| Name | beta adrenergic receptor (protein family or complex) |
|---|---|
| Synonyms | Beta adrenoceptor; Beta adrenoceptor; Beta adrenergic receptor; Beta adrenergic receptors; Beta adrenoceptor; Beta adrenoceptors; Beta adrenoceptors |
| Name | chloralose |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 12621527 | Moosavi SM, Johns EJ: The effect of isoprenaline infusion on renal renin and angiotensinogen gene expression in the anaesthetised rat. Exp Physiol. 2003 Mar;88(2):221-7. In this study, we investigated the ability of acute infusions of isoprenaline to alter renin and angiotensinogen gene expression in the kidney of rats anaesthetised with chloralose-urethane. Groups of rats received I.V. infusions of either saline or the beta-adrenoceptor agonist isoprenaline at 400 ng x kg (-1) x min (-1) for 4 h. |
2(0,0,0,2) | Details |
| 1282604 | Covert RF, Schreiber MD, Torgerson LJ: Effect of beta-adrenergic receptor antagonism on chloralose-induced hemodynamic changes in newborn lambs. J Cardiovasc Pharmacol. 1992 Dec;20(6):990-6. We conclude that tachycardia and increase in CO induced by chloralose in lambs probably are mediated by beta-adrenergic receptor stimulation, which may be direct or indirect.(ABSTRACT TRUNCATED AT 250 WORDS) |
118(1,2,3,3) | Details |
| 7727613 | Covert RF, Drummond WH: Hemodynamic interaction of chloralose pretreatment with subsequent beta-adrenergic receptor antagonism in lambs. Biol Neonate. 1994;66(6):316-23. |
112(1,2,2,2) | Details |
| 7771543 | Hill MR, Wallick DW, Martin PJ, Levy MN: Effects of repetitive vagal stimulation on heart rate and on cardiac vasoactive intestinal polypeptide efflux. Am J Physiol. 1995 May;268(5 Pt 2):H1939-46. In dogs anesthetized with alpha-chloralose, we assessed the "vagally induced tachycardia" elicited by successive 2-min periods of intense vagal stimulation (0.5 ms, 10 mA, 20 Hz) after we had blocked the animals' muscarinic and beta-adrenergic receptors with atropine and respectively. |
81(1,1,1,1) | Details |
| 7636745 | Danuser H, Bemis K, Thor KB: Pharmacological analysis of the noradrenergic control of central sympathetic and somatic reflexes controlling the lower urinary tract in the anesthetized cat. J Pharmacol Exp Ther. 1995 Aug;274(2):820-5. In the present study, we pharmacologically analyzed the role of noradrenergic systems on the sympathetic and somatic pathways to the lower urinary tract and asked: 1) Are alpha-1, alpha-2, or beta-adrenergic receptors tonically active along sympathetic and/or somatic reflex pathways? And 2) what is the net effect of increasing the extracellular levels of NE by administration of a NE reuptake inhibitor? To address these questions, we recorded evoked potentials from the central ends of the hypogastric and pudendal nerves in response to electrical stimulation of the pelvic and pudendal nerves in chloralose-anesthetized cats, and the effects of prazosin (1-300 micrograms/kg i.v.), an alpha-1-adrenergic receptor antagonist; idazoxan (1-300 micrograms/kg i.v.), an alpha-2-adrenergic receptor antagonist; (1 mg/kg i.v.), a beta-adrenergic receptor antagonist; and tomoxetine (0.003-3 mg/kg i.v.), a selective NE reuptake inhibitor, were examined. |
31(0,1,1,1) | Details |
| 1840408 | al-Obaidi M, Whitaker EM, Karim F: The effect of discrete stimulation of carotid body chemoreceptors on atrial natriuretic peptide in anaesthetized dogs. J Physiol. 1991 Nov;443:519-31. In three of the dogs, beta-adrenoceptor blockade by (2 mg kg-1, I.V.) greatly reduced the effects of chemoreceptor stimulation on plasma levels of ANP. 3. In seven chloralose-anaesthetized and artificially ventilated beagles, the carotid sinus regions were vascularly isolated and perfused with either arterial or mixed (arterial and venous) blood (PO2 46.4 +/- 1.5 mmHg, mean +/- S.E.M.) to stimulate the chemoreceptors at constant flow and pressure. |
1(0,0,0,1) | Details |
| 9728083 | Massett MP, Lewis SJ, Kregel KC: Effect of heating on the hemodynamic responses to vasoactive agents. Am J Physiol. 1998 Sep;275(3 Pt 2):R844-53. The hemodynamic responses to adrenoceptor agonists were examined in chloralose-anesthetized rats heated to colonic temperatures (Tco) of 37, 39, and 41.5 degrees C. Further heating to 41.5 degrees C markedly attenuated the hemodynamic responses to alpha- and beta-adrenoceptor agonists. |
1(0,0,0,1) | Details |
| 7621952 | Khomaziuk AI, Neshcheret OP, Shepelenko IV: [The effect of insulin on cardiac activity and on the coronary and systemic circulations]. Fiziol Zh. 1994 May-Aug;40(3-4):3-9. The studies were performed on healthy closed-chest chloralose-anaesthetized dogs using catheterization, extracorporal perfusion and resistography of coronary arteries and catheterization and continuous drainage of the coronary sinus. The second phase of the reaction is either attenuated or even absent after blockade of beta-adrenoceptors 0.5 mg/kg, i.v.). |
1(0,0,0,1) | Details |
| 8015105 | Springer JP, Kropp BP, Thor KB: Facilitatory and inhibitory effects of selective reuptake inhibitors on hypogastric nerve-evoked urethral contractions in the cat: a prominent role of urethral beta-adrenergic receptors. J Urol. 1994 Aug;152(2 Pt 1):515-9. To explore only the contribution imparted by sympathomimetic effects on the urethra by (NE) reuptake inhibition, two selective NE reuptake inhibitors (nisoxetine and tomoxetine) that possess no antimuscarinic or serotonergic properties were examined for their effects on sympathetic hypogastric nerve (HgN) evoked urethral contractions in chloralose anesthetized cats. |
2(0,0,0,2) | Details |
| 7867031 | Tanabe T, Takahashi K, Kitada M, Yoshioka K, Handa S, Mori H: Effects of sympathetic stimulation, with and without previous alpha 1 and beta adrenoceptor blockade, on refractoriness dispersion in canine heart. Cardiovasc Res. 1994 Dec;28(12):1787-93. METHODS: Chloralose anaesthetised dogs were studied 2 h after ligation of the obtuse marginal branches of the circumflex artery (OMB). |
2(0,0,0,2) | Details |
| 1723761 | Downey HF, Grice DP, Jones CE: Systemic hypoxia activates a coronary vasoconstrictor reflex response that is blocked by prazosin. J Cardiovasc Pharmacol. 1991 Nov;18(5):657-64. We assessed the presence of an alpha 1-adrenoceptor-mediated coronary vasoconstrictor reflex response during acute systemic hypoxia in eight chloralose-anesthetized dogs. Left ventricular (LV) preload, as indicated by left atrial pressure, was unchanged. beta-adrenoceptor-mediated coronary dilation and positive chronotropic and inotropic responses to hypoxia were blocked by |
1(0,0,0,1) | Details |
| 11695707 | Molinari C, Battaglia A, Grossini E, Mary DA, Ruggeri P, Surico N, Vacca G, Vassanelli C: The role of activation of the renin-angiotensin system on the reflex regional vasoconstriction caused by distension of the uterus in anaesthetized pigs. Auton Neurosci. 2001 Oct 8;93(1-2):56-64. Distension of the uterus in anaesthetized pigs has been shown to cause a reflex regional vasoconstriction and an increase in plasma renin activity (PRA) through efferent sympathetic mechanisms which respectively involved alpha- and beta-adrenergic receptors. In pigs anaesthetized with alpha-chloralose, blood flow in the left circumflex or anterior descending coronary, superior mesenteric, left renal and left external iliac arteries was assessed using electromagnetic flowmeters. |
1(0,0,0,1) | Details |
| 1356560 | Cambridge D, Whiting MV, Butterfield LJ, Allan G: The effects of combined angiotensin converting enzyme inhibition and beta-adrenoceptor blockade on plasma renin activity in anaesthetized dogs. Br J Pharmacol. 1992 Jun;106(2):342-7. The effects of beta-adrenoceptor blockade on the changes in plasma renin activity (PRA) following angiotensin enzyme (ACE) inhibition were investigated in pentobarbitone-chloralose anaesthetized dogs. 2. |
38(0,1,1,8) | Details |
| 9915601 | Vidrio H, Sanchez-Salvatori MA, Medina M: Enhanced bradycardia induced by beta-adrenoceptor antagonists in rats pretreated with isoniazid. J Auton Pharmacol. 1998 Dec;18(6):363-9. In the present study, the possible enhancement by isoniazid of bradycardia induced by beta-adrenoceptor antagonists was determined in rats anaesthetised with chloralose-urethane. |
32(0,1,1,2) | Details |
| 15120591 | Kobayashi M, Majima Y: Target site of inhibition of baroreflex vagal bradycardia by nasal stimulation. Brain Res. 2004 May 29;1009(1-2):137-46. In this study, to identify the target site of the inhibition, several types of experiments were conducted in chloralose-urethane-anesthetized, beta-adrenergic receptor-blocked rats. |
31(0,1,1,1) | Details |
| 9862392 | Vacca G, Battaglia A, Ferro R, Grossini E, Mary DA, Molinari C, Surico N: The effect of distension of the uterus on plasma renin activity (PRA) in anaesthetized pigs. J Auton Nerv Syst. 1998 Nov 10;73(2-3):163-9. The increase in PRA caused by uterine distension was abolished by bilateral section of the renal nerves (five pigs) or by blockade of beta-adrenergic receptors with The same methods were used in 14 pigs which were anaesthetized with alpha-chloralose and artificially ventilated. |
2(0,0,0,2) | Details |
| 16100243 | McClure JM, O'Leary DS, Scislo TJ: Stimulation of NTS A1 receptors evokes counteracting effects on hindlimb vasculature. Am J Physiol Heart Circ Physiol. 2005 Dec;289(6):H2536-42. Epub 2005 Aug 12. This increase in pre-ASNA causes the release of and subsequent activation of beta-adrenergic receptors that are preferentially located in the skeletal muscle vasculature. Therefore, in chloralose-urethane-anesthetized rats, we evaluated the contribution of vasodilator versus vasoconstrictor effects of stimulation of NTS A1 receptors on the hindlimb vasculature. |
2(0,0,0,2) | Details |
| 11553030 | Cheng ZB, Kobayashi M, Nosaka S: Effects of optic tract stimulation on baroreflex vagal bradycardia in rats. Clin Exp Pharmacol Physiol. 2001 Sep;28(9):721-8. In chloralose- urethane anaesthetized, beta-adrenoceptor-blocked rats, BVB was evoked by electrical stimulation of the aortic depressor nerve. 3. |
31(0,1,1,1) | Details |
| 9887031 | Kobayashi M, Cheng ZB, Nosaka S: Inhibition of baroreflex vagal bradycardia by nasal stimulation in rats. Am J Physiol. 1999 Jan;276(1 Pt 2):H176-84. In chloralose- and urethan-anesthetized, beta-adrenergic receptor-blocked rats, the aortic depressor nerves were cut and electrically stimulated to induce BVB. |
31(0,1,1,1) | Details |
| 9436824 | Tanabe T, Usui K, Kusuzaki S, Yoshitake M, Takigawa O, Iwamoto T, Handa S: Differences in refractory-period response of canine subendocardium and subepicardium to bunazosin, an alpha1-adrenoceptor antagonist, and during myocardial ischemia. J Cardiovasc Pharmacol. 1997 Dec;30(6):824-30. Our objective was to investigate the effects of alpha1- or beta-adrenoceptor blockers on endocardial and epicardial refractory-period changes during myocardial ischemia in alpha-chloralose-anesthetized dogs. |
6(0,0,1,1) | Details |
| 1348976 | Nayebpour M, Talajic M, Nattel S: Effects of beta-adrenergic receptor stimulation and blockade on rate-dependent atrioventricular nodal properties. Circ Res. 1992 May;70(5):902-11. Selective stimulation protocols and mathematical formulations were used to independently quantify AV nodal recovery, facilitation, and fatigue in 12 morphine-chloralose-anesthetized dogs. |
5(0,0,0,5) | Details |
| 8203611 | Deschamps A, Magder S: Effects of heat stress on vascular capacitance. Am J Physiol. 1994 May;266(5 Pt 2):H2122-9. To analyze these adjustments, we measured blood volumes, unstressed volumes, blood flow distribution, venous compliance, venous resistance, and the time constant of venous drainage of the splanchnic and extrasplanchnic vascular beds in dogs anesthetized with alpha-chloralose at normal and at high core temperatures. We repeated the measurements at high core temperatures with ganglionic blockade, alpha-adrenergic receptor blockade, or beta-adrenergic receptor blockade to determine the efferent neurohumoral pathway. |
1(0,0,0,1) | Details |
| 2023113 | Poucher SM, Karim F: The renal response to electrical stimulation of renal efferent sympathetic nerves in the anaesthetized greyhound. J Physiol. 1991 Mar;434:1-10. The effect of direct electrical stimulation of the renal efferent nerves upon renal haemodynamics and function was studied in greyhounds anaesthetized with chloralose and artificially ventilated. Beta-Adrenergic receptor activation was blocked by the infusion of dl- (17 micrograms kg-1 min-1). |
1(0,0,0,1) | Details |
| 14597147 | Hocht C, Opezzo JA, Taira CA: Anterior hypothalamic beta-adrenergic activity in the maintenance of hypertension in aortic coarctated rats. Pharmacol Res. 2004 Jan;49(1):17-21. Wistar urethane-chloralose anesthetized rats were used. On the other hand, the hypotensive action of isoproterenol was significantly diminished after the administration of non-specific beta-adrenoceptor antagonist in SO and ACo rats. |
1(0,0,0,1) | Details |
| 1450095 | Pettersson K, Bjork H: Inhibition of platelet accumulation by beta 1-adrenoceptor blockade in the thoracic aorta of rabbits subjected to experimental sympathetic activation. Cardiovasc Drugs Ther. 1992 Oct;6(5):505-11. Psychosocial stress has been shown to accelerate atherogenesis in animals, an effect probably mediated via beta-adrenoceptor activation. During the exposure to the labelled platelets, the animals were also exposed to 3 hours of chloralose anesthesia. |
3(0,0,0,3) | Details |
| 10831530 | Molinari C, Grossini E, Mary DA, Vacca G: Effect of distension of the gallbladder on plasma renin activity in anesthetized pigs. Circulation. 2000 May 30;101(21):2539-45. METHODS AND RESULTS: In 34 alpha-chloralose-anesthetized pigs, balloons positioned within the gallbladder were distended for 30 minutes with volumes of Ringer's solution equal to those of withdrawn bile. This increase was abolished by cervical vagotomy, section of renal nerves, or blockade of beta-adrenergic receptors. |
2(0,0,0,2) | Details |
| 10410826 | Usui K, Tanabe T, Handa S, Shinozaki Y, Mori H: Disproportional response between refractory period and blood flow to alpha 1- and beta-adrenoceptor blockade in canine ischemic myocardium. Cardiovasc Drugs Ther. 1998 Dec;12(6):561-71. Dogs were anesthetized with alpha-chloralose and divided into bunazosin (an alpha 1-blocking agent)-treated (0.1-0.2 mg/kg, i.v., n = 14), -treated (0.2 mg/kg, i.v., n = 12), and vehicle-control (n = 10) groups. |
2(0,0,0,2) | Details |
| 7485582 | Hill MR, Wallick DW, Mongeon LR, Martin PJ, Levy MN: Vasoactive intestinal polypeptide antagonists attenuate vagally induced tachycardia in the anesthetized dog. Am J Physiol. 1995 Oct;269(4 Pt 2):H1467-72. We used three vasoactive intestinal polypeptide (VIP) antagonists, VIP-(10-28), [p-Cl-D-Phe6,Leu17] VIP, and NT-VIP, to evaluate the role of VIP as a mediator of vagally induced tachycardia in chloralose-anesthetized dogs. After we administered muscarinic and beta-adrenergic receptor antagonists, we evoked vagally induced tachycardia either directly, by stimulating the vagus nerves for 2 min, or reflexly, by injecting to increase blood pressure. |
1(0,0,0,1) | Details |
| 1590463 | Broten TP, Miyashiro JK, Moncada S, Feigl EO: Role of endothelium-derived relaxing factor in parasympathetic coronary vasodilation. Am J Physiol. 1992 May;262(5 Pt 2):H1579-84. Experiments were conducted on closed-chest, alpha-chloralose-anesthetized dogs with the heart paced at a constant rate. Phentolamine and were administered to block alpha- and beta-adrenergic receptors, and was given to inhibit prostaglandin synthesis. |
1(0,0,0,1) | Details |
| 7943371 | Goodson AR, Leibold JM, Gutterman DD: Inhibition of synthesis augments centrally induced sympathetic coronary vasoconstriction in cats. Am J Physiol. 1994 Oct;267(4 Pt 2):H1272-8. Secondary effects include vasodilation due to beta-adrenoceptor stimulation and alpha 2-mediated release of endothelium-derived relaxing factor (EDRF) from the coronary vascular endothelium. In chloralose-anesthetized cats, hypothalamic stimulation produced increases in coronary vascular resistance [maximum 26 +/- 9% (SE)] and arterial pressure (41 +/- 7%) and a decrease in coronary blood flow velocity (15 +/- 6%). |
1(0,0,0,1) | Details |
| 15031132 | Osaka T: Cold-induced thermogenesis mediated by in the preoptic area of anesthetized rats. Am J Physiol Regul Integr Comp Physiol. 2004 Aug;287(2):R306-13. Epub 2004 Mar 18. Bilateral microinjections of (300 mM, 100 nl) or the (A) receptor agonist muscimol (100 microM, 100 nl) into the preoptic area (POA) of the hypothalamus increased the rate of whole body O (2) consumption (VO (2)) and the body core (colonic) temperature of urethane-chloralose-anesthetized, artificially ventilated rats. However, pretreatment with the beta-adrenoceptor (5 mg/kg i.v.) greatly attenuated the -induced increase in VO (2) and tachycardic responses. |
1(0,0,0,1) | Details |
| 12020848 | Okada S, Murakami Y, Yokotani K: Centrally applied donor elevates plasma by activation of the hypothalamic noradrenergic neurons in rats. Brain Res. 2002 Jun 7;939(1-2):26-33. The present study was undertaken to investigate the mechanisms involved in a donor [3-morpholino-sydnonimine (SIN-1)]-induced activation of hypothalamic-pituitary- axis in urethane- and alpha-chloralose-anesthetized rats. Pretreatment with phentolamine (250 microg/animal, i.c.v.), an alpha-adrenoceptor antagonist, attenuated the elevation of plasma evoked by SIN-1, but sotalol (300 microg/animal, i.c.v.), a beta-adrenoceptor antagonist, was without effects. |
1(0,0,0,1) | Details |
| 10226761 | Montastruc JL, Verwaerde P, Pelat M, Galitzky J, Langin D, Lafontan M, Berlan M: Peripheral cardiovascular actions of SR 58611 A, a beta 3-adrenoceptor agonist, in the dog: lack of central effect. Fundam Clin Pharmacol. 1999;13(2):180-6. In order to investigate the putative role of beta3-adrenoceptors in central and peripheral cardiovascular regulations, the effects of intracisternal (i.c.) and intravenous (i.v.) injections of SR 58611 A (10, 50, 100 and 200 nmol kg-1), a selective beta3-adrenoceptor agonist, were investigated in chloralose anaesthetized dogs. SR 58611 A (200 nmol kg-1) was reduced by pretreatment with beta-adrenoceptor antagonists nadolol, bupranolol or the beta3-adrenoceptor selective antagonist, SR 59230 A (2 mg kg-1 i.v.)] and suppressed after sinoaortic denervation (i.e. after removal of vagal tone to the heart). |
1(0,0,0,1) | Details |
| 12865154 | Cao WH, Morrison SF: Disinhibition of rostral raphe pallidus neurons increases cardiac sympathetic nerve activity and heart rate. Brain Res. 2003 Aug 1;980(1):1-10. We determined the cardiovascular effects of microinjecting the (A) receptor antagonist, bicuculline, into the rostral raphe pallidus (RPa) on arterial pressure (AP), heart rate (HR) and cardiac sympathetic nerve activity (CSNA) in urethane-chloralose anesthetized, artificially-ventilated rats. A similar tachycardia was evoked after removal of both adrenal glands, but was absent after beta-adrenergic receptor blockade with |
1(0,0,0,1) | Details |
| 1350625 | Abdelrahman A, Wang YX, Pang CC: Effects of anaesthetic agents on pressor response to beta-blockers in the rat. J Pharm Pharmacol. 1992 Jan;44(1):34-8. It has been shown that paradoxical pressor response to a beta-adrenoceptor antagonist occurs in conscious rats pretreated with an alpha-adrenoceptor antagonist. In the second series of experiments, a single i.v. bolus dose of was given in phentolamine-treated rats anaesthetized with pentobarbitone, amobarbitone, ketamine or chloralose. |
1(0,0,0,1) | Details |
| 8176111 | Krishnan S, Levy MN: Effects of coronary artery occlusion and reperfusion on the idioventricular rate in anesthetized dogs. J Am Coll Cardiol. 1994 May;23(6):1484-90. OBJECTIVES: We evaluated the effects of alpha 1- and beta-adrenergic receptor blockade on the changes in idioventricular rate induced by occlusion and reperfusion of the left anterior descending coronary artery in anesthetized dogs. METHODS: We injected into the AV node to induce complete AV block in 35 chloralose-anesthetized dogs. |
1(0,0,0,1) | Details |
| 1683607 | Kaplan JR, Pettersson K, Manuck SB, Olsson G: Role of sympathoadrenal medullary activation in the initiation and progression of atherosclerosis. Circulation. 1991 Dec;84(6 Suppl):VI23-32. Studies using chow-fed rabbits demonstrated that exposure to chloralose anesthesia (an agent that provokes profound sympathetic activation) induced endothelial injury (indicated by intracellular accumulation of immunoglobulin G in the aortic endothelium) and abnormal (increased) platelet accumulation. The further observation that these effects were inhibited under beta-adrenoceptor blockade implicates sympathoadrenomedullary arousal in the initiation of atherogenesis. |
1(0,0,0,1) | Details |
| 9596808 | Daly MD, Jones JF: Respiratory modulation of carotid and aortic body reflex left ventricular inotropic responses in the cat. J Physiol. 1998 Jun 15;509 ( Pt 3):895-907. The positive inotropic responses were mediated via the sympathetic nervous system, as indicated by their abolition as a result of intravenous injections of the beta-adrenoceptor blocking agent, 6. The reflex changes in the inotropic state of the left ventricle, measured as the dP/dt max (maximum rate of change of pressure), occurring in response to selective stimulation of the carotid and aortic body chemoreceptors by were studied in the cat anaesthetized with a mixture of chloralose and urethane. 2. |
1(0,0,0,1) | Details |
| 9334987 | Vacca G, Battaglia A, Grossini E, Mary DA, Molinari C, Surico N: Changes in regional blood flow in response to distension of the uterus in anaesthetised pigs. J Auton Nerv Syst. 1997 Sep 10;66(1-2):7-14. Experiments were performed in seven pigs anaesthetised with alpha-chloralose and artificially ventilated. |
0(0,0,0,0) | Details |
| 9375565 | Yasui T, Karita K, Izumi H, Tamai M: Correlation between vasodilatation and secretion in the lacrimal gland elicited by stimulation of the cornea and facial nerve root of the cat. Invest Ophthalmol Vis Sci. 1997 Nov;38(12):2476-82. METHODS: Electrical stimulation of the cornea or facial nerve root was used to evoke a blood flow increase in the lacrimal gland and tear secretion of the urethane-chloralose-anesthetized, paralyzed, and cervically sympathectomized cat. |
0(0,0,0,0) | Details |
| 9729568 | O'Halloran KD, Janssen PL, Bisgard GE: Dopaminergic modulation of respiratory motor output in peripherally chemodenervated goats. J Appl Physiol. 1998 Sep;85(3):946-54. We examined the ventilatory effects of exogenous (DA) and (NE) administration in chloralose-anesthetized, paralyzed, artificially ventilated adult goats before and after carotid body denervation (CBD). |
0(0,0,0,0) | Details |
| 8743722 | Medda BK, Koley J, Koley B: Sympathoadrenal activity in the visceral (viscerovascular) reflexes to distension of the urinary bladder. Jpn J Physiol. 1996 Feb;46(1):3-92. The experiments were performed in 37 chloralose anesthetized cats. |
0(0,0,0,0) | Details |
| 7636716 | Thor KB, Katofiasc MA: Effects of duloxetine, a combined and reuptake inhibitor, on central neural control of lower urinary tract function in the chloralose-anesthetized female cat. J Pharmacol Exp Ther. 1995 Aug;274(2):1014-24. |
0(0,0,0,0) | Details |
| 8229839 | Katafuchi T, Take S, Hori T: Roles of sympathetic nervous system in the suppression of cytotoxicity of splenic natural killer cells in the rat. J Physiol. 1993 Jun;465:343-57. To further study the mechanisms of this phenomenon, we investigated: (1) the effects of a central injection of recombinant human interferon-alpha on the electrical activity of the splenic nerve, and (2) the responses of splenic natural killer cytotoxicity on the electrical stimulation of the splenic nerve in urethane with alpha-chloralose anaesthetized rats. 2. |
0(0,0,0,0) | Details |
| 9117101 | Galinier M, Rouge P, Fourcade J, Senard JM, Albenque JP, Balanescu S, Doazan JP, Montastruc JL, Bounhoure JP, Montastruc P: Lack of haemodynamic effects of on post-capillary pulmonary hypertension induced by acute sino-aortic denervation. Br J Pharmacol. 1997 Jan;120(1):7-12. Cardiopulmonary variables of 28 male beagle dogs, anaesthetized with chloralose, 16 spontaneous breathing and 12 with assisted ventilation, were studied before and after sino-aortic denervation (SAD). |
0(0,0,0,0) | Details |