| Name | thrombin |
|---|---|
| Synonyms | Coagulation factor II; Coagulation factor II variant; F2; F2 protein; F2 protein precursor; Factor II; PT; Prothrombin… |
| Name | sodium fluoride |
|---|---|
| CAS | sodium fluoride (NaF) |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 10983468 | Narita M, Hino M, Takubo T, Tatsumi N: Analogues of ethylenediaminetetraacetic acid and sodium fluoride as anticoagulants. Osaka City Med J. 2000 Jun;46(1):71-87. EGTA could be used for hematology and coagulation tests, although platelet count was significantly decreased in comparison with EDTA treated blood due to platelet aggregation, although prothrombin time was prolonged with EGTA-treated blood. |
1(0,0,0,1) | Details |
| 11211191 | Demers P, Elkouri S, Sirois MG, Cartier R: Coronary artery endothelial dysfunction after ischemia-reperfusion and acute untreated rejection in a canine heterotopic heart transplantation model. Transplantation. 2001 Jan 15;71(1):26-32. Endothelial relaxations to sodium fluoride (Gi-protein activator) was significantly reduced in group 1 and significantly increased in group 2 compared with control. Responses to thrombin were decreased in both groups. |
1(0,0,0,1) | Details |
| 8719419 | Akiba S, Nagatomo R, Ishimoto T, Sato T: Effect of berbamine on cytosolic phospholipase A2 activation in rabbit platelets. Eur J Pharmacol. 1995 Nov 30;291(3):343-50. These results suggest that berbamine may impair GTP-binding protein-mediated activation of cytosolic phospholipase A2, probably without influencing the enzyme translocation to membranes or the increase in the enzyme activity, and thus may cause the suppression of thrombin-induced liberation. |
1(0,0,0,1) | Details |
| 16414982 | Bogatcheva NV, Wang P, Birukova AA, Verin AD, Garcia JG: Mechanism of -induced MAP kinase activation in pulmonary artery endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2006 Jun;290(6):L1139-45. Epub 2006 Jan 13. Because we have previously shown that Ca (2+)/calmodulin-dependent protein kinase II (CaMKII) participates in Erk activation in thrombin-challenged cells, we next explored if CaMKII is involved in NaF-induced EC responses. |
1(0,0,0,1) | Details |
| 8926050 | Koller M, Hilger RA, Konig W: Effect of the PAF-receptor antagonist SM-12502 on human platelets. . Inflammation. 1996 Feb;20(1):71-85. After incubation of human platelet with different concentrations of SM-12502 the cells were subsequently stimulated with either the Ca ionophore A23187, with human thrombin, or with an activator of heterotrimeric G-proteins, sodium fluoride (NaF, in the presence of Al3+). |
81(1,1,1,1) | Details |
| 8731456 | Karege F, Bovier P, Rudolph W, Gaillard JM: Platelet phosphoinositide signaling system: an overstimulated pathway in depression. Biol Psychiatry. 1996 Apr 15;39(8):697-702. Thrombin (2 units) and 10 mM sodium fluoride (NaF) also induced an increase in 3H-PIP2 metabolites. |
32(0,1,1,2) | Details |
| 11740972 | Pandey GN, Ren X, Pandey SC, Dwivedi Y, Sharma R, Janicak PG: Hyperactive phosphoinositide signaling pathway in platelets of depressed patients: effect of desipramine treatment. Psychiatry Res. 2001 Dec 15;105(1-2):23-32. To examine if the PI signaling system is altered in depression, we studied thrombin- and sodium fluoride-stimulated (1) (IP (1)) formation before and during desipramine (DMI) treatment in platelets of depressed patients and normal control subjects. |
13(0,0,2,3) | Details |
| 8647917 | Suzuki A, Kozawa O, Shinoda J, Watanabe Y, Saito H, Oiso Y: Thrombin induces proliferation of osteoblast-like cells through hydrolysis. J Cell Physiol. 1996 Jul;168(1):209-16. |
13(0,0,0,13) | Details |
| 9167640 | Ren S, Cockell KA, Fenton JW 2nd, Angel A, Shen GX: G proteins and phospholipase C mediate thrombin-induced generation of plasminogen activator inhibitor-1 from vascular smooth muscle cells. J Vasc Res. 1997 Mar-Apr;34(2):82-9. Sodium fluoride (1 mM) stimulated PAI-1 generation from BASMC. |
9(0,0,0,9) | Details |
| 9806741 | Verin AD, Gilbert-McClain LI, Patterson CE, Garcia JG: Biochemical regulation of the nonmuscle myosin light chain kinase isoform in bovine endothelium. Am J Respir Cell Mol Biol. 1998 Nov;19(5):767-76. To evaluate mechanisms of endothelial cell (EC) barrier dysfunction evoked by the serine protease thrombin, we studied the regulation of the 214-kD EC MLCK isoform expressed in bovine endothelium. |
3(0,0,0,3) | Details |