| Name | protein kinase A (protein family or complex) |
|---|---|
| Synonyms | Protein kinase A; cAMP dependent protein kinase; cAMP dependent protein kinases |
| Name | sodium azide |
|---|---|
| CAS | sodium azide |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 11583853 | Gehin G, Bonaly R, Coulon J: The role of in the Kluyveromyces bulgaricus flocculation phenomenon: transduction by cAMP-dependent protein kinase pathway?. FEMS Microbiol Lett. 2001 Sep 25;203(2):229-33. |
2(0,0,0,2) | Details |
| 17845509 | Lin PL, Lu KL, Lee YL, Chen YH, Chang YC, Chou HN, Tsai MC: Bursts of potential elicited by d-amphetamine in central snail neuron: effect of sodium azide. Basic Clin Pharmacol Toxicol. 2007 Oct;101(4):269-76. The BoP elicited by NaN (3) (30 microM) and d-amphetamine (135 microM) were decreased following treatment with KT5720 (protein kinase A inhibitor), or intracellular injection of EGTA [ethylene glycol-bis (2-aminoethyl ether)-N,N,N',N'-tetraacetic acid]. |
2(0,0,0,2) | Details |
| 15983831 | Rikhvanov EG, Varakina NN, Rusaleva TM, Rachenko EI, Knorre DA, Voinikov VK: Do mitochondria regulate the heat-shock response in Saccharomyces cerevisiae?. Curr Genet. 2005 Jul;48(1):44-59. Epub 2005 Jun 28. It is suggested that cAMP-dependent protein kinase A is participating in the mitochondrial regulation of nuclear genes. |
1(0,0,0,1) | Details |
| 10634941 | Brasaemle DL, Levin DM, Adler-Wailes DC, Londos C: The lipolytic stimulation of 3T3-L1 adipocytes promotes the translocation of hormone-sensitive lipase to the surfaces of lipid storage droplets. Biochim Biophys Acta. 2000 Jan 17;1483(2):251-62. Hormone-sensitive lipase translocation and lipolysis were inhibited by N-ethylmaleimide and a combination of deoxyglucose and sodium azide. Hormonal stimulation of cAMP formation and the activation of cAMP-dependent protein kinase leads to the phosphorylation of hormone-sensitive lipase and a large increase in lipolysis in adipocytes. |
1(0,0,0,1) | Details |
| 7531437 | Berridge MV, Tan AS: Interleukin-3 facilitates transport in a myeloid cell line by regulating the affinity of the transporter for involvement of protein phosphorylation in transporter activation. Biochem J. 1995 Feb 1;305 ( Pt 3):843-51. Additionally, the phosphatase inhibitor, orthovanadate, stimulated control and IL-3-dependent transport by 50-80% while the protein kinase A inhibitor, KT5720, inhibited transport by about 20% at plateau values. Inhibition of DNA synthesis with mitomycin C or with the respiratory poison, sodium azide, did not affect the ability of IL-3 to promote transport. |
1(0,0,0,1) | Details |
| 16770322 | Shahidullah M, Delamere NA: NO donors inhibit Na,K-ATPase activity by a protein kinase G-dependent mechanism in the nonpigmented ciliary epithelium of the porcine eye. Br J Pharmacol. 2006 Jul;148(6):871-80. Epub 2006 Jun 12. NO donors, sodium nitroprusside (SNP, 1 microM-1 mM), sodium azide (100 nM-1 microM) and S-nitroso-N-acetylpenicillamine (1 microM-1 mM) caused significant concentration-dependent inhibition of Na,K-ATPase activity. |
0(0,0,0,0) | Details |
| 10455294 | Shahidullah M, Wilson WS: Atriopeptin, sodium azide and cyclic GMP reduce secretion of aqueous humour and inhibit intracellular release in bovine cultured ciliary epithelium. Br J Pharmacol. 1999 Jul;127(6):1438-46. |
0(0,0,0,0) | Details |
| 10213179 | Pedreno J, Vila M, Masana L: Mechanisms for regulating platelet high density lipoprotein type3 binding sites: evidence that binding sites are downregulated by a protein kinase C-dependent mechanism. Thromb Res. 1999 Apr 1;94(1):33-44. Nevertheless, neither inhibitors of binding sites recycling or of pinocytosis, such as ammonium chloroquine, monensin, colchicine, and sodium azide, modified the binding characteristics. |
0(0,0,0,0) | Details |
| 11547566 | Nakano H, Gasparro FP, Uitto J: UVA-340 as energy source, mimicking natural sunlight, activates the transcription factor AP-1 in cultured fibroblasts: evidence for involvement of protein kinase-C. Photochem Photobiol. 2001 Aug;74(2):274-82. Free radical quenchers, sodium azide and did not affect the AP-1 binding activity. |
0(0,0,0,0) | Details |