| Name | glycerophosphate dehydrogenase |
|---|---|
| Synonyms | Alpha glycerol 3 phosphate dehydrogenase; GPD C; GPD 1; GPD1; GPDH C; Glycerol 3 phosphate dehydrogenase 1; Glycerophosphate dehydrogenase; glycerol 3 phosphate dehydrogenase 1 (soluble)… |
| Name | rotenone |
|---|---|
| CAS |
| PubMed | Abstract | RScore(About this table) | |
|---|---|---|---|
| 2321971 | Bolter CJ, Chefurka W: Extramitochondrial release of peroxide from insect and mouse liver mitochondria using the respiratory inhibitors phosphine, myxothiazol, and antimycin and spectral analysis of inhibited cytochromes. Arch Biochem Biophys. 1990 Apr;278(1):65-72. Data obtained using antimycin and myxothiazol, both of which stimulated the release of H2O2 from insect mitochondria, lead to the conclusion that glycerophosphate dehydrogenase is a source of H2O2. Other respiratory inhibitors, antimycin, myxothiazol, and rotenone were used with insect mitochondria. |
1(0,0,0,1) | Details |
| 8392019 | Sohal RS: Aging, cytochrome oxidase activity, and peroxide release by mitochondria. Free Radic Biol Med. 1993 Jun;14(6):583-8. Results are interpreted to suggest that partial inhibition of cytochrome c oxidase activity can lead to the stimulation of mitochondrial H2O2 production in the housefly at site (s) other than NADH dehydrogenase and /cytochrome b region; a possible source may be glycerophosphate dehydrogenase. H2O2 release by mitochondria, inhibited with rotenone and antimycin A, was greatly increased upon supplementation with however, the further addition of KCN or myxothiazol, to such preparations, caused a depression of H2O2 generation. |
1(0,0,0,1) | Details |