| Name | cholinesterase |
|---|---|
| Synonyms | Acylcholine acylhydrolase; BCHE; BCHE protein; Butyrylcholine esterase; Butyrylcholinesterase; CHE1; Choline esterase II; Cholinesterase… |
| Name | dimethoate |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 8215602 | Lopez-Carillo L, Lopez-Cervantes M: Effect of exposure to organophosphate pesticides on serum cholinesterase levels. Arch Environ Health. 1993 Sep-Oct;48(5):359-63. |
4(0,0,0,4) | Details |
| 8222687 | De Bleecker J, Van den Neucker K, Colardyn F: Intermediate syndrome in organophosphorus poisoning: a prospective study. Crit Care Med. 1993 Nov;21(11):1706-11. Agents such as methylparathion, fenthion, and dimethoate carry a high risk, but we also noted a prolonged intermediate syndrome in an ethyl-parathion-poisoned patient. OBJECTIVES: To estimate the frequency of the intermediate syndrome in organophosphorus-poisoned patients, and examine its relationship to cholinesterase inhibition and electromyographic findings. |
3(0,0,0,3) | Details |
| 16243090 | Eddleston M, Eyer P, Worek F, Mohamed F, Senarathna L, von Meyer L, Juszczak E, Hittarage A, Azhar S, Dissanayake W, Sheriff MH, Szinicz L, Dawson AH, Buckley NA: Differences between organophosphorus insecticides in human self-poisoning: a prospective cohort study. Lancet. 2005 Oct 22-28;366(9495):1452-9. METHODS: We prospectively studied 802 patients with chlorpyrifos, dimethoate, or fenthion self-poisoning admitted to three hospitals. Blood cholinesterase activity and insecticide concentration were measured to determine the compound and the patients' response to insecticide and therapy. |
1(0,0,0,1) | Details |
| 985225 | Stone BF, Wilson JT, Youlton NJ: Linkage and dominance characteristics of genes for resistance to organophosphorus acaricides and allelic inheritance of decreased brain cholinesterase activity in three strains of the cattle tick, Boophilus microplus. Aust J Biol Sci. 1976 Jul;29(3):251-63. Resistance to the organophosphorus acaricides diazinon, dimethoate and formothion in the Biarra (B), Mackay (M) and Ridgelands (R) strains respectively of the cattle tick B. microplus has been shown previously to be controlled in each strain by a single incompletely dominant autosomal genetic factor. |
1(0,0,0,1) | Details |
| 19450343 | Buckley N, Eddleston M: poisoning. Clin Evid. 2007 Dec 4;2007. pii: 2101. INTRODUCTION: Acute organophosphorus poisoning occurs after dermal, respiratory, or oral exposure to either low-volatility pesticides (e.g. chlorpyrifos, dimethoate) or high-volatility nerve gases (e.g. sarin, tabun). CONCLUSIONS: In this systematic review we present information relating to the effectiveness and safety of the following interventions: activated charcoal, alpha2 adrenergic receptor agonists, atropine, benzodiazepines, butyrylcholinesterase replacement therapy, cathartics, extracorporeal clearance, gastric lavage, glycopyrronium bromide, ipecacuanha, magnesium sulphate, milk or other home remedies, N-methyl-D-aspartate receptor antagonists, organophosphorus hydrolases, oximes, sodium bicarbonate, washing the poisoned person and removing contaminated clothing. |
1(0,0,0,1) | Details |
| 19454054 | : Organophosphorus poisoning (acute). Clin Evid. 2007 Mar 1;2007. pii: 2102. INTRODUCTION: Acute organophosphorus poisoning occurs after dermal, respiratory, or oral exposure to either low-volatility pesticides (e.g. chlorpyrifos, dimethoate) or high-volatility nerve gases (e.g. sarin, tabun). CONCLUSIONS: In this systematic review we present information relating to the effectiveness and safety of the following interventions: activated charcoal, alpha2 adrenergic receptor agonists, atropine, benzodiazepines, butyrylcholinesterase replacement therapy, cathartics, extracorporeal clearance, gastric lavage, glycopyrronium bromide, ipecacuanha, magnesium sulphate, milk or other home remedies, N-methyl-D-aspartate receptor antagonists, organophosphorus hydrolases, oximes, sodium bicarbonate, washing the poisoned person and removing contaminated clothing. |
1(0,0,0,1) | Details |
| 11802337 | Vos EM, Hens JJ, Lau HS, Boon ES, Bartelink AK: [Poisoning with organophosphate compounds] . Ned Tijdschr Geneeskd. 2002 Jan 5;146(1):34-7. A 50-year-old man swallowed 200 ml of an insecticide containing the organophosphates dimethoate and phenitrotion in an attempted suicide. Although the serum concentrations of both organophosphate compounds rapidly decreased, the activity of cholinesterase showed a prolonged inhibition. |
1(0,0,0,1) | Details |
| 2328150 | Jovanovic D, Randjelovic S, Joksovic D: A case of unusual suicidal poisoning by the organophosphorus insecticide dimethoate. Hum Exp Toxicol. 1990 Jan;9(1):49-51. General weakness, muscular fibrillations and a marked inhibition of red blood cell and serum cholinesterases were the prominent signs of intoxication. |
3(0,0,0,3) | Details |
| 2803006 | Reena K, Ajay K, Sharma CB: Haematological changes induced by dimethoate in rat. Arh Hig Rada Toksikol. 1989 Mar;40(1):23-7. After 30 days of exposure, the levels of blood urea, total and the activities of glutamic-oxalacetic transaminase, glutamic- pyruvic transaminase and amylase markedly increased, but the activities of acid phosphatase and cholinesterase significantly decreased. |
1(0,0,0,1) | Details |
| 16718740 | Kristensen M, Huang J, Qiao CL, Jespersen JB: Variation of Musca domestica L. acetylcholinesterase in Danish housefly populations. Pest Manag Sci. 2006 Aug;62(8):738-45. Anti-cholinesterase resistance is in many cases caused by modified acetylcholinesterase (MACE). A comparison was made of toxicological data and AChE activity gathered from 21 field populations and nine laboratory strains of houseflies, Musca domestica L., to elucidate the best way of generating data to provide advice for management strategies and gathering information for resistance risk assessment on the organophosphates azamethiphos and dimethoate and the methomyl, which have been the primary insecticides used against adult houseflies in Denmark. |
2(0,0,0,2) | Details |
| 17869494 | Sayim F: Dimethoate-induced biochemical and histopathological changes in the liver of rats. Exp Toxicol Pathol. 2007 Nov;59(3-4):237-43. Epub 2007 Sep 14. At the end of the experiment, body weight gain, absolute and relative liver weights, liver cholinesterase activities and total protein levels were determined. |
2(0,0,0,2) | Details |
| 1028593 | Voitenko GA, Larionov VG: [Toxic effect of phosphamide after simultaneous entrance into the body through the gastrointestinal tract and respiratory organs]. Farmakol Toksikol. 1976 Sep-Oct;39(5):622-5. In evidence was also a more marked anticholinesterase action, a higher level of the insecticide in biological substrates and greater changes of hematological indices. |
1(0,0,0,1) | Details |
| 17972302 | Kristensen M, Jespersen JB: Susceptibility to thiamethoxam of Musca domestica from Danish livestock farms. Pest Manag Sci. 2008 Feb;64(2):126-32. BACKGROUND: Neonicotinoid baits are currently replacing anticholinesterase baits for control of adult houseflies (Musca domestica L.). There was no correlation between the toxicities of thiamethoxam and spinosad, dimethoate, methomyl, bioresmethrin or azamethiphos. |
1(0,0,0,1) | Details |
| 19079407 | Paudyal BP: Organophosphorus poisoning. JNMA J Nepal Med Assoc. 2008 Oct-Dec;47(172):251-8. Metacid (Methyl parathion) and Nuvan (Dichlorovos) are commonly ingested OP pesticides; Dimethoate, Profenofos, and Chlorpyrifos are other less frequently ingested compounds in Nepal. |
0(0,0,0,0) | Details |
| 8473001 | Krieger RI, Thongsinthusak T: Metabolism and excretion of dimethoate following ingestion of overtolerance peas and a bolus dose. Food Chem Toxicol. 1993 Mar;31(3):177-82. |
0(0,0,0,0) | Details |
| 1324992 | De Bleecker J, Van Den Neucker K, Willems J: The intermediate syndrome in organophosphate poisoning: presentation of a case and review of the literature. J Toxicol Clin Toxicol. 1992;30(3):321-9; discussion 331-2. A dimethoate-poisoned woman gradually developed a moderately severe cholinergic crisis that was readily treated by atropine. Cholinesterase inhibition was severe, and EMG revealed marked decrements at low rates of repetitive nerve stimulation, and increments at a high rate. |
2(0,0,0,2) | Details |
| 15800032 | Moser VC, Casey M, Hamm A, Carter WH Jr, Simmons JE, Gennings C: Neurotoxicological and statistical analyses of a mixture of five organophosphorus pesticides using a ray design. Toxicol Sci. 2005 Jul;86(1):101-15. Epub 2005 Mar 30. In this study, we tested for interaction (s) in a mixture of five organophosphorus (OP) pesticides (chlorpyrifos, diazinon, dimethoate, acephate, and malathion). A series of behavioral measures were evaluated in adult male Long-Evans rats at the time of peak effect following a single oral dose, and then tissues were collected for measurement of cholinesterase (ChE) activity. |
2(0,0,0,2) | Details |
| 19428953 | Aurbek N, Thiermann H, Eyer F, Eyer P, Worek F: Suitability of human butyrylcholinesterase as therapeutic marker and pseudo catalytic scavenger in organophosphate poisoning: a kinetic analysis. Toxicology. 2009 May 17;259(3):133-9. Epub 2009 Mar 6. As oxime efficacy is limited with certain OPs (e.g. dimethoate, tabun, soman) alternative therapeutic approaches, e.g. the application of scavengers (BChE) which may sequester OPs before they reach their physiological target, are under investigation. |
2(0,0,0,2) | Details |
| 9490325 | Aprea C, Sciarra G, Sartorelli P, Mancini R, Di Luca V: Environmental and biological monitoring of exposure to mancozeb, ethylenethiourea, and dimethoate during industrial formulation. J Toxicol Environ Health A. 1998 Feb 20;53(4):263-81. |
0(0,0,0,0) | Details |
| 8065512 | Mehl A, Schanke TM, Johnsen BA, Fonnum F: The effect of trichlorfon and other organophosphates on prenatal brain development in the guinea pig. Neurochem Res. 1994 May;19(5):569-74. The organophosphates trichlorfon, dichlorvos, dimethoate, soman, triortho-cresyl (TOCP), and the diethoxy-analogue of trichlorfon (O,O-diethyl 2,2,2-trichloro-1-hydroxyethylphosphonate, ethyl-trichlorfon), were administered to guinea pigs between day 42 and 46 of gestation. |
0(0,0,0,0) | Details |
| 3247000 | Nagymajtenyi L, Desi I, Lorencz R: Neurophysiological markers as early signs of organophosphate neurotoxicity. Neurotoxicol Teratol. 1988 Sep-Oct;10(5):429-34. The central and peripheral nervous system effects of acute and subchronic exposure to three organic phosphoro-acid esters (dimethoate, dichlorvos, parathion-methyl) were studied. Cholinesterase activity was measured in blood, brain and other organs. |
2(0,0,0,2) | Details |
| 3241808 | Izmirova N, Kaloianova F, Kalinova G, Simeonov P: [A new approach to setting hygienic pesticide standards] . Probl Khig. 1988;13:98-109. Studies are performed on tobacco-pickers, which are in contact with dimethoate and tomato-pickers, sprayed with methylparathion. The content of pesticides in washes from leaves and hands, as well as inhibitors and cholinesterase activity in serum and erythrocytes is measured. |
1(0,0,0,1) | Details |
| 2593091 | Audegond L, Catez D, Foulhoux P, Fournex R, Le Rumeur C, L'Hotellier M, Stepniewski JP: [Potentiation of deltamethrin toxicity by organophosphorus insecticides] . J Toxicol Clin Exp. 1989 May-Jun;9(3):163-76. It was demonstrated that some of them (azinphos, dichlorvos, dimethoate, fenitrothion, omethoate) induce an increase of toxicity of deltamethrin. Cholinesterase inhibitors of the group are ineffective. |
1(0,0,0,1) | Details |
| 10207609 | Worek F, Diepold C, Eyer P: Dimethylphosphoryl-inhibited human cholinesterases: inhibition, reactivation, and aging kinetics. Arch Toxicol. 1999 Feb;73(1):7-14. Human poisoning by organophosphates bearing two methoxy groups, e.g. by malathion, paraoxon-methyl, dimethoate and oxydemeton-methyl, is generally considered to be rather resistant to oxime therapy. |
1(0,0,0,1) | Details |
| 18584359 | Samarawickrema N, Pathmeswaran A, Wickremasinghe R, Peiris-John R, Karunaratna M, Buckley N, Dawson A, de Silva J: Fetal effects of environmental exposure of pregnant women to organophosphorus compounds in a rural farming community in Sri Lanka. Clin Toxicol. 2008 Jul;46(6):489-95. METHODS: Toxicity was assessed by measuring (i) OPC levels in breast milk and plasma from maternal and cord blood using gas chromatography, (ii) maternal and fetal butyrylcholinesterase (BChE) activity using inhibition assays, (iii) antioxidant status of the fetus using superoxide dismutase activity assays, (iv) oxidative stress in the fetus by determining malondialdehyde (MDA) concentrations, and (v) examining for fetal DNA fragmentation using electrophoresis. RESULTS: Organophosphate residues were detected in only two subjects (chlorpyrifos in maternal and cord blood of one during the spray season and dimethoate in breast milk of another during the in between spray season), but the test employed was capable of only detecting concentrations above 0.05 mg/l. |
1(0,0,0,1) | Details |
| 9211701 | Brunet R, McDuff J: Recovery of brain cholinesterases of brown-headed cowbirds from organophosphorus intoxication: effect of environmental temperature. Bull Environ Contam Toxicol. 1997 Aug;59(2):285-91. |
1(0,0,0,1) | Details |
| 8693683 | Tsatsakis AM, Aguridakis P, Michalodimitrakis MN, Tsakalov AK, Alegakis AK, Koumantakis E, Troulakis G: Experiences with acute organophosphate poisonings in Crete. Vet Hum Toxicol. 1996 Apr;38(2):101-7. Significant fluctuations of the plasma cholinesterase activity were observed during therapy. |
1(0,0,0,1) | Details |
| 7387183 | Barker RJ, Lehner Y, Kunzman MR: Pesticides and honey bees: nectar and pollen contamination in alfalfa treated with dimethoate. Arch Environ Contam Toxicol. 1980;9(2):125-33. As little as 1 ppm added to syrup fed to caged workers of Apis mellifera L. (Apidae) inhibited cholinesterase and reduced survival. |
1(0,0,0,1) | Details |
| 17531286 | Domingues I, Guilhermino L, Soares AM, Nogueira AJ: Assessing dimethoate contamination in temperate and tropical climates: potential use of biomarkers in bioassays with two chironomid species. Chemosphere. 2007 Aug;69(1):145-54. Epub 2007 May 24. Preliminary studies included investigations of the effects of temperature on larval development and the influence of larval age on normal variability of cholinesterase (ChE) and glutathione S-transferase (GST) activities and protein content. |
1(0,0,0,1) | Details |
| 7250311 | Kunec-Vajic E: Effect of cholinesterase on the chemiluminescence of luminol. Experientia. 1981 May 15;37(5):462-3. |
1(0,0,0,1) | Details |
| 813485 | Richter D, Runde H: [Effect of organophosphorus compounds on esterases in rat intestine- a comparing histochemical and biochemical investigation (author's transl)]. Acta Histochem. 1975;54(1):56-65. They differ in cholinesterases. |
1(0,0,0,1) | Details |
| 4026575 | Marosi G, Ivan J, Nagymajtenyi L, Csatlos I, Toszegi A: Dimethoate-induced toxic cardiac failure in the guinea pig. Arch Toxicol. 1985 Jun;57(2):142-3. The toxic cardiac phenomena appeared to be unrelated to the degree of cholinesterase inhibition, but showed a close correlation with myocardial dimethoate concentration. |
82(1,1,1,2) | Details |
| 17370803 | Booth LH, Wratten SD, Kehrli P: Effects of reduced rates of two insecticides on enzyme activity and mortality of an aphid and its lacewing predator. J Econ Entomol. 2007 Feb;100(1):11-9. Cholinesterase activity was reduced by dimethoate exposure in the laboratory in both species, but there were species-specific differences. |
82(1,1,1,2) | Details |
| 16611628 | Moser VC, Simmons JE, Gennings C: Neurotoxicological interactions of a five-pesticide mixture in preweanling rats. Toxicol Sci. 2006 Jul;92(1):235-45. Epub 2006 Apr 11. We have conducted interaction studies using a mixture of five organophosphorus (OP) pesticides (chlorpyrifos, diazinon, dimethoate, acephate, and malathion) in both adult (published previously) and preweanling rats using a fixed-ratio ray design. In the present study, cholinesterase inhibition and behavioral changes (motor activity, gait, and tail-pinch response) were measured in 17-day-old Long-Evans male rats following acute exposure to the OPs. |
1(0,0,0,1) | Details |
| 14694629 | Xiao C, Niu Y, He F: [Stimulation single fiber electromyography in rats with myasthenia induced by organophosphorus insecticides and their mixtures poisoning]. Zhonghua Lao Dong Wei Sheng Zhi Ye Bing Za Zhi. 2002 Apr;20(2):125-7. OBJECTIVE: To study the neuromuscular function and its relation with the occurrence of myasthenia in rats poisoned by dimethoate (D), phoxim (P), methomyl (M), M + D and M + P respectively. The whole blood cholinesterase (ChE) activity was measured 1 h before and after poisoning. |
1(0,0,0,1) | Details |
| 3775801 | Yasoshima M, Masuda Y: Effect of carbon disulfide on the anticholinesterase action of several organophosphorus insecticides in mice. Toxicol Lett. 1986 Sep;32(3):179-84. CS2 pretreatment potentiated the anticholinesterase action of parathion and EPN, but suppressed that of dimethoate and diazinon. |
34(0,1,1,4) | Details |
| 7481747 | Johnston G: The study of interactive effects of pollutants: a biomarker approach. Sci Total Environ. 1995 Oct 27;171(1-3):205-12. Birds pretreated with prochloraz showed a trend towards greater inhibition of serum BuChE activity at most time points following dosing with the OPs dimethoate and chlorpyriphos. |
34(0,1,1,4) | Details |
| 14694657 | Sun Y, Zhou Z, Hu Y, Chen J, Jin T: [The acute effects of dimethoate on the muscarinic-receptors of rat brains and the relationship between muscarinic-receptors and cholinesterase]. Zhonghua Lao Dong Wei Sheng Zhi Ye Bing Za Zhi. 2002 Aug;20(4):293-5. |
32(0,1,1,2) | Details |
| 5971874 | Zech R, Engelhard H, Erdmann WD: [Reactions of pyridinium oximes with the alkyl dimethoate and its derivatives. Biochim Biophys Acta. 1966 Nov 15;128(2):363-71. Effects upon cholinesterases] |
1(0,0,0,1) | Details |
| 72085 | Bradway DE, Shafik TM, Lores EM: Comparison of cholinesterase activity, residue levels, and urinary metabolite excretion of rats exposed to organophosphorus pesticides. J Agric Food Chem. 1977 Nov-Dec;25(6):1353-8. |
1(0,0,0,1) | Details |
| 9591287 | Papp A, Gyorgyi K, Nagymajtenyi L, Desi I: Effects of different types of anticholinesterase agents on in vivo hippocampal population spikes in rats. Neurobiology. 1997;5(4):495-8. |
1(0,0,0,1) | Details |
| 19034799 | DeSesso JM, Watson RE, Keen CL, Hazelden KP, Haws LC, Li AA: Analysis and integration of developmental neurotoxicity and ancillary data into risk assessment: a case study of dimethoate. J Toxicol Environ Health A. 2009;72(2):94-109. Based on an analysis of a developmental neurotoxicity (DNT) study, a cholinesterase (ChE) sensitivity study, a cross-fostering study, and several single- and multigenerational reproductive toxicity studies, two potential critical endpoints for dimethoate were identified: brain ChE inhibition (ChEI) in adult females, and pup mortality. |
31(0,1,1,1) | Details |
| 8308950 | Buckley NA, Dawson AH, Whyte IM: Organophosphate poisoning: peripheral vascular resistance--a measure of adequate atropinization. J Toxicol Clin Toxicol. 1994;32(1):61-8. We propose that stability of the plasma cholinesterase at 6 to 8 h after temporarily suspending oxime provides a rapid guide to the duration of therapy, especially in patients whose complications make clinical assessment difficult. |
1(0,0,0,1) | Details |
| 19778190 | Thiermann H, Worek F, Eyer P, Eyer F, Felgenhauer N, Zilker T: Obidoxime in acute organophosphate poisoning: 2 - PK/PD relationships. Clin Toxicol. 2009 Sep;47(8):807-13. METHODS: We measured the activity of cholinesterase in plasma and AChE in red blood cells (RBC) and related these data with neuromuscular transmission analysis (ulnar nerve stimulation). |
1(0,0,0,1) | Details |
| 3143911 | Tripathy NK, Majhi B, Dey L, Das CC: Genotoxicity of Rogor studied in the sex-linked recessive lethal test and wing, eye and female germ-line mosaic assays in Drosophila melanogaster. Mutat Res. 1988 Nov;206(3):351-60. The genotoxic potential of Rogor (dimethoate), an anticholinesterase organophosphate insecticide, has been studied in the sex-linked recessive lethal test and the wing, eye and female germ-line mosaic assays in Drosophila melanogaster. |
31(0,1,1,1) | Details |
| 2588727 | Deike S, Wosnitzka H: [Behavior of serum cholinesterase and acetylcholinesterase activity in acute dimethoate poisoning]. Z Gesamte Inn Med. 1989 Sep 15;44(18):548-9. |
14(0,0,2,4) | Details |
| 19778163 | Eyer F, Worek F, Eyer P, Felgenhauer N, Haberkorn M, Zilker T, Thiermann H: Obidoxime in acute organophosphate poisoning: 1 - clinical effectiveness. Clin Toxicol. 2009 Sep;47(8):798-806. In this article we report clinical findings and activity of cholinesterase in plasma and acetylcholinesterase (AChE) in red blood cells. METHODS: We studied 34 atropinized patients with severe parathion, oxydemeton methyl, and dimethoate self-poisoning who were treated with obidoxime in a standard protocol. |
1(0,0,0,1) | Details |
| 16833148 | Long SM, Dawson A, Shore RF: A comparison of the effects of single and repeated exposure to an organophosphate insecticide on acetylcholinesterase activity in mammals. Environ Toxicol Chem. 2006 Jul;25(7):1857-63. Our results indicate that acute dose-response toxicity studies are suitable models for predicting the likely occurrence of adverse effects from either short- or longer-term exposure of wild mammals to anticholinesterase compounds. We compared the effects of single or repeated (hourly and daily) exposure to dimethoate on acetylcholinesterase (AChE) activity in laboratory mice to assess the suitability of standard laboratory tests for assessing risk. |
1(0,0,0,1) | Details |
| 16099569 | Reiss R, Gaylor D: Use of benchmark dose and meta-analysis to determine the most sensitive endpoint for risk assessment for dimethoate. Regul Toxicol Pharmacol. 2005 Oct;43(1):55-65. Benchmark dose (BMD) modeling was used to determine the appropriate point of departure for regulatory purposes, the lower limit of the BMD for a 5% incidence for pup mortality (BMDL (5)) and the lower limit of a 10% inhibition of brain cholinesterase (BMDL (10)*), the asterisk denotes that the BMD is based on continuous response variable as opposed to an incidence level. |
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| 8481105 | Willems JL, De Bisschop HC, Verstraete AG, Declerck C, Christiaens Y, Vanscheeuwyck P, Buylaert WA, Vogelaers D, Colardyn F: Cholinesterase reactivation in organophosphorus poisoned patients depends on the plasma concentrations of the oxime pralidoxime methylsulphate and of the organophosphate. Arch Toxicol. 1993;67(2):79-84. We measured in nine patients, poisoned by organophosphorus agents (ethyl parathion, ethyl and methyl parathion, dimethoate, or bromophos), erythrocyte and serum cholinesterase activities, and plasma concentrations of the organophosphorus agent. |
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| 7256362 | Areekul S, Srichairat S, Kirdudom P: Serum and red cell cholinesterase activity in people exposed to organophosphate insecticides. Southeast Asian J Trop Med Public Health. 1981 Mar;12(1):94-8. |
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| 2588705 | Goedicke HJ, Hermes H, Wagner R: [Exposure to residues on plant surfaces following the use of plant pesticides in the greenhouse]. Z Gesamte Hyg. 1989 Sep;35(9):531-3. Initial residues on the surface of cucumber, tomato and ornamental plants, and half-life periods for residue degradation are outlined for carbendazim, dimethoate, fenazox, malathion, methamidophos and pirimiphos-methyl. Residues on plants, concentration in the air, dermal exposition, and inhibition of serum choline esterase activity are shown for methamidophos and aldicarb, respective reentry times being discussed. |
1(0,0,0,1) | Details |
| 17997881 | Wu QE, Yao XM, Ban TT, Jiang N, Shao CF, Chang XL, Zhou ZJ: [Effects of 90-day oral dimethoate exposure on glutamatergic system and neurobehavioral performance in rats]. Zhonghua Lao Dong Wei Sheng Zhi Ye Bing Za Zhi. 2007 Sep;25(9):513-7. OBJECTIVE: To investigate the spatial learning and exploration along with the CNS excitatory amino acid neurotransmitters profiles in adult rats subchronically exposed to the anticholinesterase organophosphorus insecticide dimethoate. |
7(0,0,1,2) | Details |
| 19069555 | Nazarian A: Utilization of dimethoate by wild type Pseudomonas putida from polluted sites in Iran. Pak J Biol Sci. 2007 Feb 15;10(4):664-7. Anti-acetyl choline esterase (AChE) Activity was proportionally displayed of dimethoate level (0.2-2 g L (-1)). |
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| 18375477 | Eddleston M, Eyer P, Worek F, Sheriff MH, Buckley NA: Predicting outcome using butyrylcholinesterase activity in organophosphorus pesticide self-poisoning. QJM. 2008 Jun;101(6):467-74. Epub 2008 Mar 28. METHODS: We prospectively studied 91 and 208 patients with proven dimethoate or chlorpyrifos self-poisoning treated using a standard protocol. |
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| 8687995 | Dell'Omo G, Shore RF: Behavioral and physiological effects of acute sublethal exposure to dimethoate on wood mice, Apodemus sylvaticus. Arch Environ Contam Toxicol. 1996 Jul;31(1):91-7. The effects on behavior and cholinesterase (ChE) of an OP pesticide, dimethoate, were examined in wood mice under laboratory conditions. |
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| 18384757 | Eyer P, Radtke M, Worek F: Reactions of isodimethoate with human red cell acetylcholinesterase. . Biochem Pharmacol. 2008 May 15;75(10):2045-53. Epub 2008 Feb 26. Isodimethoate is a thermal decomposition product that is present in usual pesticide formulations of dimethoate. Owing to its PO structure the compound is a direct anticholinesterase agent whose properties, to the best of our knowledge, are presented here for the first time. |
1(0,0,0,1) | Details |
| 16479380 | Hoffmann U, Papendorf T: Organophosphate poisonings with parathion and dimethoate. . Intensive Care Med. 2006 Mar;32(3):464-8. Epub 2006 Feb 15. The serum pseudo-cholinesterase activity was below the limit of detection at admission and recovered during the following 3weeks. |
1(0,0,0,1) | Details |
| 954759 | Okonek S, Boelcke G, Hollmann H: Therapeutic properties.of haemodialysis and blood exchange transfusion in organophosphate poisoning. Eur J Intensive Care Med. 1976;2(1):13-8. Human blood was contaminated with nitrostigmine, dimethoate and demeton-S-methyl sulfoxide. The influence of blood exchange transfusion on poison elimination as well as on the cholinesterase activity of blood, brain and muscle was studied in rats poisoned with nitrostigmine. |
1(0,0,0,1) | Details |