| Name | glial fibrillary acidic protein |
|---|---|
| Synonyms | GFAP; Glial fibrillary acidic protein; Glial fibrillary acidic proteins |
| Name | carbon tetrachloride |
|---|---|
| CAS | tetrachloromethane |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 19747501 | Domitrovic R, Jakovac H, Tomac J, Sain I: Liver fibrosis in mice induced by carbon tetrachloride and its reversion by Toxicol Appl Pharmacol. 2009 Dec 15;241(3):311-21. Epub 2009 Sep 8. Concomitantly, the expression of glial fibrillary acidic protein and alpha-smooth muscle actin indicated deactivation of hepatic stellate cells. |
1(0,0,0,1) | Details |
| 18042797 | Miyata E, Masuya M, Yoshida S, Nakamura S, Kato K, Sugimoto Y, Shibasaki T, Yamamura K, Ohishi K, Nishii K, Ishikawa F, Shiku H, Katayama N: Hematopoietic origin of hepatic stellate cells in the adult liver. . Blood. 2008 Feb 15;111(4):2427-35. Epub 2007 Nov 27. To test this hypothesis, we generated chimeric mice by transplantation of clonal populations of cells derived from single enhanced green fluorescent protein (EGFP)-marked Lin (-) Sca-1 (+) c-kit (+) CD34 (-) cells and examined the histology of liver tissues obtained from the chimeric mice with carbon tetrachloride (CCl (4))-induced injury. |
0(0,0,0,0) | Details |
| 15382010 | Roskams T, Cassiman D, De Vos R, Libbrecht L: Neuroregulation of the neuroendocrine compartment of the liver. . Anat Rec A Discov Mol Cell Evol Biol. 2004 Sep;280(1):910-23. Progenitor cells express chromogranin-A and neural cell adhesion molecule, parathyroid hormone-related peptide, S-100 protein, neurotrophins, and neurotrophin receptors, while hepatic stellate cells express synaptophysin, glial fibrillary acidic protein, neural cell adhesion molecule, nestin, neurotrophins, and their receptors. Moreover, inhibition of the SNS reduced fibrosis in carbon tetrachloride-induced liver injury. |
1(0,0,0,1) | Details |
| 18509379 | Chen SW, Chen YX, Zhang XR, Qian H, Chen WZ, Xie WF: Targeted inhibition of platelet-derived growth factor receptor-beta subunit in hepatic stellate cells ameliorates hepatic fibrosis in rats. Gene Ther. 2008 Nov;15(21):1424-35. Epub 2008 May 29. The double-staining immunofluorescence examination indicated that GFAP promoter could target the transgene expression into HSCs in carbon tetrachloride induced acute injured rat's liver and bile duct ligation (BDL)-induced chronic injured rat's liver. |
83(1,1,1,3) | Details |
| 16640026 | Jiroutova A, Majdiakova L, Cermakova M, Kohlerova R, Kanta J: Expression of cytoskeletal proteins in hepatic stellate cells isolated from normal and cirrhotic rat liver. Acta Medica (Hradec Kralove). 2005;48(3-4):137-44. We have studied the expression of alpha-smooth muscle actin and intermediate filament proteins vimentin, desmin and glial fibrillary acidic protein (GFAP) by immunocytochemistry in HSC cultured for 2 or 7 days after isolation. Liver cirrhosis was produced in rats by repeated carbon tetrachloride administration. |
2(0,0,0,2) | Details |
| 14739096 | Baba S, Fujii H, Hirose T, Yasuchika K, Azuma H, Hoppo T, Naito M, Machimoto T, Ikai I: Commitment of bone marrow cells to hepatic stellate cells in mouse. J Hepatol. 2004 Feb;40(2):255-60. We then examined the histology of liver tissues obtained from BM-transplanted mice with and without carbon tetrachloride-induced injury. These cells expressed the HSC lineage markers, such as desmin and glial fibrillary acidic protein (GFAP), by both RT-PCR and immunostaining. |
2(0,0,0,2) | Details |