Name | Atrial natriuretic factor |
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Synonyms | ANF; ANP; Atrial natriuretic factor; Atrial natriuretic factor precursor; Atrial natriuretic peptide; Atrial natriuretic polypeptide; Atrionatriureticfactor; Atriopeptin… |
Name | diphenylamine |
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CAS |
PubMed | Abstract | RScore(About this table) | |
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2153599 | Moriarty KJ, Higgs NB, Lees M, Tonge A, Wardle TD, Warhurst G: Influence of atrial natriuretic peptide on mammalian large intestine. . Gastroenterology. 1990 Mar;98(3):647-53. The response to atrial natriuretic peptide was inhibited by (a) -free solution on the serosal surface; (b) pretreatment of the tissues with the chloride channel blocker, diphenylamine-2-carboxylate (10 (-3) M mucosally); (c) pretreatment with d,l- (10 (-4) M mucosally and serosally); (d) -free solution on the serosal surface; (e) pretreatment with tetrodotoxin (10 (-7) M to serosal surface); and (f) pretreatment with atropine (10 (-5) M serosally). |
37(0,1,1,7) | Details |
7684181 | Grotmol T, Rodnes JT, Buanes T, Christensen G, Landsverk T: Atrial natriuretic factor (ANF) does not affect ion transport in human intestine but does in porcine intestine. Acta Physiol Scand. 1993 Apr;147(4):417-29. The aim of this study was to test whether atrial natriuretic factor (ANF) exerts any effect on human intestinal ion transport, and the porcine intestine was used as a positive control of ANF's effects. The porcine Isc response was partly inhibited by -free solution on the serosal side, by serosal application of bumetanide (10 (-4) M) and BaCl2 (10 (-3) M), and mucosal application of the chloride-channel blocker diphenylamine-2-carboxylate (DPC) (10 (-3) M). |
1(0,0,0,1) | Details |
9888876 | Suenobu N, Shichiri M, Iwashina M, Marumo F, Hirata Y: Natriuretic peptides and mechanism. Arterioscler Thromb Vasc Biol. 1999 Jan;19(1):140-6. Three natriuretic peptides (atrial natriuretic peptide, brain natriuretic peptide, and C-type natriuretic peptide) induced endothelial apoptosis as demonstrated by nucleosomal laddering on agarose gel electrophoresis and by the terminal deoxynucleotidyl transferase-mediated nick end labeling method. This dose-dependent relation was assessed by quantifying the fragmented and intact DNA contents by the diphenylamine method. |
induce endothelial apoptosis via a -dependent 2(0,0,0,2) | Details |