PubMed |
Abstract |
RScore(About this table) |
15228952 |
Abdollahi M, Donyavi M, Pournourmohammadi S, Saadat M: Hyperglycemia associated with increased hepatic glycogen phosphorylase and phosphoenolpyruvate carboxykinase in rats following subchronic exposure to malathion. Comp Biochem Physiol C Toxicol Pharmacol. 2004 Apr;137(4):343-7.
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7(0,0,1,2) |
Details |
16621213 |
Rezg R, Mornagui B, El-Arbi M, Kamoun A, El-Fazaa S, Gharbi N: Effect of subchronic exposure to malathion on glycogen phosphorylase and hexokinase activities in rat liver using native PAGE. Toxicology. 2006 Jun 1;223(1-2):9-14. Epub 2006 Apr 18.
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7(0,0,1,2) |
Details |
2095780 |
Matin MA, Sattar S, Husain K: Modification of malathion induced neurochemical changes by adrenalectomy in rats. Mol Chem Neuropathol. 1990 Aug-Oct;13(1-2):119-28.
The activities of cholinesterase and succinic dehydrogenase were reduced whereas those of glycogen phosphorylase, phosphoglucomutase, and hexokinase were increased; the lactate content of brain was also increase. |
1(0,0,0,1) |
Details |
3579975 |
Matin MA, Husain K: Cerebral glycogenolysis and glycolysis in malathion-treated hyperglycaemic animals. Biochem Pharmacol. 1987 Jun 1;36(11):1815-7.
The activities of glycogenolytic enzymes (glycogen phosphorylase and phosphoglucomutase) were increased significantly in the brain, whereas that of glucose-6-phosphatase remained unchanged. |
1(0,0,0,1) |
Details |