Name | cholinesterase |
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Synonyms | Acylcholine acylhydrolase; BCHE; BCHE protein; Butyrylcholine esterase; Butyrylcholinesterase; CHE1; Choline esterase II; Cholinesterase… |
Name | bromophos |
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CAS |
PubMed | Abstract | RScore(About this table) | |
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3341046 | Shivanandappa T, Joseph P, Krishnakumari MK: Response of blood and brain cholinesterase to dermal exposure of bromophos in the rat. Toxicology. 1988 Feb;48(2):199-208. In a subacute study, daily dermal application of 50 mg/kg body weight of Bromophos for 5 and 10 days, resulted in high inhibition of the serum cholinesterase and brain acetylcholinesterase, the former being more marked which was reversible after 10 or 15 days of post-exposure period. |
132(1,2,5,7) | Details |
4715482 | Muacevic G: Acute toxicity and cholinesterase inhibition in vivo of bromophos-ethyl. Toxicol Appl Pharmacol. 1973 Jun;25(2):180-9. |
81(1,1,1,1) | Details |
8481105 | Willems JL, De Bisschop HC, Verstraete AG, Declerck C, Christiaens Y, Vanscheeuwyck P, Buylaert WA, Vogelaers D, Colardyn F: Cholinesterase reactivation in organophosphorus poisoned patients depends on the plasma concentrations of the oxime pralidoxime methylsulphate and of the organophosphate. Arch Toxicol. 1993;67(2):79-84. The bromophos poisoning was rather mild, cholinesterases were moderately inhibited and increased under oxime therapy. |
38(0,1,2,3) | Details |
7935256 | Santhoshkumar P, Shivanandappa T: Differential in vivo inhibition of the foetal and maternal brain acetylcholinesterase by bromophos in the rat. Neurotoxicol Teratol. 1994 May-Jun;16(3):227-32. This study measured the in vivo time-course of cholinesterase (ChE) inhibition and recovery in rat maternal serum, brain, and foetal brain after administration of a single acute oral dose of Bromophos (500 mg/kg b.w.) on Day 18 of pregnancy. |
31(0,1,1,1) | Details |
212825 | Stalberg E, Hilton-Brown P, Kolmodin-Hedman B, Holmstedt B, Augustinsson KB: Effect of occupational exposure to organophosphorus insecticides on neuromuscular function. Scand J Work Environ Health. 1978 Sep;4(3):255-61. Neurophysiological investigations and determinations of cholinesterase activity on plasma and erythrocytes were carried out on 11 Swedish spraymen exposed to bromophos, diazinon, dursbane, and malathion. |
8(0,0,1,3) | Details |
5422207 | Muacevic G, Dirks E, Kinkel HJ, Leuschner F: Anticholinesterase activity of bromophos in rats and dogs. Toxicol Appl Pharmacol. 1970 May;16(3):585-96. |
6(0,0,1,1) | Details |
52151 | Ramisz A, Komorowski A: [Histochemical study of the effect of phosphoroorganic esters Z-50 (fenchlorfos) and Z-51 (bromophos) on cholinesteras activity in the course of the muscle phase in experimental trichinellosis in mice]. Pol Arch Weter. 1975;17(4):623-31. The influence of esters on the activity of cholinesterases was investigated with the Koelle-Friedenwald method, modified by Gomori. |
4(0,0,0,4) | Details |
8812210 | Santhoshkumar P, Karanth S, Shivanandappa T: Neurotoxicity and pattern of acetylcholinesterase inhibition in the brain regions of rat by bromophos and ethylbromophos. Fundam Appl Toxicol. 1996 Jul;32(1):23-30. In vivo cholinesterase inhibition was in the order plasma > erythrocytes > brain. |
2(0,0,0,2) | Details |
8727219 | Nagayama M, Akahori F, Chiwata H, Shirai M, Motoya M, Masaoka T, Sakaguchi K: Effects of selected organophosphate insecticides on serum cholinesterase isoenzyme patterns in the rat. Vet Hum Toxicol. 1996 Jun;38(3):196-9. The effects of organophosphates (fenthion, chlorpyrifos, diazinon, bromophos, propaphos, haloxon, and DFP) on serum ChE isoenzyme bands were studied in 32 male and 32 female 6-w-old Sprague-Dawley rats. |
2(0,0,0,2) | Details |
985225 | Stone BF, Wilson JT, Youlton NJ: Linkage and dominance characteristics of genes for resistance to organophosphorus acaricides and allelic inheritance of decreased brain cholinesterase activity in three strains of the cattle tick, Boophilus microplus. Aust J Biol Sci. 1976 Jul;29(3):251-63. No F1 larval progeny from the following crossings were appreciably more resistant than their parents to these chemicals: R x B--bromophos ethyl and fenthion; B x M--carbaryl, chlorfenvinphos, chlorpyrifos, diazinon, dimethoate, ethion, fenthion and formothion; M x R--chlorfenvinphos, diazinon, dimethoate, ethion, formothion. |
1(0,0,0,1) | Details |
2051507 | Koppel C, Thomsen T, Heinemeyer G, Roots I: Acute poisoning with bromofosmethyl (bromophos). . J Toxicol Clin Toxicol. 1991;29(2):203-7. In contrast to poisoning by most organophosphates, red blood cell acetyl cholinesterase was only minimally depressed but the plasma butyryl cholinesterase was initially decreased and normalized within a few days. |
1(0,0,0,1) | Details |
10557009 | Guven M, Bayram F, Unluhizarci K, Kelestimur F: Endocrine changes in patients with acute organophosphate poisoning. Hum Exp Toxicol. 1999 Oct;18(10):598-601. In animals, cholinesterase inhibitors were shown to modify the pituitary-thyroid and pituitary- axes, and to affect prolactin levels. |
1(0,0,0,1) | Details |