| Name | EDRF |
|---|---|
| Synonyms | AHSP; Alpha hemoglobin stabilizing protein; EDRF; ERAF; Erythroid associated factor; Erythroid differentiation related factor; Alpha hemoglobin stabilizing proteins; Erythroid associated factors… |
| Name | amitrole |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 2337161 | Kontos HA, Wei EP, Kukreja RC, Ellis EF, Hess ML: Differences in endothelium-dependent cerebral dilation by bradykinin and Am J Physiol. 1990 May;258(5 Pt 2):H1261-6. The findings are consistent with the view that the cerebral arteriolar dilation from bradykinin is caused by radicals generated in association with accelerated metabolism via cyclooxygenase, whereas the dilation from is caused by an endothelium-derived relaxing factor (EDRF) similar to that generated by this agent in large vessels in vitro. |
2(0,0,0,2) | Details |
| 1847583 | Mugge A, Elwell JH, Peterson TE, Harrison DG: Release of intact endothelium-derived relaxing factor depends on endothelial superoxide dismutase activity. Am J Physiol. 1991 Feb;260(2 Pt 1):C219-25. Endothelium-derived relaxing factor (EDRF) is rapidly inactivated by radicals. |
1(0,0,0,1) | Details |
| 11874445 | Kocak G, Karasu C: Elimination of *O (2)(-)/H (2) O (2) by mediates the recovery of basal EDRF/NO availability and the reversal of superoxide dismutase-induced relaxation in diabetic rat aorta. Diabetes Obes Metab. 2002 Jan;4(1):69-74. CONCLUSION: Findings suggested that: (i) in normal physiological conditions, the relaxant effect of SOD is related to the inhibition of radicals (*O (2)(-))-induced endothelium-derived relaxing factor/ (EDRF/NO) destruction in the rat aorta; (ii) in diabetic state, excess *O (2)(-) increasingly inhibits basal EDRF/NO, and the dismutation of excess *O (2)(-) to H (2) O (2) is enhanced by exogenous SOD. |
1(0,0,0,1) | Details |