| Name | Acetylcholinesterase |
|---|---|
| Synonyms | ACHE; ACHE protein; AChE; ARACHE; AcChoEase; Acetylcholine acetylhydrolase; Acetylcholinesterase; Acetylcholinesterase isoform E4 E6 variant… |
| Name | imidacloprid |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 18767757 | Ghadamyari M, Talebi K, Mizuno H, Kono Y: Oxydemeton-methyl resistance, mechanisms, and associated fitness cost in green peach aphids (Hemiptera: Aphididae). J Econ Entomol. 2008 Aug;101(4):1432-8. Susceptibility to oxydemeton-methyl and imidacloprid, and the inhibitory effects of oxydemeton-methyl and some organophosphate compounds on acetylcholinesterase (AChE) and carboxylesterase activity were studied in two populations (Karaj and Rasht) of green peach aphids, Myzus persicae (Sulzer). |
83(1,1,1,3) | Details |
| 12587870 | Foster SP, Denholm I, Thompson R: Variation in response to neonicotinoid insecticides in peach-potato aphids, Myzus persicae (Hemiptera: Aphididae). Pest Manag Sci. 2003 Feb;59(2):166-73. Laboratory bioassays applying the neonicotinoid insecticides imidacloprid, acetamiprid and nitenpyram against clones of the peach-potato aphid Myzus persicae (Sulzer) demonstrated that these compounds effectively circumvent the known carboxylesterase, modified acetylcholinesterase (MACE) and knock-down (kdr) insecticide resistance mechanisms in this species. |
81(1,1,1,1) | Details |
| 12020021 | Wang KY, Liu TX, Yu CH, Jiang XY, Yi MQ: Resistance of aphis gossypii (Homoptera: Aphididae) to fenvalerate and imidacloprid and activities of detoxification enzymes on cotton and cucumber. J Econ Entomol. 2002 Apr;95(2):407-13. Activities of acetylcholinesterase (AChE) and alpha-naphthylacetate (alpha-NA) esterases of A. gossypii were significantly different among the three strains, with the R-fenvalerate strains having the highest, followed by the R-imidacloprid strain, and the S strain the lowest. |
7(0,0,1,2) | Details |
| 20146932 | Bhardwaj S, Srivastava MK, Kapoor U, Srivastava LP: A 90 days oral toxicity of imidacloprid in female rats: Morphological, biochemical and histopathological evaluations. Food Chem Toxicol. 2010 Feb 8. In clinical chemistry parameters high dose of imidacloprid has caused significant elevation of serum GOT, GPT, and BUN and decreased the activity of AChE in serum and brain. |
6(0,0,1,1) | Details |
| 18080902 | Abou-Donia MB, Goldstein LB, Bullman S, Tu T, Khan WA, Dechkovskaia AM, Abdel-Rahman AA: Imidacloprid induces neurobehavioral deficits and increases expression of glial fibrillary acidic protein in the motor cortex and hippocampus in offspring rats following in utero exposure. J Toxicol Environ Health A. 2008;71(2):119-30. On postnatal day (PND) 30, all male and female offspring were evaluated for (a) acetylcholinesterase (AChE) and butyrylcholinesterase (BuChE) activity, (b) ligand binding for nicotinic acetylcholine receptors (nAChR) and muscarinic acetylcholine receptors (m2 mAChR), (c) sensorimotor performance (inclined plane, beam-walking, and forepaw grip), and (d) pathological alterations in the brain (using cresyl violet and glial fibrillary acidic protein [GFAP] immunostaining). |
2(0,0,0,2) | Details |
| 18399471 | Bullangpoti V, Visetson S, Milne J, Milne M, Sudthongkong C, Pronbanlualap S: Effects of alpha-mangostin from mangosteen pericarp extract and imidacloprid on Nilaparvata lugens (Stal.) and non-target organisms: toxicity and detoxification mechanism. Commun Agric Appl Biol Sci. 2007;72(3):431-41. In vitro detoxification enzyme activities of carboxylesterase, acetylcholinesterase and glutathione-s-transferase from BPH after 24 hours exposure were also observed. |
1(0,0,0,1) | Details |
| 19115232 | Roditakis E, Grispou M, Morou E, Kristoffersen JB, Roditakis N, Nauen R, Vontas J, Tsagkarakou A: Current status of insecticide resistance in Q biotype Bemisia tabaci populations from Crete. Pest Manag Sci. 2009 Mar;65(3):313-22. A propoxur-based AChE diagnostic test indicated that iAChE was widespread in most populations. High resistance levels were identified in the majority of populations (> 80%) for imidacloprid (RF: 38-1958x) and alpha-cypermethrin (RF: 30-600x). |
1(0,0,0,1) | Details |
| 8841090 | Narahashi T: Neuronal ion channels as the target sites of insecticides. Pharmacol Toxicol. 1996 Jul;79(1):1-14. Nitromethylene heterocycles including imidacloprid act on nicotinic acetylcholine receptors. With the exception of organophosphate and insecticides which inhibit acetylcholinesterases, most insecticide commercially developed act on the sodium channel and the system. |
1(0,0,0,1) | Details |
| 17573686 | Margaritopoulos JT, Skouras PJ, Nikolaidou P, Manolikaki J, Maritsa K, Tsamandani K, Kanavaki OM, Bacandritsos N, Zarpas KD, Tsitsipis JA: Insecticide resistance status of Myzus persicae (Hemiptera: Aphididae) populations from peach and tobacco in mainland Greece. Pest Manag Sci. 2007 Aug;63(8):821-9. The susceptibility of 88 and 38 field samples of Myzus persicae (Sulzer) to imidacloprid and deltamethrin respectively was examined using the FAO dip test bioassay. In addition, 497, 349 and 370 clones originating from peach and tobacco were screened for the three known resistance mechanisms, elevated esterases, modified acetylcholinesterase (MACE) and knockdown resistance (kdr) respectively, using biochemical and DNA diagnostics. |
1(0,0,0,1) | Details |