| Name | glucagon |
|---|---|
| Synonyms | GCG; GLP 1; GLP1; GLP 2; GLP2; GRPP; Glicentin related polypeptide; Glucagon… |
| Name | sodium fluoride |
|---|---|
| CAS | sodium fluoride (NaF) |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 15306227 | Hashim S, Li Y, Nagakura A, Takeo S, Anand-Srivastava MB: Modulation of G-protein expression and adenylyl cyclase signaling by high in vascular smooth muscle. Cardiovasc Res. 2004 Sep 1;63(4):709-18. On the other hand, the stimulatory effects of GTPgammaS, glucagon, isoproterenol, FSK and sodium fluoride on adenylyl cyclase were significantly augmented in hyperglycemic cells as compared to control cells, whereas basal adenylyl cyclase activity was significantly lower in hyperglycemic cells as compared to control cells. |
6(0,0,1,1) | Details |
| 11311219 | Evans MJ, Livesey JH, Ellis MJ, Yandle TG: Effect of anticoagulants and storage temperatures on stability of plasma and serum hormones. Clin Biochem. 2001 Mar;34(2):107-12. DESIGN AND METHODS: Human blood samples were collected from volunteers into EDTA, lithium / or tubes without anticoagulant, plasma and serum left at -20 degrees C, 4 degrees C or 30 degrees C for 24 and 120 hours then assayed for ACTH, alpha-subunit, AVP, CRH, C-peptide, FSH, glucagon, GH, IGF-1, IGFBP-3, insulin, leptin, LH, PPP, PTH, prolactin and VIP, or at room temperature for 0 to 72 hours (BNP, NT-BNP)(n = 6 per condition). |
1(0,0,0,1) | Details |
| 20141037 | Derkach KV, Shpakov AO, Uspenskaia ZI, Kuznetsova LA: [Inhibition of the adenylyl cyclase signaling system by colchicine and vinblastine in ciliate Dileptus anser]. Tsitologiia. 2009;51(12):1013-8. The treatment of D. anser with colchicine and vinblastine (10 (-5)-10 (-6) M) weakly influenced basal activity of AC, but caused essential decrease or complete blocking of AC stimulation by non-hormonal (GppNHp, sodium fluoride) and hormonal (aldrenaline, glucagon) agents. |
31(0,1,1,1) | Details |
| 14754571 | Meyer A, Seidler FJ, Slotkin TA: Developmental effects of chlorpyrifos extend beyond neurotoxicity: critical periods for immediate and delayed-onset effects on cardiac and hepatic cell signaling. Environ Health Perspect. 2004 Feb;112(2):170-8. In addition to basal AC activity, we assessed the responses to direct AC stimulants (forskolin, Mn2+); to isoproterenol and glucagon, which activate signaling through specific membrane receptors; and to sodium fluoride, which activates the G-proteins that couple the receptors to AC. |
31(0,1,1,1) | Details |
| 8927025 | Anand-Srivastava MB, McNeill JH, Yang XP: Reversal of defective G-proteins and adenylyl cyclase/cAMP signal transduction in diabetic rats by vanadyl sulphate therapy. Mol Cell Biochem. 1995 Dec 6-20;153(1-2):113-9. The basal enzyme activity, as well as the stimulatory effects of nucleotides, glucagon, N-Ethylcarboxamideadenosine (NECA), isoproterenol, forskolin and sodium fluoride (NaF) on adenylyl cyclase were significantly increased in STZ-D rat liver as compared to control. |
7(0,0,1,2) | Details |
| 11900377 | Anand-Srivastava MB, Wang R, Liu YY: Alterations in g-protein-linked signal transduction in vascular smooth muscle in diabetes. Adv Exp Med Biol. 2001;498:263-71. On the other hand, the stimulatory effects of GTPgammaS, isoproterenol, glucagon, forskolin (FSK) and sodium fluoride on adenylyl cyclase were enhanced in STZ-rats. |
6(0,0,1,1) | Details |
| 10608476 | Ge C, Garcia R, Anand-Srivastava MB: Altered expression of Gi-protein and adenylyl cyclase activity in hearts from one kidney one clip hypertensive rats: effect of captopril. J Hypertens. 1999 Nov;17(11):1617-26. 5'-[3'-thio] (GTPgammaS), isoproterenol, glucagon, sodium fluoride (NaF) and forskolin (FSK) stimulated adenylyl cyclase activity in hearts from control and hypertensive rats to varying degrees; however, the stimulations were significantly less in hypertensive rats compared with control rats. |
6(0,0,1,1) | Details |
| 15106810 | Hashim S, Anand-Srivastava MB: Losartan-induced attenuation of blood pressure in L-NAME hypertensive rats is associated with reversal of the enhanced expression of Gi alpha proteins. J Hypertens. 2004 Jan;22(1):181-90. In addition, the stimulatory effects of 5'-gamma-thiotriphosphate (GTPgammaS), isoproterenol, 5'-N-ethylcarboxamideadenosine (NECA), glucagon, forskolin (FSK) and sodium fluoride (NaF) on adenylyl cyclase, which were diminished in L-NAME-treated rats, were reversed by losartan treatment. |
6(0,0,1,1) | Details |
| 15779331 | Shpakov AO, Korol'kov VI, Plesneva SA, Kuznetsova LA, Pertseva MN: Effects of the C-terminal peptide of the alphaS subunit of the G protein on the regulation of adenylyl cyclase and protein kinase A activities by biogenic amines and glucagon in mollusk and rat muscles. Neurosci Behav Physiol. 2005 Feb;35(2):177-86. |
2(0,0,0,2) | Details |
| 11007313 | Seiyama A, Kosaka H: Effect of glycolysis on pancreatic microcirculation and cellular functions in anesthetized rats. Pflugers Arch. 2000 Sep;440(5):721-6. Inhibition of glycolysis, by superfusing the pancreas of anesthetized rats with 2-deoxyglucose (10 mM) or sodium fluoride (2 mM), significantly decreased O2 release from erythrocytes flowing in the microvessels by 30-40%. Inhibiting glycolysis decreased blood levels by approximately or = to 40% and increased glucagon release twofold. |
1(0,0,0,1) | Details |
| 8985310 | Scarpace PJ, Dove J, Matheny M: Effects of age on beta adrenergic subtype activation of adenylyl cyclase in brown adipose tissue. Proc Soc Exp Biol Med. 1996 Dec;213(3):262-7. Adenylyl cyclase stimulated by the nonspecific agonist, isoproterenol, and by the specific beta3 agonist, BRL 37344, also declined by 50% with age, whereas glucagon stimulation decreased by more than 70%. |
1(0,0,0,1) | Details |
| 16998537 | Ge C, Garcia R, Anand-Srivastava MB: Enhanced expression of Gialpha protein and adenylyl cyclase signaling in aortas from 1 kidney 1 clip hypertensive rats. Can J Physiol Pharmacol. 2006 Jul;84(7):739-46. GTPgammaS, isoproterenol, glucagon, NaF, and FSK stimulated the AC activity in aortas from control and hypertensive rats to varying degrees; however, the stimulations were significantly lower in hypertensive rats than in control rats. |
1(0,0,0,1) | Details |