteins, blocking mitochondrial respiration with sodium cyanide or inactivation of flavoproteins with diphenyleneiodonium substantially reduced the optical signal. |
0(0,0,0,0) | Details |
| 8645875 | Yamada M, Momose K, Richelson E, Yamada M: Sodium nitroprusside-induced apoptotic cellular death via production of peroxide in murine neuroblastoma N1E-115 cells. J Pharmacol Toxicol Methods. 1996 Feb;35(1):11-7. Furthermore, sodium cyanide, which is formed by the metabolism of sodium nitroprusside, did not cause cellular death. |
0(0,0,0,0) | Details |