| Name | Ach 1 |
|---|---|
| Synonyms | ACH 1; Long chain acyl CoA thioester hydrolase; Acyl CoA thioesterase 2; ACH1; ACOT 7; ACOT7; ACT; Acyl CoA thioesterase 7… |
| Name | 4-aminopyridine |
|---|---|
| CAS | 4-pyridinamine |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 19272127 | Shih HC, Hsu CS, Yang LL: In vitro study of the tocolytic effect of oroxylin A from Scutellaria baicalensis root. J Biomed Sci. 2009 Mar 4;16:27. Contractions of the uterus were induced with (Ach) (1 microM), PGF2alpha (0.1 microM), (10-3 U/ml), KCl (56.3 mM), tetraethylammonium (TEA; 1 and 10 mM), 4-aminopyridine (4-AP; 5 mM), glipizide (30 microM), a synthase (NOS) inhibitor (LNNA; 10-3M), a beta-receptor blocker 10 microM), and a cyclooxygenase inhibitor (indomethacin; 60 microM). |
31(0,1,1,1) | Details |
| 11217060 | Kwon SC: Mechanisms of NO-resistant relaxation induced by in rabbit renal arteries. J Vet Med Sci. 2001 Jan;63(1):37-40. (ACh, 1 nM-10 microM) induced endothelium-dependent relaxation of arterial rings precontracted with (NE, 1 microM) in a concentration-dependent manner. The -induced relaxation observed in the presence of L-NAME was significantly reduced by a combination of iberiotoxin (0.1 microM) and apamin (1 microM), and almost completely blocked by 4-aminopyridine (5 mM). |
1(0,0,0,1) | Details |
| 11682450 | Ghisdal P, Morel N: Cellular target of voltage and -dependent K (+) channel blockers involved in EDHF-mediated responses in rat superior mesenteric artery. Br J Pharmacol. 2001 Nov;134(5):1021-8. In arteries contracted with prostaglandin F (2 alpha) (2.5 - 10 microM), relaxation evoked by (0.01 - 3 microM) was abolished by a combination of charybdotoxin (ChTX, 0.1 microM) plus apamin (Apa, 0.1 microM) and was inhibited by 68+/-6% (n=6) by 4-aminopyridine (4-AP, 5 mM). 3. SMCs hyperpolarization to ACh (1 microM) was abolished by high K (+) solution or by ChTX/Apa. |
3(0,0,0,3) | Details |