| Name | fibronectin |
|---|---|
| Synonyms | CIG; MSF; Cold insoluble globulin; FINC; FN; FN1; Fibronectin; Fibronectin 1… |
| Name | sodium azide |
|---|---|
| CAS | sodium azide |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 16595934 | Kawazu K, Oshita A, Nakamura T, Nakashima M, Ichikawa N, Sasaki H: Transport of acebutolol through rabbit corneal epithelium. Biol Pharm Bull. 2006 Apr;29(4):846-9. Primary RCEC were seeded on a filter membrane of Transwell-COL insert coated with fibronectin and were grown in Dulbecco's modified Eagle's medium/nutrient mixture F-12 with various supplements. Sodium azide, and cyclosporin A enhanced the transcellular permeability of acebutolol in the A-to-B direction. |
1(0,0,0,1) | Details |
| 17015968 | Sakanaka K, Kawazu K, Nishida K, Nakamura J, Nakashima M, Nakamura T, Oshita A, Ichikawa N, Sasaki H: Transport of timolol and tilisolol in rabbit corneal epithelium. Biol Pharm Bull. 2006 Oct;29(10):2143-7. Primary RCEC were seeded on a filter membrane of Transwell-COL insert coated with fibronectin and grown in Dulbecco's modified Eagle's medium/nutrient mixture F-12 with various supplements. The transcellular permeability of tilisolol from the apical to the basal side was inhibited by sodium azide, tetraethylammonium, quinidine, and |
1(0,0,0,1) | Details |
| 8724599 | Khaspekova SG, Vyzova TV, Lukin VV, Tikhomirov OIu, Berndt M, Kouns W, Mazurov AV: [Conformational changes of the platelet membrane glycoprotein IIb-IIIa complex stimulated by a monoclonal antibody to the N-terminal segment of glycoprotein IIIa]. Biokhimiia. 1996 Mar;61(3):412-28. CRC54 also stimulated the binding to platelets of GPIIb-IIIa ligand fibrinogen, labelled with 125I as well as adhesion of 51Cr-labelled platelets to immobilized ligands-fibrinogen and fibronectin. However, the platelet activation inhibitor, prostaglandin EI, and the mixture of metabolic inhibitors, deoxyglucose-sodium azide, only party inhibited the CRC54-dependent aggregation. |
1(0,0,0,1) | Details |
| 19191104 | Kikuchi K, Kohyama T, Yamauchi Y, Kato J, Takami K, Okazaki H, Desaki M, Nagase T, Rennard SI, Takizawa H: C-reactive protein modulates human lung fibroblast migration. Exp Lung Res. 2009 Feb;35(1):48-58. The aim of this study was to investigate the effect of CRP on migration of human fetal lung fibroblasts (HFL-1) to human plasma fibronectin (HFn). |
1(0,0,0,1) | Details |
| 8406852 | Ollert MW, Sohnchen R, Korting HC, Ollert U, Brautigam S, Brautigam W: Mechanisms of adherence of Candida albicans to cultured human epidermal keratinocytes. Infect Immun. 1993 Nov;61(11):4560-8. C. albicans adherence was blocked by the acid protease inhibitor A and the metabolic inhibitor sodium azide. |
0(0,0,0,0) | Details |
| 7558718 | O'Sullivan NL, Raja R, Montgomery PC: Lymphocyte adhesive interactions with lacrimal gland acinar epithelial cells in primary culture. Invest Ophthalmol Vis Sci. 1995 Oct;36(11):2246-53. Thoracic duct lymphocyte binding required intact metabolic and membrane-cytoskeletal function and was inhibited by treating the lymphocytes with sodium azide, or cytochalasin B (23%, 12%, and 10% of control binding, respectively). |
0(0,0,0,0) | Details |
| 9028260 | O'Sullivan NL, Montgomery PC: Lymphocyte adhesive interactions with cultured parotid salivary gland epithelial cells from rats. Oral Microbiol Immunol. 1996 Oct;11(5):337-42. TDL binding was inhibited by sodium azide or cytochalasin B (60% and 80%, respectively). |
0(0,0,0,0) | Details |
| 8548838 | Schroen DJ, Cheung HT: Interaction of mouse thymocytes and a thymocyte-like cell line with the ECM glycoprotein entactin. Cell Immunol. 1996 Jan 10;167(1):141-9. Preincubation of the S49.1 cells in medium containing the metabolic inhibitors sodium azide or 2-deoxy- inhibited adhesion to entactin 47.2 and 79.5%, respectively. A high percentage (approximately 90-100%) of S49.1 cells also bound to the lectin concanavalin A and to fibronectin, while laminin promoted only 19.3% adhesion. |
4(0,0,0,4) | Details |
| 9235931 | Page K, Lange Y: Cell adhesion to fibronectin regulates membrane lipid biosynthesis through 5'-AMP-activated protein kinase. J Biol Chem. 1997 Aug 1;272(31):19339-42. For example, in our system, sodium azide and 2-deoxy- increased the ratio of cellular AMP to ATP and caused a decrease in lipid biosynthesis. |
3(0,0,0,3) | Details |
| 8769278 | Ebert EC: Mechanisms of colon cancer binding to substratum and cells. Dig Dis Sci. 1996 Aug;41(8):1551-6. Binding began within minutes and peaked by 1 hr, with 80-95% of HT-29 cells binding to the ECM proteins, collagen IV, laminin, fibronectin, and vitronectin and 40-75% binding to monolayers of fibroblasts, smooth muscle cells, and HT-29 cells. Attachment of HT-29 cells to cell monolayers was inhibited by cytochalasin D and sodium azide, but not cycloheximide or neuraminidase. |
2(0,0,0,2) | Details |
| 8751977 | Herrmann G, Wlaschek M, Bolsen K, Prenzel K, Goerz G, Scharffetter-Kochanek K: Photosensitization of uroporphyrin augments the ultraviolet A-induced synthesis of matrix metalloproteinases in human dermal fibroblasts. J Invest Dermatol. 1996 Sep;107(3):398-403. In this study, we therefore asked whether long-wave ultraviolet irradiation (340-450 nm) in conjunction with could modulate the synthesis of MMPs with substrate specificities for dermal (collagens I, III, V; proteoglycans) and basement membrane components (collagens IV, VII; fibronectin; laminin) and whether synthesis of the counteracting tissue inhibitor of metalloproteinases is also affected. Using deuterium oxide as enhancer and sodium azide as quencher of singlet we could increase or reduce MMP synthesis, suggesting that singlet is the major intermediate in the upregulation of MMPs after irradiation of uroporphyrin-pretreated fibroblasts. |
1(0,0,0,1) | Details |
| 10362101 | Runnels JM, Chen N, Ortel B, Kato D, Hasan T: BPD-MA-mediated photosensitization in vitro and in vivo: cellular adhesion and beta1 integrin expression in ovarian cancer cells. Br J Cancer. 1999 Jun;80(7):946-53. Mild BPD-MA photosensitization (approximately 85% cell survival) of OVCAR 3 transiently decreased adhesion to collagen IV, fibronectin, laminin and vitronectin to a greater extent than could be attributed to cell death. Photosensitized adhesion was reduced in the presence of sodium azide and enhanced in deuterium oxide, suggesting mediation by singlet |
1(0,0,0,1) | Details |