| Name | alpha 2 adrenergic |
|---|---|
| Synonyms | Alpha 2A Adrenoceptor; ZNF32; ADRA2; ADRA2A; ADRA2R; ADRAR; ALPHA2AAR; Alpha 2A adrenergic receptor… |
| Name | sodium fluoride |
|---|---|
| CAS | sodium fluoride (NaF) |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 10712398 | Perrault LP, Mahlberg F, Breugnot C, Bidouard JP, Villeneuve N, Vilaine JP, Vanhoutte PM: Hypercholesterolemia increases coronary endothelial dysfunction, lipid content, and accelerated atherosclerosis after heart transplantation. Arterioscler Thromb Vasc Biol. 2000 Mar;20(3):728-36. Endothelium-dependent relaxations to to the alpha (2)-adrenergic agonist UK14,304, and to the direct G-protein activator sodium fluoride were decreased significantly in allografted hearts compared with native hearts from both groups. |
81(1,1,1,1) | Details |
| 10536676 | Perrault LP, Bidouard JP, Janiak P, Villeneuve N, Bruneval P, Vilaine JP, Vanhoutte PM: Impairment of G-protein-mediated signal transduction in the porcine coronary endothelium during rejection after heart transplantation. Cardiovasc Res. 1999 Aug 1;43(2):457-70. Endothelium-dependent relaxations to and to the alpha 2-adrenergic agonist UK14304 (which both activate receptors coupled to Gi-proteins) and to sodium fluoride (a direct activator of G-proteins) were decreased 30 days after transplantation, while those to the ionophore, A23187, and bradykinin were shifted to the right and those to ADP were normal. |
31(0,1,1,1) | Details |
| 9229295 | Perrault LP, Bidouard JP, Janiak P, Villeneuve N, Bruneval P, Vilaine JP, Vanhoutte PM: Time course of coronary endothelial dysfunction in acute untreated rejection after heterotopic heart transplantation. J Heart Lung Transplant. 1997 Jun;16(6):643-57. Endothelium-dependent relaxations to and alpha 2-adrenergic agonist UK 14304 (which activate receptors coupled to Gi-proteins) and to sodium fluoride (a direct G-protein activator) deteriorated progressively over time. |
31(0,1,1,1) | Details |
| 8731456 | Karege F, Bovier P, Rudolph W, Gaillard JM: Platelet phosphoinositide signaling system: an overstimulated pathway in depression. Biol Psychiatry. 1996 Apr 15;39(8):697-702. In order to test a possible depression-associated defect in signal transduction, platelet alpha 2-adrenergic-mediated phosphoinositide (PI) hydrolysis was measured, both in drug-free major depressed patients and in control healthy subjects. Thrombin (2 units) and 10 mM sodium fluoride (NaF) also induced an increase in 3H-PIP2 metabolites. |
1(0,0,0,1) | Details |