| Name | IL 8 |
|---|---|
| Synonyms | 3 10C; AMCF I; CXC chemokine ligand 8; CXCL 8; CXCL8; Emoctakin; GCP 1; GCP1… |
| Name | sulfur |
|---|---|
| CAS | sulfur |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 19748599 | Pourfarzam S, Ghazanfari T, Yaraee R, Ghasemi H, Hassan ZM, Faghihzadeh S, Ardestani SK, Kariminia A, Fallahi F, Soroush MR, Merasizadeh J, Mahlojirad M, Naghizadeh MM, Ghanei M: Serum levels of IL-8 and IL-6 in the long term pulmonary complications induced by sulfur mustard: Sardasht-Iran Cohort Study. Int Immunopharmacol. 2009 Dec;9(13-14):1482-8. Epub 2009 Sep 11. |
10(0,0,1,5) | Details |
| 19373630 | Yang YF, Hsu JY, Fu LS, Weng YS, Chu JJ: Asthma drugs counter-regulate interleukin-8 release stimulated by in an A549 cell line. J Asthma. 2009 Apr;46(3):238-43. Sulfites are formed at bronchial mucosa from inhaled sulfur dioxide. |
9(0,0,0,9) | Details |
| 19735741 | Ghazanfari T, Yaraee R, Kariminia A, Ebtekar M, Faghihzadeh S, Vaez-Mahdavi MR, Rezaei A, Vojgani M, Soroush MR, Kermani-Jalilvand A, Mohammadi P, Foroutan A, Hassan ZM: Alterations in the serum levels of chemokines 20 years after sulfur mustard exposure: Sardasht-Iran Cohort Study. Int Immunopharmacol. 2009 Dec;9(13-14):1471-6. Epub 2009 Sep 6. The serum levels of four important and well characterized inflammatory chemokines including MCP-1/CCL2, RANTES/CCL5, IL-8/CXCL8 and Fractalkine/CX3CL1 were evaluated in sulfur mustard (SM) exposed Iranian population 20 years after exposure. |
8(0,0,1,3) | Details |
| 19932683 | Chen W, Kajiya M, Giro G, Ouhara K, Mackler HE, Mawardi H, Boisvert H, Duncan MJ, Sato K, Kawai T: Bacteria-derived promotes IL-8 production from epithelial cells. Biochem Biophys Res Commun. 2010 Jan 1;391(1):645-50. Epub 2009 Nov 22. (H (2) S), a volatile sulfur compound, is implicated as a cause of inflammation, especially when it is produced by bacteria colonizing gastrointestinal organs. |
4(0,0,0,4) | Details |
| 19776584 | Lee JS, Kwon KA, Jung HS, Kim JH, Hahm KB: Korea red ginseng on Helicobacter pylori-induced halitosis: newer therapeutic strategy and a plausible mechanism. Digestion. 2009;80(3):192-9. Epub 2009 Sep 16. BACKGROUND: Gas chromatographic documentation of volatile sulfur compounds in Helicobacter pylori cultures and the amelioration of halitosis after eradication suggested a causal link between H. pylori infection and halitosis. The expressions of cystathionine gamma-lyase (CSE), beta-synthetase (CBS), IL-6, IL-8 and IL-1beta mRNA were compared in H. pylori-infected or NaHS-treated gastric epithelial cells according to red ginseng treatment. |
2(0,0,0,2) | Details |
| 19651324 | Kehe K, Balszuweit F, Steinritz D, Thiermann H: Molecular toxicology of sulfur mustard-induced cutaneous inflammation and blistering. Toxicology. 2009 Sep 1;263(1):12-9. Epub 2009 Jan 23. Additionally, SM upregulates many inflammatory mediators including interleukin (IL)-1alpha, IL-1beta, IL-6, IL-8, tumor necrosis factor-alpha (TNF-alpha) and others. |
1(0,0,0,1) | Details |
| 19887117 | Mishra NC, Rir-sima-ah J, March T, Weber W, Benson J, Jaramillo R, Seagrave JC, Schultz G, Grotendorst G, Sopori M: Sulfur mustard induces immune sensitization in hairless guinea pigs. Int Immunopharmacol. 2010 Feb;10(2):193-9. Epub 2009 Nov 1. To that end, in this communication, we show that dermal exposure of euthymic hairless guinea pigs induced infiltration of both CD4 (+) and CD8 (+) T cells into the SM-exposed skin and strong upregulated expression of proinflammatory cytokines and chemokines (TNF-alpha, IFN-gamma, and IL-8) in distal tissues such as the lung and the lymph nodes. |
1(0,0,0,1) | Details |
| 19728558 | Markovic M, Majkic-Singh N, Ignjatovic S: Beneficial effects of cellular stress response in traditional spa treatment of rheumatoid arthritis. Clin Lab. 2009;55(5-6):235-41. We investigated the effect of hyperthermia and sulfur as possible stress factors at transcriptional level in several proinflammatory genes in fibroblast like synoviocytes. Stimulation of the cells with IL1beta induced a series of proinflammatory genes (IL1alpha, IL1beta, TNFalpha, IL8, monocyte chemoattractant peptide-1 and COX-2), but if the cells were treated with hyperthermia prior to IL1beta expression, gene expressions were significantly decreased up to 8 h. |
1(0,0,0,1) | Details |
| 19748996 | Pohl C, Papritz M, Moisch M, Wubbeke C, Hermanns MI, Uboldi C, Dei-Anang J, Mayer E, Kirkpatrick CJ, Kehe K: Acute morphological and toxicological effects in a human bronchial coculture model after sulfur mustard exposure. Toxicol Sci. 2009 Dec;112(2):482-9. Epub 2009 Sep 11. Changes in the proinflammatory cytokines interleukin (IL)-6 and IL-8 were also observed. |
1(0,0,0,1) | Details |
| 19733693 | Amiri S, Ghazanfari T, Yaraee R, Salimi H, Ebtekar M, Shams J, Ghasemi H, Pourfarzam S, Moin A, Sharifnia Z, Soroush MR, Faghihzadeh S, Hassan ZM: Serum levels of GM-CSF 20 years after sulfur mustard exposure: Sardasht-Iran Cohort Study. Int Immunopharmacol. 2009 Dec;9(13-14):1499-503. Epub 2009 Sep 4. |
0(0,0,0,0) | Details |