| Name | catalase |
|---|---|
| Synonyms | CAT; Catalase; Erythrocyte derived growth promoting factor; Carnitine O acetyltransferase; Carnitine acetylase; Carnitine acetyltransferase; CAT; Catalases… |
| Name | mercuric chloride |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 10390852 | Hussain S, Atkinson A, Thompson SJ, Khan AT: Accumulation of mercury and its effect on antioxidant enzymes in brain, liver, and kidneys of mice. J Environ Sci Health B. 1999 Jul;34(4):645-60. The effect of mercuric chloride (HgCl2) on the activities of catalase, superoxide dismutase (SOD), peroxidase (GPx), glutathione reductase (GR) and its effect on (GSH) content were evaluated in different organs (liver, kidneys, and brain) of mice after administration at 0, 0.25, 0.5 and 1.0 mg/kg/day for 14 days. |
6(0,0,1,1) | Details |
| 17706852 | Sharma MK, Sharma A, Kumar A, Kumar M: Spirulina fusiformis provides protection against mercuric chloride induced oxidative stress in Swiss albino mice. Food Chem Toxicol. 2007 Dec;45(12):2412-9. Epub 2007 Jun 28. The activities of antioxidants enzymes superoxide dismutase, catalase and glutathione-S-transferase were also concomitantly restored to near normal level by Spirulina supplementation to mercuric chloride intoxicated mice. |
6(0,0,1,1) | Details |
| 17436531 | Kavitha AV, Jagadeesan G: Role of Tribulus terrestris (Linn.) (Zygophyllacea) against mercuric chloride induced nephrotoxicity in mice, Mus musculus (Linn.). J Environ Biol. 2006 May;27(2 Suppl):397-400. At median-lethal dose of mercuric chloride (12.9 mg/kg body weight), the whole kidney tissue showed an increased level of lipid peroxidation (LPO), peroxidase (GPx) activities and simultaneously a decreased level of superoxide dismutase (SOD), catalase (CAT), |
6(0,0,1,1) | Details |
| 15977196 | Bando I, Reus MI, Andres D, Cascales M: Endogenous antioxidant defence system in rat liver following mercury oral intoxication. J Biochem Mol Toxicol. 2005;19(3):154-61. Superoxide dismutases, catalase, and peroxidase are proteins involved in the endogenous antioxidant defence system. |
3(0,0,0,3) | Details |
| 8872981 | Nath KA, Croatt AJ, Likely S, Behrens TW, Warden D: Renal oxidant injury and oxidant response induced by mercury. Kidney Int. 1996 Sep;50(3):1032-43. The role of oxidative stress in mercuric chloride (HgCl2)-induced nephrotoxicity is uncertain and controversial. HgCl2 exerts a dose-dependent cytotoxicity which is attenuated by and catalase. |
2(0,0,0,2) | Details |
| 11141179 | Henzler T, Steudle E: Transport and metabolic degradation of peroxide in Chara corallina: model calculations and measurements with the pressure probe suggest transport of H (2) O (2) across water channels. J Exp Bot. 2000 Dec;51(353):2053-66. When water channels were inhibited with the blocker mercuric chloride (HgCl (2)), the permeabilities of both water and H (2) O (2) were substantially reduced. A mathematical model is presented that describes permeation of peroxide across a cell membrane and the implications of solute decomposition by catalase inside the cell. |
2(0,0,0,2) | Details |
| 16674986 | Cargnelutti D, Tabaldi LA, Spanevello RM, de Oliveira Jucoski G, Battisti V, Redin M, Linares CE, Dressler VL, de Moraes Flores EM, Nicoloso FT, Morsch VM, Schetinger MR: Mercury toxicity induces oxidative stress in growing cucumber seedlings. Chemosphere. 2006 Nov;65(6):999-1006. Epub 2006 May 3. In this study, the effects of exogenous mercury (HgCl (2)) on time-dependent changes in the activities of antioxidant enzymes (catalase and peroxidase), lipid peroxidation, chlorophyll content and protein oxidation in cucumber seedlings (Cucumis sativus L.) were investigated. |
2(0,0,0,2) | Details |
| 19917674 | Venturini C, Beatson SA, Djordjevic SP, Walker MJ: Multiple antibiotic resistance gene recruitment onto the enterohemorrhagic Escherichia coli virulence plasmid. FASEB J. 2010 Apr;24(4):1160-6. Epub 2009 Nov 16. Determination of the 111,481-bp sequence of pO26-CRL revealed genes encoding a functional enterohemolysin operon (ehxCABD), STEC-specific extracellular serine protease (espP), putative EHEC adhesin (toxB), catalase/peroxidase (katP), and myristoyl transferase (msbB) involved in lipid A synthesis. A 22,609-bp Tn21 derivative is inserted within the conjugal transfer gene traC and encodes resistance to trimethoprim, streptomycin, sulfathiozole, kanamycin, neomycin, beta-lactams, and mercuric chloride. |
1(0,0,0,1) | Details |
| 19382722 | Zhao JQ, Wen YF, Bhadauria M, Nirala SK, Sharma A, Shrivastava S, Shukla S, Agrawal OP, Mathur R: Protective effects of propolis on inorganic mercury induced oxidative stress in mice. Indian J Exp Biol. 2009 Apr;47(4):264-9. Exposure to mercuric chloride (HgCl2; 5 mg/kg; ip) induced oxidative stress by increasing lipid peroxidation and level along with concomitant decrease in and various antioxidant enzymes. Activities of antioxidants enzymes, i.e., superoxide dismutase, catalase, glutathione-S-transferase and glucose-6-phosphate dehydrogenase were also restored concomitantly towards control after propolis administration. |
1(0,0,0,1) | Details |
| 9022032 | Wolfreys K, Oliveira DB: Alterations in intracellular reactive species generation and redox potential modulate mast cell function. Eur J Immunol. 1997 Jan;27(1):297-306. The administration of mercuric chloride (HgCl2), gold compounds, or D-penicillamine to Brown Norway (BN) rats causes a T helper (Th) 2 cell-associated autoimmune syndrome characterized by the production of a number of autoantibodies, marked elevation of serum IgE concentration, and tissue injury in the form of a vasculitis and arthritis. Inhibition of ROS formation by desferrioxamine or catalase diminished the enhancement of IgE-mediated release caused by HgCl2, as did replenishment of intracellular 2-Mercaptoethanol exacerbated the toxicity of HgCl2, perhaps due to the formation of a lipophilic complex that enhanced HgCl2 uptake. |
1(0,0,0,1) | Details |
| 11053052 | Nava M, Romero F, Quiroz Y, Parra G, Bonet L, Rodriguez-Iturbe B: attenuates acute renal failure and oxidative stress induced by mercuric chloride in rats. Am J Physiol Renal Physiol. 2000 Nov;279(5):F910-8. We also determined the renal content of malondialdehyde (MDA) and (GSH) and the activities of peroxidase and catalase. |
1(0,0,0,1) | Details |
| 20229497 | Agarwal R, Goel SK, Behari JR: Detoxification and antioxidant effects of in rats experimentally exposed to mercury. J Appl Toxicol. 2010 Mar 12. Therefore, the detoxification and antioxidant effects of were examined to determine its prophylactic/therapeutic role in rats experimentally exposed to mercury (in the from of mercuric chloride-HgCl (2), 12 micromol kg (-1) b.w. single intraperitoneal injection). treatment (80 mg kg (-1) b.w. daily for 3 days, orally) was found to have a protective effect on mercury-induced oxidative stress parameters, namely, lipid peroxidation and levels and superoxide dismutase, peroxidase and catalase activities in the liver, kidney and brain. |
1(0,0,0,1) | Details |
| 7680428 | Sugiyama M, Tsuzuki K, Haramaki N: DNA single-strand breaks and cytotoxicity induced by chromate (VI) in peroxide-resistant cell lines. Mutat Res. 1993 Apr;299(2):95-102. Catalase activity in resistant cells was 2-fold and the cellular content of was 3-fold that in parental cells. |
1(0,0,0,1) | Details |
| 17516994 | Augusti PR, Conterato GM, Somacal S, Einsfeld L, Ramos AT, Hosomi FY, Graca DL, Emanuelli T: Effect of on nephrotoxicity induced by mercuric chloride in rats. Basic Clin Pharmacol Toxicol. 2007 Jun;100(6):398-402. peroxidase and catalase activities were enhanced (approximately 71% and approximately 41%), while superoxide dismutase activity was depressed (approximately 44%) in HgCl (2)-treated rats when compared to control and these effects were prevented by |
1(0,0,0,1) | Details |
| 17634687 | Agarwal R, Behari JR: Role of in mercury intoxication in mice. Ind Health. 2007 Jun;45(3):388-95. Studies were conducted to examine the effect of pre and post-treatment of in mercury intoxication (20 micromole/ kg b.w. each given intraperitoneally) in mice in terms of lipid peroxidation (LPO), (GSH) content, activities of superoxide dismutase (SOD), peroxidase (GPx), catalase (CAT) and mercury concentration in liver, kidney and brain. |
1(0,0,0,1) | Details |
| 17207565 | Sarwar Alam M, Kaur G, Jabbar Z, Javed K, Athar M: Eruca sativa seeds possess antioxidant activity and exert a protective effect on mercuric chloride induced renal toxicity. Food Chem Toxicol. 2007 Jun;45(6):910-20. Epub 2006 Nov 29. Oxidative modulation of renal tissues following HgCl (2) exposure was evident from a significant elevation in lipid peroxidation and attenuation in (GSH) contents and activities of antioxidant enzymes viz., catalase (CAT), peroxidase (GPX), superoxide dismutase (SOD) and glutathione reductase (GR). |
1(0,0,0,1) | Details |
| 7896172 | Girardi G, Elias MM: Mercuric chloride effects on rat renal redox enzymes activities: SOD protection. Free Radic Biol Med. 1995 Jan;18(1):61-6. peroxidase (GSH-Px) and catalase (Cat) activities were measured in kidney homogenates from rats with different nonprotein sulfhydrils levels and different mercury content. |
1(0,0,0,1) | Details |
| 19636703 | Monteiro DA, Rantin FT, Kalinin AL: Inorganic mercury exposure: toxicological effects, oxidative stress biomarkers and bioaccumulation in the tropical freshwater fish matrinxa, Brycon amazonicus (Spix and Agassiz, 1829). Ecotoxicology. 2010 Jan;19(1):105-23. Epub 2009 Jul 28. Increases in superoxide dismutase, catalase, peroxidase (GPx), glutathione S-transferase (GST) and glutathione reductase (GR) were observed in all tissues after HgCl (2) exposure, except for white muscle GR activity and hepatic GPx. |
1(0,0,0,1) | Details |
| 9712749 | Gobert AP, Semballa S, Daulouede S, Lesthelle S, Taxile M, Veyret B, Vincendeau P: Murine macrophages use - and -dependent mechanisms to synthesize S-nitroso-albumin and to kill extracellular trypanosomes. Infect Immun. 1998 Sep;66(9):4068-72. S nitrosylation was confirmed by the Saville reaction, using mercuric chloride. The immunoenzymatic signal decreased by 66 and 30% when superoxide dismutase and catalase, respectively, were added to the culture medium of macrophages from infected mice. |
1(0,0,0,1) | Details |
| 18758054 | Ghosh A, Sil PC: A protein from Cajanus indicus Spreng protects liver and kidney against mercuric chloride-induced oxidative stress. Biol Pharm Bull. 2008 Sep;31(9):1651-8. Intraperitoneal administration of HgCl (2) at a dose of 5 mg/kg body weight for 1 d significantly reduced the activities of antioxidant enzymes like superoxide dismutase (SOD), catalase (CAT) and peroxidase (GPx). |
1(0,0,0,1) | Details |
| 19406194 | Brandao R, Borges LP, Nogueira CW: Concomitant administration of 2,3-dimercapto-1-propanesulphonate (DMPS) and diphenyl diselenide reduces effectiveness of DMPS in restoring damage induced by mercuric chloride in mice. Food Chem Toxicol. 2009 Aug;47(8):1771-8. Epub 2009 May 3. Thiobarbituric acid-reactive substances (TBARS), and Hg (2+) levels and glutathione S-transferase (GST) and catalase (CAT) activities were carried out in kidney. |
1(0,0,0,1) | Details |
| 18187036 | Folmer V, Pedroso N, Matias AC, Lopes SC, Antunes F, Cyrne L, Marinho HS: H2O2 induces rapid biophysical and permeability changes in the plasma membrane of Saccharomyces cerevisiae. Biochim Biophys Acta. 2008 Apr;1778(4):1141-7. Epub 2008 Jan 8. Moreover, a 40% decrease in plasma membrane permeability to H2O2 was observed at 15min with a concomitant two-fold increase in catalase activity. H2O2 diffusion through the plasma membrane in S. cerevisiae cells is not mediated by aquaporins since the H2O2 permeability constant is not altered in the presence of the aquaporin inhibitor mercuric chloride. |
1(0,0,0,1) | Details |
| 17881112 | Augusti PR, Conterato GM, Somacal S, Sobieski R, Spohr PR, Torres JV, Charao MF, Moro AM, Rocha MP, Garcia SC, Emanuelli T: Effect of on kidney function impairment and oxidative stress induced by mercuric chloride in rats. Food Chem Toxicol. 2008 Jan;46(1):212-9. Epub 2007 Aug 14. peroxidase and catalase activities were enhanced, while superoxide dismutase activity was depressed in HgCl (2)-treated rats when compared to control and these effects were prevented by ASX. |
1(0,0,0,1) | Details |
| 12781854 | Farina M, Soares FA, Feoli A, Roehring C, Brusque AM, Rotta L, Perry ML, Souza DO, Rocha JB: In vitro effects of selenite and mercuric chloride on liver thiobarbituric acid-reactive substances and non-protein thiols from rats: influences of dietary and polyunsaturated and saturated fatty acids. Nutrition. 2003 Jun;19(6):531-5. Catalase activity was higher in animals fed diets containing soybean oil. |
1(0,0,0,1) | Details |
| 17944542 | Franco JL, Braga HC, Stringari J, Missau FC, Posser T, Mendes BG, Leal RB, Santos AR, Dafre AL, Pizzolatti MG, Farina M: Mercurial-induced peroxide generation in mouse brain mitochondria: protective effects of Chem Res Toxicol. 2007 Dec;20(12):1919-26. Epub 2007 Oct 19. These oxidative phenomena were prevented by co-incubation with or catalase. In this work, we have examined the potential protective effects of three compounds (7-prenyloxy-6-methoxycoumarin, and 1,5-dihidroxi-2,3-dimethoxy xanthone) from Polygala species against MeHg- and mercuric chloride (HgCl2)-induced disruption of mitochondrial function under in vitro conditions using mitochondrial-enriched fractions from mouse brain. |
1(0,0,0,1) | Details |
| 12110286 | Aleo MF, Morandini F, Bettoni F, Tanganelli S, Vezzola A, Giuliani R, Steimberg N, Apostoli P, Mazzoleni G: Antioxidant potential and gap junction-mediated intercellular communication as early biological markers of mercuric chloride toxicity in the MDCK cell line. Toxicol In Vitro. 2002 Aug;16(4):457-65. The cell (GSH) content and the activity of peroxidase (Gpx) and catalase (Cat), two enzymes engaged in the H (2) O (2) degradation, were quantified. |
1(0,0,0,1) | Details |
| 15466269 | Jo SK, Hu X, Yuen PS, Aslamkhan AG, Pritchard JB, Dear JW, Star RA: Delayed DMSO administration protects the kidney from mercuric chloride-induced injury. J Am Soc Nephrol. 2004 Oct;15(10):2648-54. |
0(0,0,0,0) | Details |
| 9530530 | Anuradha B, Rajeswari M, Varalakshmi P: Degree of peroxidative status in neuronal tissues by different routes of inorganic mercury administration. Drug Chem Toxicol. 1998 Feb;21(1):47-55. Mercuric chloride was administered by three routes--subcutaneous, intramuscular and intraperitoneal to adult female Wistar rats. All the experimental groups show high levels of and increased activities of peroxidase, superoxide dismutase and catalase. |
1(0,0,0,1) | Details |
| 18405108 | Jagadeesan G, Sankarsami Pillai S: Hepatoprotective effects of against mercury induced toxicity in rats. J Environ Biol. 2007 Oct;28(4):753-6. The animals were treated with sublethal dose of mercuric chloride (2 mg/kg body wt.) for 30 days. During the mercury treatment, the level ofAspartate transaminase (AST), transaminase (ALT) and Alkaline phosphatase (ALP) in serum and lipid peroxidation (LPO) in liver tissue significantly increased whereas (GSH), peroxidase (GPx), Catalase (CAT) and Superoxide dismutase (SOD) were simultaneously decreased in the liver tissue. |
1(0,0,0,1) | Details |
| 12180707 | Ferrat L, Romeo M, Gnassia-Barelli M, Pergent-Martini C: Effects of mercury on antioxidant mechanisms in the marine phanerogam Posidonia oceanica. Dis Aquat Organ. 2002 Jul 8;50(2):157-60. Biochemical markers of oxidative stress such as catalase activity, glutathione S-transferase (GST) activity and levels of lipid peroxidation evaluated in terms of thiobarbituric acid reactive substances (TBARS) were measured in the sheaths of the marine phanerogam Posidonia oceanica (L.) Delile experimentally exposed to 0.01, 0.1 and 1 microgHg l (-1) for 48 h. |
1(0,0,0,1) | Details |
| 2219185 | Pfaller W, Gstraunthaler G, Willinger CC: Morphology of renal tubular damage from nephrotoxins. Toxicol Lett. 1990 Sep;53(1-2):39-43. Quantitative morphological-stereological-analysis of at least two, the mercuric chloride- and the -induced experimental models of toxic acute renal failure, show a very early substantial loss of ATP-generating mitochondrial inner membrane surface as well as substantial decrease in those functions protecting cells against oxidative or auto-oxidative processes, i.e. content, activities of the free-radical-scavenging systems superoxide dismutase, peroxidase and glutathione reductase and catalase. |
31(0,1,1,1) | Details |
| 15092159 | Subhadra AV, Nanda AK, Behera PK, Panda BB: Acceleration of catalase and peroxidase activities in Lemna minor L. and Allium cepa L. in response to low levels of aquatic mercury. Environ Pollut. 1991;69(2-3):169-79. Following a 96-h exposure of plants to nutrient medium contaminated with known levels of mercuric chloride (HgCl (2)), 0.001 to 4 mg litre (-1) (0.0007 to 2.95 mg Hg litre (-1)) or methyl mercuric chloride (MeHgCl (2)), 0.0001 to 0.1 mg litre (-1) (0.0007 to 0.07 mg Hg litre (-1)), the physiological endpoints measured were the growth of fronds (Lemna minor) or roots (Allium cepa), and catalase and peroxidase activities in both plant assays. |
8(0,0,1,3) | Details |
| 19576259 | Rao MV, Chhunchha B: Protective role of against the mercury induced oxidative stress in the rat thyroid. Food Chem Toxicol. 2010 Jan;48(1):7-10. Epub 2009 Jul 1. Present study investigated the protective role of (MLT, 5mg/kg body wt., ip) against the long term effects of mercuric chloride (MC; 2 and 4 mg/kg body wt., po) in the thyroid gland of the rats through certain antioxidative indices like superoxide dismutase (SOD), peroxidase (GPx), glutathione reductase (GR), (GSH), catalase (CAT) and lipid peroxidation (LPO), other biochemical parameters such as succinate dehydrogenase (SDH), triphosphatase (ATPase), acid phosphatase (ACPase) and alkaline phosphatase (ALPase) were also measured. |
6(0,0,1,1) | Details |
| 2260496 | Meguro T, Ishii K, Ogata M: Exhaled metallic mercury in acatalasemic, hypocatalasemic and normal mice injected with mercury (II) Acta Med Okayama. 1990 Oct;44(5):251-6. To clarify the relationship between the catalase activity in mouse organs and the amounts of metallic mercury exhaled, normal, homozygous hypocatalasemic and acatalasemic mice were injected with mercuric chloride. |
7(0,0,1,2) | Details |